Frequent clones of p53-mutated keratinocytes in normal human skin - Wikidata - wikimedia - TriplyDB

Frequent clones of p53-mutated keratinocytes in normal human skin

Frequent clones of p53-mutated keratinocytes in normal human skin

http://www.wikidata.org/entity/Q35940911

about

Regulation of global genome nucleotide excision repair by SIRT1 through xeroderma pigmentosum C The basal layer in human squamous tumors harbors more UVA than UVB fingerprint mutations: a role for UVA in human skin carcinogenesis UVB-induced apoptosis drives clonal expansion during skin tumor development Galectin-7 overexpression is associated with the apoptotic process in UVB-induced sunburn keratinocytes Induction of basal cell carcinomas and trichoepitheliomas in mice overexpressing GLI-1 Digital PCR A Model for UV-Induction of Skin Cancer Current concepts in the diagnosis and pathobiology of intraepithelial neoplasia: A review by organ system Incidence, risk factors and outcomes of de novo malignancies post liver transplantation Response of transposable elements to environmental stressors Cells of origin and tumor-initiating cells for nonmelanoma skin cancers Field cancerisation in colorectal cancer: a new frontier or pastures past?Multifocal epithelial tumors and field cancerization: stroma as a primary determinant Epithelial cancers in the post-genomic era: should we reconsider our lifestyle?An evolutionary paradigm for carcinogenesis?Persistent p53 mutations in single cells from normal human skin More epidermal p53 patches adjacent to skin carcinomas in renal transplant recipients than in immunocompetent patients: the role of azathioprine.FOXM1 allows human keratinocytes to bypass the oncogene-induced differentiation checkpoint in response to gain of MYC or loss of p53.Irradiation selects for p53-deficient hematopoietic progenitors.Ultraviolet light induces apoptosis via direct activation of CD95 (Fas/APO-1) independently of its ligand CD95L Clinical implications and utility of field cancerization Stochastic fate of p53-mutant epidermal progenitor cells is tilted toward proliferation by UV B during preneoplasia.Randomized Vehicle-Controlled Study of Short Drug Incubation Aminolevulinic Acid Photodynamic Therapy for Actinic Keratoses of the Face or Scalp Field cancerization, clonality, and epithelial stem cells: the spread of mutated clones in epithelial sheets.ATR-Chk1 pathway inhibition promotes apoptosis after UV treatment in primary human keratinocytes: potential basis for the UV protective effects of caffeine.SRC induces podoplanin expression to promote cell migration Reduction of IKKalpha expression promotes chronic ultraviolet B exposure-induced skin inflammation and carcinogenesis Carcinogenesis in mouse and human cells: parallels and paradoxes.Multiple clonal abnormalities in the bronchial epithelium of patients with lung cancer.The clonal origin and clonal evolution of epithelial tumours.The production and applications of genetically modified skin cells.Senescent fibroblasts promote epithelial cell growth and tumorigenesis: a link between cancer and aging.Escaping the stem cell compartment: sustained UVB exposure allows p53-mutant keratinocytes to colonize adjacent epidermal proliferating units without incurring additional mutations Applications of the human p53 knock-in (Hupki) mouse model for human carcinogen testing.Regional analysis of p53 mutations in rheumatoid arthritis synovium The role of mutant p53 in human cancer.Infrequent p53 gene mutation but UV gradient-like p53 protein positivity in keloids.Analysis of Tp53 codon 72 polymorphisms, Tp53 mutations, and HPV infection in cutaneous squamous cell carcinomas.Induction of KLF4 in basal keratinocytes blocks the proliferation-differentiation switch and initiates squamous epithelial dysplasia.Cutaneous field cancerization: clinical, histopathological and therapeutic aspects.

P2860

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P2860

Frequent clones of p53-mutated keratinocytes in normal human skin

http://www.wikidata.org/entity/Q35940911

description

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Frequent clones of p53-mutated keratinocytes in normal human skin

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Frequent clones of p53-mutated keratinocytes in normal human skin

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Frequent clones of p53-mutated keratinocytes in normal human skin

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Frequent clones of p53-mutated keratinocytes in normal human skin

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Frequent clones of p53-mutated keratinocytes in normal human skin

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Frequent clones of p53-mutated keratinocytes in normal human skin

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Proceedings of the National Academy of Sciences of the United States of America

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Frequent clones of p53-mutated keratinocytes in normal human skin

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D E Brash

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S Kunala

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P2860

Disruption of epithelial cell-matrix interactions induces apoptosis

A genetic model for colorectal tumorigenesis

The clonal evolution of tumor cell populations

Early p53 alterations in mouse skin carcinogenesis by UVB radiation: immunohistochemical detection of mutant p53 protein in clusters of preneoplastic epidermal cells.

Involvement of the cell-cycle inhibitor Cip1/WAF1 and the E1A-associated p300 protein in terminal differentiation.

The role of childhood exposure to sunlight in the development of solar keratoses and non-melanocytic skin cancer.

Analysis of the p53 gene in human precancerous actinic keratosis lesions and squamous cell cancers.

Aberrations of the tumor suppressor p53 gene and p53 protein in solar keratosis in human skin.

UV and skin cancer: specific p53 gene mutation in normal skin as a biologically relevant exposure measurement

A role for sunlight in skin cancer: UV-induced p53 mutations in squamous cell carcinoma.

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