A predominant role of integrin alpha 4 in the spontaneous development of autoimmune diabetes in nonobese diabetic mice.
about
CCR10 expression is a common feature of circulating and mucosal epithelial tissue IgA Ab-secreting cellsRole of the intestinal tight junction modulator zonulin in the pathogenesis of type I diabetes in BB diabetic-prone rats.Newer insights into the pathogenesis of experimental autoimmune thyroiditis.Lymphocyte homing to bronchus-associated lymphoid tissue (BALT) is mediated by L-selectin/PNAd, alpha4beta1 integrin/VCAM-1, and LFA-1 adhesion pathwaysLoss of T cell CD98 H chain specifically ablates T cell clonal expansion and protects from autoimmunityEnteropathy precedes type 1 diabetes in the BB rat.Development of new strategies to prevent type 1 diabetes: the role of animal models.Harnessing memory adaptive regulatory T cells to control autoimmunity in type 1 diabetes.Can CD44 Be a Mediator of Cell Destruction? The Challenge of Type 1 Diabetes.Blocking adhesion molecules as therapy for multiple sclerosis: natalizumab.IL-7 uniquely maintains FoxP3(+) adaptive Treg cells that reverse diabetes in NOD mice via integrin-β7-dependent localizationGlucosamine Modulates T Cell Differentiation through Down-regulating N-Linked Glycosylation of CD25.Protein-mediated interactions of pancreatic islet cellsInhibition of submandibular and lacrimal gland infiltration in nonobese diabetic mice by transgenic expression of soluble TNF-receptor p55.The development of experimental autoimmune encephalomyelitis in the mouse requires alpha4-integrin but not alpha4beta7-integrin.Autoimmunity and inflammation: murine models and translational studies.Vitamin A and immune regulation: role of retinoic acid in gut-associated dendritic cell education, immune protection and tolerance.Integrin β7-mediated regulation of multiple myeloma cell adhesion, migration, and invasion.The discovery of natalizumab, a potent therapeutic for multiple sclerosis.IgG2a antibodies non-specifically delay the onset of diabetes in NOD mice.Islet beta-cell-specific T cells can use different homing mechanisms to infiltrate and destroy pancreatic islets.Insulitis in type 1 diabetes: a sticky problem.Clostridium butyricum CGMCC0313.1 Protects against Autoimmune Diabetes by Modulating Intestinal Immune Homeostasis and Inducing Pancreatic Regulatory T Cells.Serosal Cavities Contain Two Populations of Innate-like integrin α4highCD4+ T Cells, Integrin α4β1+α6β1+α4β7− and α4β1+α6β1−α4β7+ Cells.Fatalities in natalizumab treatment--a 'no go' for leukocyte recirculation approaches?Effects of VLA-4 antagonists in rat whole embryo culture.
P2860
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P2860
A predominant role of integrin alpha 4 in the spontaneous development of autoimmune diabetes in nonobese diabetic mice.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
A predominant role of integrin ...... tes in nonobese diabetic mice.
@ast
A predominant role of integrin ...... tes in nonobese diabetic mice.
@en
type
label
A predominant role of integrin ...... tes in nonobese diabetic mice.
@ast
A predominant role of integrin ...... tes in nonobese diabetic mice.
@en
prefLabel
A predominant role of integrin ...... tes in nonobese diabetic mice.
@ast
A predominant role of integrin ...... tes in nonobese diabetic mice.
@en
P2093
P2860
P356
P1476
A predominant role of integrin ...... tes in nonobese diabetic mice.
@en
P2093
P2860
P304
12604-12608
P356
10.1073/PNAS.91.26.12604
P407
P577
1994-12-01T00:00:00Z