Mechanical stress is communicated between different cell types to elicit matrix remodeling - Wikidata - wikimedia - TriplyDB

Mechanical stress is communicated between different cell types to elicit matrix remodeling

Mechanical stress is communicated between different cell types to elicit matrix remodeling

http://www.wikidata.org/entity/Q35933851

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Epithelial-derived TGF-beta2 modulates basal and wound-healing subepithelial matrix homeostasis Cellular Biomechanics in Drug Screening and Evaluation: Mechanopharmacology TRPV4 regulates calcium homeostasis, cytoskeletal remodeling, conventional outflow and intraocular pressure in the mammalian eye.Mechanical Stimulation by Postnasal Drip Evokes Cough A mechanical design principle for tissue structure and function in the airway tree Physical properties of alginate hydrogels and their effects on in vitro follicle development.Using gold nanorods to probe cell-induced collagen deformation.Acoustically detectable cellular-level lung injury induced by fluid mechanical stresses in microfluidic airway systems.Microporous poly(L-lactic acid) membranes fabricated by polyethylene glycol solvent-cast/particulate leaching technique Manipulating the microvasculature and its microenvironment.Mimicking the host and its microenvironment in vitro for studying mucosal infections by Pseudomonas aeruginosa.Mathematical modeling of left ventricular dimensional changes in mice during aging.Differences in MAP kinase phosphorylation in response to mechanical strain in asthmatic fibroblasts.Computational modeling of extracellular mechanotransduction.Normal scarring: importance of myofibroblasts.Smad3 deficiency alters key structural elements of the extracellular matrix and mechanotransduction of wound closure.Bronchoconstriction Induces TGF-β Release and Airway Remodelling in Guinea Pig Lung Slices.Mechanical stimuli and IL-13 interact at integrin adhesion complexes to regulate expression of smooth muscle myosin heavy chain in airway smooth muscle tissue.TNF-α-converting enzyme/a disintegrin and metalloprotease-17 mediates mechanotransduction in murine tracheal epithelial cells.Cellular stress failure in ventilator-injured lungs Longitudinal changes in tear fluid lipidome brought about by eyelid-warming treatment in a cohort of meibomian gland dysfunction.Unjamming and cell shape in the asthmatic airway epithelium.Epithelial-mesenchymal interactions in pulmonary fibrosis.Tissue factor-bearing exosome secretion from human mechanically stimulated bronchial epithelial cells in vitro and in vivo.Topical negative pressure therapy: mechanisms and indications.IL-13 induces a bronchial epithelial phenotype that is profibrotic.Tissue inhibitor of metalloproteinase-1 moderates airway re-epithelialization by regulating matrilysin activity The management of deep sternal wound infections using vacuum assisted closure (V.A.C.) therapy.Matrix metalloproteinases in lung: multiple, multifarious, and multifaceted.IL-13 Augments Compressive Stress-Induced Tissue Factor Expression in Human Airway Epithelial Cells Putting the Squeeze on Airway Epithelia Chemical sensing and imaging with metallic nanorods.Mechanical compression attenuates normal human bronchial epithelial wound healing.Recent advances and new opportunities in lung mechanobiology.Defining a stimuli-response relationship in compensatory lung growth following major resection.The sentinel role of the airway epithelium in asthma pathogenesis.Artificial airways for the study of respiratory disease.Computational and bioengineered lungs as alternatives to whole animal, isolated organ, and cell-based lung models.Airway remodelling in asthma: role for mechanical forces.Novel device for application of continuous mechanical tensile strain to mammalian cells.

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P2860

Mechanical stress is communicated between different cell types to elicit matrix remodeling

http://www.wikidata.org/entity/Q35933851

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2001 nî lūn-bûn

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2001年论文

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2001年论文

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name

Mechanical stress is communica ...... es to elicit matrix remodeling

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Mechanical stress is communica ...... es to elicit matrix remodeling

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Mechanical stress is communica ...... es to elicit matrix remodeling

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P2093

D J Tschumperlin

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J M Drazen

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M A Swartz

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R D Kamm

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P2860

Evidence for a glycerol pathway through aquaporin 1 (CHIP28) channels

Biosynthetic response of cartilage explants to dynamic compression

Inhaled corticosteroids decrease subepithelial collagen deposition by modulation of the balance between matrix metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 expression in asthma

Airway wall mechanics.

On the mechanism of mucosal folding in normal and asthmatic airways.

The pathobiology of bronchial asthma.

Histological features of the airways in asthma and COPD.

The transcription factor EGR-1 suppresses transformation of human fibrosarcoma HT1080 cells by coordinated induction of transforming growth factor-beta1, fibronectin, and plasminogen activator inhibitor-1.

Fluid shear stress activation of egr-1 transcription in cultured human endothelial and epithelial cells is mediated via the extracellular signal-related kinase 1/2 mitogen-activated protein kinase pathway.

Growth factors secreted by bronchial epithelial cells control myofibroblast proliferation: an in vitro co-culture model of airway remodeling in asthma.

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6180-6185

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scholarly article

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10.1073/PNAS.111133298

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11

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98

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2001-05-15T00:00:00Z

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11983763

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11353845

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33442

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