Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
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Suppression of poly (ADP-ribose) polymerase activation by 3-aminobenzamide in a rat model of myocardial infarction: long-term morphological and functional consequencesBioenergetic metabolites regulate base excision repair-dependent cell death in response to DNA damage.Vincristine attenuates N-methyl-N'-nitro-N-nitrosoguanidine-induced poly-(ADP) ribose polymerase activity in cardiomyocytes.Poly(ADP-ribose) polymerase-1 dependence of stress-induced transcription factors and associated gene expression in glia.Loss of poly(ADP-ribose) polymerase-1 causes increased tumour latency in p53-deficient miceARTD1/PARP1 negatively regulates glycolysis by inhibiting hexokinase 1 independent of NAD+ depletion.Measurement and characterization of superoxide generation from xanthine dehydrogenase: a redox-regulated pathway of radical generation in ischemic tissues.Endothelial dysfunction in aging animals: the role of poly(ADP-ribose) polymerase activation.Excessive stimulation of poly(ADP-ribosyl)ation contributes to endothelial dysfunction in pre-eclampsia.Differentiation-Associated Downregulation of Poly(ADP-Ribose) Polymerase-1 Expression in Myoblasts Serves to Increase Their Resistance to Oxidative Stress.The late phase of ischemic preconditioning induces a prosurvival genetic program that results in marked attenuation of apoptosis.Inhibition of GAPDH activity by poly(ADP-ribose) polymerase activates three major pathways of hyperglycemic damage in endothelial cells.Salvage of nicotinamide adenine dinucleotide plays a critical role in the bioenergetic recovery of post-hypoxic cardiomyocytes.Poly (ADP-ribose) synthetase inhibitor has a heart protective effect in a rat model of experimental sepsis.Poly(ADP-ribosyl)ation in mammalian ageing.Role of poly(ADP-ribose) polymerase 1 (PARP-1) in cardiovascular diseases: the therapeutic potential of PARP inhibitorsTherapeutic applications of PARP inhibitors: anticancer therapy and beyondVitamin A depletion causes oxidative stress, mitochondrial dysfunction, and PARP-1-dependent energy deprivationPoly(ADP-ribosyl)ation basally activated by DNA strand breaks reflects glutamate-nitric oxide neurotransmission.CD38 Deficiency Protects the Heart from Ischemia/Reperfusion Injury through Activating SIRT1/FOXOs-Mediated Antioxidative Stress Pathway.Outer mitochondrial membrane localization of apoptosis-inducing factor: mechanistic implications for release.Noncleavable poly(ADP-ribose) polymerase-1 regulates the inflammation response in mice.Mitochondria mediates caspase-dependent and independent retinal cell death in Staphylococcus aureus endophthalmitis.Myocardial protection by selective poly(ADP-ribose) polymerase inhibitors.Targeting Sentinel Proteins and Extrasynaptic Glutamate Receptors: a Therapeutic Strategy for Preventing the Effects Elicited by Perinatal Asphyxia?Olaparib protects cardiomyocytes against oxidative stress and improves graft contractility during the early phase after heart transplantation in rats.Luteolinidin Protects the Postischemic Heart through CD38 Inhibition with Preservation of NAD(P)(H).
P2860
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P2860
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
description
2000 nî lūn-bûn
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2000年の論文
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2000年論文
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2000年論文
@zh-hant
2000年論文
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2000年論文
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2000年論文
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2000年论文
@wuu
2000年论文
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2000年论文
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name
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
@ast
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
@en
type
label
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
@ast
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
@en
prefLabel
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
@ast
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
@en
P2093
P1433
P1476
Myocardial postischemic injury is reduced by polyADPripose polymerase-1 gene disruption.
@en
P2093
A A Pieper
E E Clements
J L Zweier
S H Snyder
P304
P407
P577
2000-04-01T00:00:00Z