Ceftriaxone ameliorates motor deficits and protects dopaminergic neurons in 6-hydroxydopamine-lesioned rats
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Recent Advance in the Relationship between Excitatory Amino Acid Transporters and Parkinson's DiseaseCurrent approaches to enhance glutamate transporter function and expressionExcess cerebral TNF causing glutamate excitotoxicity rationalizes treatment of neurodegenerative diseases and neurogenic pain by anti-TNF agentsAntibiotic treatment attenuates behavioral and neurochemical changes induced by exposure of rats to group a streptococcal antigenGlial Expression of the Caenorhabditis elegans Gene swip-10 Supports Glutamate Dependent Control of Extrasynaptic Dopamine SignalingModulating the Amyloidogenesis of α-SynucleinRegulation of glutamate transporter trafficking by Nedd4-2 in a Parkinson's disease model.Glutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease.α-Synuclein aggregation modulation: an emerging approach for the treatment of Parkinson's disease.Astrocyte Elevated Gene-1 (AEG-1) and the A(E)Ging HIV/AIDS-HAND.Ceftriaxone attenuates acute cocaine-evoked dopaminergic neurotransmission in the nucleus accumbens of the rat.Ceftriaxone blocks the polymerization of α-synuclein and exerts neuroprotective effects in vitroCeftriaxone increases glutamate uptake and reduces striatal tyrosine hydroxylase loss in 6-OHDA Parkinson's model.Cysteine mutagenesis reveals alternate proximity between transmembrane domain 2 and hairpin loop 1 of the glutamate transporter EAAT1.The effects of the β-lactam antibiotic, ceftriaxone, on forepaw stepping and L-DOPA-induced dyskinesia in a rodent model of Parkinson's disease.Antiallodynic Activity of Ceftriaxone and Clavulanic Acid in Acute Administration is Associated with Serum TNF-α Modulation and Activation of Dopaminergic and Opioidergic Systems.Ceftriaxone reduces L-dopa-induced dyskinesia severity in 6-hydroxydopamine parkinson's disease model.Glial loss of the metallo β-lactamase domain containing protein, SWIP-10, induces age- and glutamate-signaling dependent, dopamine neuron degeneration.Ceftriaxone Protects Astrocytes from MPP(+) via Suppression of NF-κB/JNK/c-Jun Signaling.Global untargeted serum metabolomic analyses nominate metabolic pathways responsive to loss of expression of the orphan metallo β-lactamase, MBLAC1
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P2860
Ceftriaxone ameliorates motor deficits and protects dopaminergic neurons in 6-hydroxydopamine-lesioned rats
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2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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2011年论文
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2011年论文
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name
Ceftriaxone ameliorates motor ...... -hydroxydopamine-lesioned rats
@ast
Ceftriaxone ameliorates motor ...... -hydroxydopamine-lesioned rats
@en
type
label
Ceftriaxone ameliorates motor ...... -hydroxydopamine-lesioned rats
@ast
Ceftriaxone ameliorates motor ...... -hydroxydopamine-lesioned rats
@en
prefLabel
Ceftriaxone ameliorates motor ...... -hydroxydopamine-lesioned rats
@ast
Ceftriaxone ameliorates motor ...... -hydroxydopamine-lesioned rats
@en
P2093
P2860
P356
P1476
Ceftriaxone ameliorates motor ...... -hydroxydopamine-lesioned rats
@en
P2093
P2860
P356
10.1021/CN200072H
P577
2011-10-26T00:00:00Z