Multiple functions of Drosophila BLM helicase in maintenance of genome stability. - Wikidata - wikimedia - TriplyDB

Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

http://www.wikidata.org/entity/Q35945619

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Rif1 provides a new DNA-binding interface for the Bloom syndrome complex to maintain normal replication Mus81 nuclease and Sgs1 helicase are essential for meiotic recombination in a protist lacking a synaptonemal complex The SMC-5/6 Complex and the HIM-6 (BLM) Helicase Synergistically Promote Meiotic Recombination Intermediate Processing and Chromosome Maturation during Caenorhabditis elegans Meiosis Delineation of joint molecule resolution pathways in meiosis identifies a crossover-specific resolvase The Drosophila STUbL protein Degringolade limits HES functions during embryogenesis.The Arabidopsis BLAP75/Rmi1 homologue plays crucial roles in meiotic double-strand break repair.dRecQ4 is required for DNA synthesis and essential for cell proliferation in Drosophila.Dual roles for DNA polymerase theta in alternative end-joining repair of double-strand breaks in Drosophila.The Drosophila Werner exonuclease participates in an exonuclease-independent response to replication stress.Three structure-selective endonucleases are essential in the absence of BLM helicase in Drosophila.Mitotic misbehavior of a Drosophila melanogaster satellite in ring chromosomes: insights into intragenomic conflict among heterochromatic sequences.Drosophila FANCM helicase prevents spontaneous mitotic crossovers generated by the MUS81 and SLX1 nucleases.Heteroduplex DNA position defines the roles of the Sgs1, Srs2, and Mph1 helicases in promoting distinct recombination outcomes.Bloom syndrome helicase stimulates RAD51 DNA strand exchange activity through a novel mechanism Helicase-inactivating mutations as a basis for dominant negative phenotypes.Interhomolog recombination and loss of heterozygosity in wild-type and Bloom syndrome helicase (BLM)-deficient mammalian cells.P transposable elements in Drosophila and other eukaryotic organisms Loss of the bloom syndrome helicase increases DNA ligase 4-independent genome rearrangements and tumorigenesis in aging Drosophila.Synthetic lethality of Drosophila in the absence of the MUS81 endonuclease and the DmBlm helicase is associated with elevated apoptosis.Annealing of Complementary DNA Sequences During Double-Strand Break Repair in Drosophila Is Mediated by the Ortholog of SMARCAL1.Evolution of an MCM complex in flies that promotes meiotic crossovers by blocking BLM helicase.Shank Modulates Postsynaptic Wnt Signaling to Regulate Synaptic Development.Drosophila homologue of the Rothmund-Thomson syndrome gene: essential function in DNA replication during development.Stwl modifies chromatin compaction and is required to maintain DNA integrity in the presence of perturbed DNA replication.DNA damage responses in Drosophila nbs mutants with reduced or altered NBS function.Genetic analysis of zinc-finger nuclease-induced gene targeting in Drosophila.Sources and structures of mitotic crossovers that arise when BLM helicase is absent in Drosophila.Removal of the bloom syndrome DNA helicase extends the utility of imprecise transposon excision for making null mutations in Drosophila.Bloom's Syndrome: Clinical Spectrum, Molecular Pathogenesis, and Cancer Predisposition.Double-strand break repair assays determine pathway choice and structure of gene conversion events in Drosophila melanogaster.Bloom syndrome helicase in meiosis: Pro-crossover functions of an anti-crossover protein.Repair of Meiotic DNA Breaks and Homolog Pairing in Mouse Meiosis Requires a Minichromosome Maintenance (MCM) Paralog.DNA Repair in Drosophila: Mutagens, Models, and Missing Genes.Somatic recombination in adult tissues: What is there to learn?The absence of crossovers on chromosome 4 in Drosophila melanogaster: Imperfection or interesting exception?Biochemical Activities and Genetic Functions of the Drosophila melanogaster Fancm Helicase in DNA Repair.Drosophila RecQ5 is required for efficient SSA repair and suppression of LOH in vivo.Proliferation of Double-Strand Break-Resistant Polyploid Cells Requires Drosophila FANCD2.Bloom Syndrome Helicase Promotes Meiotic Crossover Patterning and Homolog Disjunction.The Role of Blm Helicase in Homologous Recombination, Gene Conversion Tract Length, and Recombination Between Diverged Sequences in Drosophilamelanogaster.

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Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

http://www.wikidata.org/entity/Q35945619

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Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

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Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

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Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

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Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

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Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

@ast

Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

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Multiple functions of Drosophila BLM helicase in maintenance of genome stability.

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Mitch McVey

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Sabrina L Andersen

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Yuri Broze

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P2860

Replication protein A physically interacts with the Bloom's syndrome protein and stimulates its helicase activity

The Bloom's syndrome gene product is homologous to RecQ helicases

Recql5 and Blm RecQ DNA helicases have nonredundant roles in suppressing crossovers.

Holliday junction resolution in human cells: two junction endonucleases with distinct substrate specificities

Direct association of Bloom's syndrome gene product with the human mismatch repair protein MLH1

The Bloom's syndrome gene product promotes branch migration of holliday junctions

Functional interaction between BLM helicase and 53BP1 in a Chk1-mediated pathway during S-phase arrest

The Bloom's syndrome helicase promotes the annealing of complementary single-stranded DNA

Slx1-Slx4 is a second structure-specific endonuclease functionally redundant with Sgs1-Top3.

SGS1, the Saccharomyces cerevisiae homologue of BLM and WRN, suppresses genome instability and homeologous recombination.

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