Vascular hyporeactivity to vasoconstrictor agents and hemodynamic decompensation in hemorrhagic shock is mediated by nitric oxide - Wikidata - wikimedia - TriplyDB

Vascular hyporeactivity to vasoconstrictor agents and hemodynamic decompensation in hemorrhagic shock is mediated by nitric oxide

Vascular hyporeactivity to vasoconstrictor agents and hemodynamic decompensation in hemorrhagic shock is mediated by nitric oxide

http://www.wikidata.org/entity/Q36041762

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A critical review of pharmacological significance of Hydrogen Sulfide in hypertension Vasopressin in hemorrhagic shock: a systematic review and meta-analysis of randomized animal trials Tacrolimus suppresses tumour necrosis factor-alpha and protects against splanchnic artery occlusion shock Low dose nitrite enhances perfusion after fluid resuscitation from hemorrhagic shock.Role of nitric oxide in young patients with vasovagal syncope.Postsynaptic α1-Adrenergic Vasoconstriction Is Impaired in Young Patients With Vasovagal Syncope and Is Corrected by Nitric Oxide Synthase Inhibition An adequately robust early TNF-alpha response is a hallmark of survival following trauma/hemorrhage.The β-d-Endoglucuronidase Heparanase Is a Danger Molecule That Drives Systemic Inflammation and Correlates with Clinical Course after Open and Endovascular Thoracoabdominal Aortic Aneurysm Repair: Lessons Learnt from Mice and Men Early physiologic responses to hemorrhagic hypotension Trauma hemorrhagic shock-induced lung injury involves a gut-lymph-induced TLR4 pathway in mice Physiopathology of shock Co-induction of nitric oxide synthase and cyclo-oxygenase: interactions between nitric oxide and prostanoids.Attenuation of the induction of nitric oxide synthase by endogenous glucocorticoids accounts for endotoxin tolerance in vivo.Role of nitric oxide in inflammation and tissue injury during endotoxemia and hemorrhagic shock.Role of hydrogen sulphide in haemorrhagic shock in the rat: protective effect of inhibitors of hydrogen sulphide biosynthesis Methylene blue modulates transendothelial migration of peripheral blood cells.Adrenal dysfunction in portal hypertensive rats with acute hemorrhage 'Preconditioning' with low dose lipopolysaccharide aggravates the organ injury / dysfunction caused by hemorrhagic shock in rats Hemodynamic responses elicited by systemic injections of isotonic and hypertonic saline in hemorrhaged rats.Activation of MDL-1 (CLEC5A) on immature myeloid cells triggers lethal shock in mice Pathogenesis of malaria and clinically similar conditions.Beneficial effects and improved survival in rodent models of septic shock with S-methylisothiourea sulfate, a potent and selective inhibitor of inducible nitric oxide synthase Inhibition of IκB Kinase Attenuates the Organ Injury and Dysfunction Associated with Hemorrhagic Shock Transfused older stored red blood cells improve the clinical course and outcome in a canine lethal hemorrhage and reperfusion model Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.Inducible nitric oxide synthase contributes to immune dysfunction following trauma Essential role of induced nitric oxide in the initiation of the inflammatory response after hemorrhagic shock Ca(2+)-independent nitric oxide synthase activity in human lung after cardiopulmonary bypass.Beneficial haemodynamic effect of indomethacin during endotoxin shock in anaesthetized pigsputative involvement of nitric oxide?TGF beta 1 and TNF alpha potentiate nitric oxide production in astrocyte cultures by recruiting distinct subpopulations of cells to express NOS-2.Induction of nitric oxide release by MRC OX-44 (anti-CD53) through a protein kinase C-dependent pathway in rat macrophages.Endothelial NOS (NOS3) impairs myocardial function in developing sepsis.Iron chelates bind nitric oxide and decrease mortality in an experimental model of septic shock.Prolonged NO treatment decreases alpha-adrenoreceptor agonist responsiveness in porcine pulmonary artery due to persistent soluble guanylyl cyclase activation.Combined neuronal and inducible nitric oxide synthase inhibition in ovine acute lung injury Protective role of endogenous carbon monoxide in hepatic microcirculatory dysfunction after hemorrhagic shock in rats.Preservation of myocardial contractile function by aminoguanidine, a nitric oxide synthase inhibitors, in a rat model of hemorrhagic shock.Histological and Immunohistochemical Basis of the Effect of Aminoguanidine on Renal Changes Associated with Hemorrhagic Shock in a Rat Model Cardiogenic shock in ACS. Part 2: Role of mechanical circulatory support.Does vasopressor therapy have an indication in hemorrhagic shock?

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Vascular hyporeactivity to vasoconstrictor agents and hemodynamic decompensation in hemorrhagic shock is mediated by nitric oxide

http://www.wikidata.org/entity/Q36041762

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1993 nî lūn-bûn

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Proceedings of the National Academy of Sciences of the United States of America

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Vascular hyporeactivity to vas ...... ck is mediated by nitric oxide

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Mitchell JA

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Szabó C

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Thiemermann C

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Vane JR

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P2860

Isolation of nitric oxide synthetase, a calmodulin-requiring enzyme

Role of endothelium-derived nitric oxide in the regulation of blood pressure

Glucocorticoids inhibit the expression of an inducible, but not the constitutive, nitric oxide synthase in vascular endothelial cells.

Interleukin 1 induces prolonged L-arginine-dependent cyclic guanosine monophosphate and nitrite production in rat vascular smooth muscle cells

Activated murine macrophages secrete a metabolite of arginine with the bioactivity of endothelium-derived relaxing factor and the chemical reactivity of nitric oxide

Endothelium-dependent responses and their potential relevance to the distribution of blood flow during hemorrhagic shock.

An improved chromogenic substrate endotoxin assay for clinical use.

Mediators produced by the endothelial cell.

Vascular adrenergic interactions during hemorrhagic shock.

Characterization of nitric oxide synthases in non-adrenergic non-cholinergic nerve containing tissue from the rat anococcygeus muscle.

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267-271

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