STING Activation by Translocation from the ER Is Associated with Infection and Autoinflammatory Disease. - Wikidata - wikimedia - TriplyDB

STING Activation by Translocation from the ER Is Associated with Infection and Autoinflammatory Disease.

STING Activation by Translocation from the ER Is Associated with Infection and Autoinflammatory Disease.

http://www.wikidata.org/entity/Q35956285

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Type I interferonopathies in pediatric rheumatology Implications of Spatiotemporal Regulation of Shigella flexneri Type Three Secretion Activity on Effector Functions: Think Globally, Act Locally Cytosolic Innate Immune Sensing and Signaling upon Infection Extracellular vesicles during Herpes Simplex Virus type 1 infection: an inquire DNA polymerase-α regulates the activation of type I interferons through cytosolic RNA:DNA synthesis IFI16 is required for DNA sensing in human macrophages by promoting production and function of cGAMP ER Adaptor SCAP Translocates and Recruits IRF3 to Perinuclear Microsome Induced by Cytosolic Microbial DNAs.The Chlamydia trachomatis Inclusion Membrane Protein CpoS Counteracts STING-Mediated Cellular Surveillance and Suicide Programs.Membrane Perturbation-Associated Ca2+ Signaling and Incoming Genome Sensing Are Required for the Host Response to Low-Level Enveloped Virus Particle Entry Activation of STING requires palmitoylation at the Golgi.MAP4-regulated dynein-dependent trafficking of BTN3A1 controls the TBK1-IRF3 signaling axis.How Bacteria Subvert Animal Cell Structure and Function.Chronic innate immune activation of TBK1 suppresses mTORC1 activity and dysregulates cellular metabolism.IFI16 and cGAS cooperate in the activation of STING during DNA sensing in human keratinocytes.Multifaceted roles of TRIM38 in innate immune and inflammatory responses.The Emerging Roles of STING in Bacterial Infections.Ubiquitination of STING at lysine 224 controls IRF3 activation.STING Requires the Adaptor TRIF to Trigger Innate Immune Responses to Microbial Infection Viral evasion of DNA-stimulated innate immune responses.The role of cGAS in innate immunity and beyond.Regulation and function of the cGAS-STING pathway of cytosolic DNA sensing.cGAS-cGAMP-STING: The three musketeers of cytosolic DNA sensing and signaling.How Do the Virulence Factors of Shigella Work Together to Cause Disease?Immune Diseases Associated with TREX1 and STING Dysfunction.Genomics, Biology, and Human Illness: Advances in the Monogenic Autoinflammatory Diseases.Emerging roles of rhomboid-like pseudoproteases in inflammatory and innate immune responses.Methods of Assessing STING Activation and Trafficking.Cyclic Dinucleotides in the Scope of the Mammalian Immune System.Trafficking-Mediated STING Degradation Requires Sorting to Acidified Endolysosomes and Can Be Targeted to Enhance Anti-tumor Response.Innate Immune Activation by cGMP-AMP Nanoparticles Leads to Potent and Long-Acting Antiretroviral Response against HIV-1.STING-associated vasculopathy develops independently of IRF3 in mice.Delicate regulation of the cGAS-MITA-mediated innate immune response.The cytosolic sensor STING is required for intestinal homeostasis and control of inflammation.Cooperative Immune Suppression by Escherichia coli and Shigella Effector Proteins.TANK-Binding Kinase 1-Dependent Responses in Health and Autoimmunity.Attenuation of cGAS-STING signaling is mediated by a p62/SQSTM1-dependent autophagy pathway activated by TBK1.Virulent poxviruses inhibit DNA sensing by preventing STING activation.Pro-inflammation Associated with a Gain-of-Function Mutation (R284S) in the Innate Immune Sensor STING.UBXN3B positively regulates STING-mediated antiviral immune responses.Herpes Simplex Virus 1 γ34.5 Protein Inhibits STING Activation That Restricts Viral Replication

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STING Activation by Translocation from the ER Is Associated with Infection and Autoinflammatory Disease.

http://www.wikidata.org/entity/Q35956285

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

STING Activation by Translocat ...... and Autoinflammatory Disease.

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STING Activation by Translocat ...... and Autoinflammatory Disease.

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type

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STING Activation by Translocat ...... and Autoinflammatory Disease.

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STING Activation by Translocat ...... and Autoinflammatory Disease.

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STING Activation by Translocat ...... and Autoinflammatory Disease.

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STING Activation by Translocat ...... and Autoinflammatory Disease.

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J.CHOM.2015.07.001

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Cell Host & Microbe

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STING Activation by Translocat ...... and Autoinflammatory Disease.

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Didi Chen

http://www.w3.org/2001/XMLSchema#string

Nan Yan

http://www.w3.org/2001/XMLSchema#string

Neal M Alto

http://www.w3.org/2001/XMLSchema#string

Nicole Dobbs

http://www.w3.org/2001/XMLSchema#string

Nikolay Burnaevskiy

http://www.w3.org/2001/XMLSchema#string

Vijay K Gonugunta

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P2860

Structurally distinct bacterial TBC-like GAPs link Arf GTPase to Rab1 inactivation to counteract host defenses

c-di-AMP secreted by intracellular Listeria monocytogenes activates a host type I interferon response

STING is a direct innate immune sensor of cyclic di-GMP

Atg9a controls dsDNA-driven dynamic translocation of STING and the innate immune response

Cyclic GMP-AMP Containing Mixed Phosphodiester Linkages Is An Endogenous High-Affinity Ligand for STING

STING regulates intracellular DNA-mediated, type I interferon-dependent innate immunity

Selective protection of an ARF1-GTP signaling axis by a bacterial scaffold induces bidirectional trafficking arrest.

Bacteria fighting back: how pathogens target and subvert the host innate immune system.

The cytosolic exonuclease TREX1 inhibits the innate immune response to human immunodeficiency virus type 1.

Activated STING in a vascular and pulmonary syndrome

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10.1016/J.CHOM.2015.07.001

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