Mitochondrial permeability transition in CNS trauma: cause or effect of neuronal cell death?
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Therapeutic window analysis of the neuroprotective effects of cyclosporine A after traumatic brain injuryTraumatic brain injury: can the consequences be stopped?MicroRNAs: Key Regulators in the Central Nervous System and Their Implication in Neurological DiseasesBrain injury following cardiac arrest: pathophysiology for neurocritical careRole of Glia in Memory Deficits Following Traumatic Brain Injury: Biomarkers of Glia DysfunctionMitochondrial permeability transition pore is a potential drug target for neurodegenerationMitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategiesReperfusion promotes mitochondrial biogenesis following focal cerebral ischemia in ratsBattery of behavioral tests in mice to study postoperative delirium.Emerging therapies in traumatic brain injurySpreading Depression, Spreading Depolarizations, and the Cerebral VasculatureThioredoxin-Mimetic-Peptides Protect Cognitive Function after Mild Traumatic Brain Injury (mTBI)Dietary fructose aggravates the pathobiology of traumatic brain injury by influencing energy homeostasis and plasticity.Mitochondrial proteomics in experimental autoimmune uveitis oxidative stressMethamphetamine- and trauma-induced brain injuries: comparative cellular and molecular neurobiological substratesSafety and tolerability of cyclosporin a in severe traumatic brain injury patients: results from a prospective randomized trialEarly mitochondrial dysfunction after cortical contusion injuryTraumatic brain injury: an overview of pathobiology with emphasis on military populations.Chronic treatment with anesthetic propofol improves cognitive function and attenuates caspase activation in both aged and Alzheimer's disease transgenic miceCyclosporine A, FK506, and NIM811 ameliorate prolonged CBF reduction and impaired neurovascular coupling after cortical spreading depression.Brain metabolism is significantly impaired at blood glucose below 6 mM and brain glucose below 1 mM in patients with severe traumatic brain injury.Acetyl-L-carnitine ameliorates mitochondrial dysfunction following contusion spinal cord injury.Peripheral nerve graft with immunosuppression modifies gene expression in axotomized CNS neurons.Brain-on-a-chip microsystem for investigating traumatic brain injury: Axon diameter and mitochondrial membrane changes play a significant role in axonal response to strain injuries.Role of oxidative stress and the mitochondrial permeability transition in methylmercury cytotoxicityNeuroproteomics: a biochemical means to discriminate the extent and modality of brain injury.Neuroprotective effects of N-acetyl-cysteine and acetyl-L-carnitine after spinal cord injury in adult rats.Mitochondrial protection after traumatic brain injury by scavenging lipid peroxyl radicals.Mitochondria, Amyloid β, and Alzheimer's DiseaseMitochondrial mechanisms of cell death and neuroprotection in pediatric ischemic and traumatic brain injury.Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in ratsBroad-spectrum neuroprotection against traumatic brain injury by agonism of peroxisome proliferator-activated receptorsMitochondria, oxidative stress, and temporal lobe epilepsyGlucose administration after traumatic brain injury exerts some benefits and no adverse effects on behavioral and histological outcomes.Establishing a Clinically Relevant Large Animal Model Platform for TBI Therapy Development: Using Cyclosporin A as a Case StudyKetone bodies mediate antiseizure effects through mitochondrial permeability transition.Lipid peroxidation in brain or spinal cord mitochondria after injuryAcetyl-L-carnitine treatment following spinal cord injury improves mitochondrial function correlated with remarkable tissue sparing and functional recovery.The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.Role of calcium and mitochondria in MeHg-mediated cytotoxicity.
P2860
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P2860
Mitochondrial permeability transition in CNS trauma: cause or effect of neuronal cell death?
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Mitochondrial permeability tra ...... effect of neuronal cell death?
@ast
Mitochondrial permeability tra ...... effect of neuronal cell death?
@en
type
label
Mitochondrial permeability tra ...... effect of neuronal cell death?
@ast
Mitochondrial permeability tra ...... effect of neuronal cell death?
@en
prefLabel
Mitochondrial permeability tra ...... effect of neuronal cell death?
@ast
Mitochondrial permeability tra ...... effect of neuronal cell death?
@en
P2093
P2860
P356
P1476
Mitochondrial permeability tra ...... effect of neuronal cell death?
@en
P2093
Rabchevsky AG
Springer JE
Sullivan PG
Waldmeier PC
P2860
P304
P356
10.1002/JNR.20292
P577
2005-01-01T00:00:00Z