Activation of two distinct Sox9-EGFP-expressing intestinal stem cell populations during crypt regeneration after irradiation.
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Reserve stem cells: Differentiated cells reprogram to fuel repair, metaplasia, and neoplasia in the adult gastrointestinal tractDefining hierarchies of stemness in the intestine: evidence from biomarkers and regulatory pathwaysRadiation enteropathy--pathogenesis, treatment and preventionSOX9-regulated cell plasticity in colorectal metastasis is attenuated by rapamycinmiR-30 Family Controls Proliferation and Differentiation of Intestinal Epithelial Cell Models by Directing a Broad Gene Expression Program That Includes SOX9 and the Ubiquitin Ligase PathwayUnraveling intestinal stem cell behavior with models of crypt dynamicsYY1 is indispensable for Lgr5+ intestinal stem cell renewal.Impact of diet-induced obesity on intestinal stem cells: hyperproliferation but impaired intrinsic function that requires insulin/IGF1Krüppel-like factor 4 is a radioprotective factor for the intestine following γ-radiation-induced gut injury in mice.Localized intestinal radiation and liquid diet enhance survival and permit evaluation of long-term intestinal responses to high dose radiation in miceIntestinal stem cells remain viable after prolonged tissue storage.Cell lineage identification and stem cell culture in a porcine model for the study of intestinal epithelial regeneration.Mesenchymal stem cell therapy stimulates endogenous host progenitor cells to improve colonic epithelial regeneration.Deletion of intestinal epithelial insulin receptor attenuates high-fat diet-induced elevations in cholesterol and stem, enteroendocrine, and Paneth cell mRNAs.Transcriptional corepressor MTG16 regulates small intestinal crypt proliferation and crypt regeneration after radiation-induced injuryLow- and high-LET radiation drives clonal expansion of lung progenitor cells in vivo.Notch receptor regulation of intestinal stem cell homeostasis and crypt regeneration.Porcine models of digestive disease: the future of large animal translational research.IGF1 stimulates crypt expansion via differential activation of 2 intestinal stem cell populations.Snai1 regulates cell lineage allocation and stem cell maintenance in the mouse intestinal epitheliumTissue underlying the intestinal epithelium elicits proliferation of intestinal stem cells following cytotoxic damage.Vascular Endothelial Growth Factor (VEGF) Bioavailability Regulates Angiogenesis and Intestinal Stem and Progenitor Cell Proliferation during Postnatal Small Intestinal DevelopmentBrief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury.CD24 can be used to isolate Lgr5+ putative colonic epithelial stem cells in miceElp3 drives Wnt-dependent tumor initiation and regeneration in the intestine.Lawsonia intracellularis exploits β-catenin/Wnt and Notch signalling pathways during infection of intestinal crypt to alter cell homeostasis and promote cell proliferation.Crypt base columnar stem cells in small intestines of mice are radioresistant.Nkx2.2 is expressed in a subset of enteroendocrine cells with expanded lineage potential.SOX9 maintains reserve stem cells and preserves radioresistance in mouse small intestine.BVES Regulates Intestinal Stem Cell Programs and Intestinal Crypt Viability after Radiation.Brachyury identifies a class of enteroendocrine cells in normal human intestinal crypts and colorectal cancer.Paneth cells expand from newly created and preexisting cells during repair after doxorubicin-induced damage.Isolation and characterization of intestinal stem cells based on surface marker combinations and colony-formation assay.Brief report: CD24 and CD44 mark human intestinal epithelial cell populations with characteristics of active and facultative stem cellsDistal airway epithelial progenitor cells are radiosensitive to High-LET radiationSOX9 is targeted for proteasomal degradation by the E3 ligase FBW7 in response to DNA damage.Identification, visualization and clonal analysis of intestinal stem cells in fish.Insulin receptor isoform switching in intestinal stem cells, progenitors, differentiated lineages and tumors: evidence that IR-B limits proliferation.Orphan Gpr182 suppresses ERK-mediated intestinal proliferation during regeneration and adenoma formation.Inhibition of Notch signaling reduces the number of surviving Dclk1+ reserve crypt epithelial stem cells following radiation injury.
P2860
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P2860
Activation of two distinct Sox9-EGFP-expressing intestinal stem cell populations during crypt regeneration after irradiation.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Activation of two distinct Sox ...... egeneration after irradiation.
@ast
Activation of two distinct Sox ...... egeneration after irradiation.
@en
type
label
Activation of two distinct Sox ...... egeneration after irradiation.
@ast
Activation of two distinct Sox ...... egeneration after irradiation.
@en
prefLabel
Activation of two distinct Sox ...... egeneration after irradiation.
@ast
Activation of two distinct Sox ...... egeneration after irradiation.
@en
P2093
P2860
P356
P1476
Activation of two distinct Sox ...... regeneration after irradiation
@en
P2093
Adrienne E Krebs
Amanda T Mah
Brooks P Scull
Jeffrey J Dehmer
Kirk McNaughton
M Agostina Santoro
P Kay Lund
Scott T Magness
P2860
P304
P356
10.1152/AJPGI.00519.2011
P577
2012-02-23T00:00:00Z