Brain PPAR-γ promotes obesity and is required for the insulin-sensitizing effect of thiazolidinediones - Wikidata - wikimedia - TriplyDB

Brain PPAR-γ promotes obesity and is required for the insulin-sensitizing effect of thiazolidinediones

Brain PPAR-γ promotes obesity and is required for the insulin-sensitizing effect of thiazolidinediones

http://www.wikidata.org/entity/Q36050852

about

Neurological Effects of Bisphenol A and its Analogues Central nervous system mechanisms linking the consumption of palatable high-fat diets to the defense of greater adiposity Amorfrutins are potent antidiabetic dietary natural products GQ-16, a Novel Peroxisome Proliferator-activated Receptor (PPAR ) Ligand, Promotes Insulin Sensitization without Weight Gain Targeting tumor-associated macrophages to combat pancreatic cancer Pruning of the adipocyte peroxisome proliferator-activated receptor γ cistrome by hematopoietic master regulator PU.1 PPARγ in vagal neurons regulates high-fat diet induced thermogenesis A randomized, double blind, placebo-controlled trial of pioglitazone in combination with riluzole in amyotrophic lateral sclerosis Macrophage plasticity and polarization: in vivo veritas Leptin revisited: its mechanism of action and potential for treating diabetes.Macrophage polarization in inflammatory diseases.Glut4 expression defines an insulin-sensitive hypothalamic neuronal population Targeting Peroxisome Proliferator-Activated Receptors Using Thiazolidinediones: Strategy for Design of Novel Antidiabetic Drugs Local effect of IL-4 delivery on polyethylene particle induced osteolysis in the murine calvarium.Identification of Differentially Expressed Micrornas Associate with Glucose Metabolism in Different Organs of Blunt Snout Bream (Megalobrama amblycephala)Caffeine inhibits hypothalamic A1R to excite oxytocin neuron and ameliorate dietary obesity in mice Oxaliplatin neurotoxicity involves peroxisome alterations. PPARγ agonism as preventive pharmacological approach.Causal inference in longitudinal comparative effectiveness studies with repeated measures of a continuous intermediate variable.Interference with peroxisome proliferator-activated receptor-γ in vascular smooth muscle causes baroreflex impairment and autonomic dysfunction.PPARγ ablation sensitizes proopiomelanocortin neurons to leptin during high-fat feeding.Thiazolidinediones and the promise of insulin sensitization in type 2 diabetes.Neuron-specific deletion of peroxisome proliferator-activated receptor delta (PPARδ) in mice leads to increased susceptibility to diet-induced obesity.Neuroanatomical characterisation of the expression of the lipodystrophy and motor-neuropathy gene Bscl2 in adult mouse brain Wired on sugar: the role of the CNS in the regulation of glucose homeostasis.Evaluation of the synuclein-γ (SNCG) gene as a PPARγ target in murine adipocytes, dorsal root ganglia somatosensory neurons, and human adipose tissue.Neurotrophic factor-α1 prevents stress-induced depression through enhancement of neurogenesis and is activated by rosiglitazone.Regulation of hypothalamic neuropeptides gene expression in diet induced obesity resistant rats: possible targets for obesity prediction?Physiological responses to acute psychological stress are reduced by the PPARγ agonist rosiglitazone.PPARγ mRNA in the adult mouse hypothalamus: distribution and regulation in response to dietary challenges.Rosiglitazone-activated PPARγ induces neurotrophic factor-α1 transcription contributing to neuroprotection.Dual actions of a novel bifunctional compound to lower glucose in mice with diet-induced insulin resistance Obesogens, stem cells and the developmental programming of obesity.Naturally improving insulin resistance with amorfrutins.Idealized PPARγ-Based Therapies: Lessons from Bench and Bedside Peroxisome proliferation-associated control of reactive oxygen species sets melanocortin tone and feeding in diet-induced obesity GPR105 ablation prevents inflammation and improves insulin sensitivity in mice with diet-induced obesity Systemic PPARγ deletion causes severe disturbance in fluid homeostasis in mice.Coordinated Regulation of PPARγ Expression and Activity through Control of Chromatin Structure in Adipogenesis and Obesity.Rosiglitazone improves insulin sensitivity and baroreflex gain in rats with diet-induced obesity Hypophagia and metabolic adaptations in mice with defective ATGL-mediated lipolysis cause resistance to HFD-induced obesity.

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Brain PPAR-γ promotes obesity and is required for the insulin-sensitizing effect of thiazolidinediones

http://www.wikidata.org/entity/Q36050852

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2011 nî lūn-bûn

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2011年论文

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2011年论文

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2011年论文

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Brain PPAR-γ promotes obesity ...... g effect of thiazolidinediones

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Nature Medicine

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Brain PPAR-γ promotes obesity ...... g effect of thiazolidinediones

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David A Sarruf

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Gautam Bandyopadhyay

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Jiaur R Gayen

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Michael W Schwartz

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Min Lu

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Nicholas J Webster

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Sarah Nalbandian

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Shweta Sharma

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Sushil K Mahata

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WuQiang Fan

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P2860

Ablation of NF1 function in neurons induces abnormal development of cerebral cortex and reactive gliosis in the brain

Muscle-specific Pparg deletion causes insulin resistance

Macrophage-specific PPARgamma controls alternative activation and improves insulin resistance

Central nervous system control of food intake and body weight

A new antidiabetic compound attenuates inflammation and insulin resistance in Zucker diabetic fatty rats.

Identification of body fat mass as a major determinant of metabolic rate in mice.

Inducible nitric oxide synthase deficiency in myeloid cells does not prevent diet-induced insulin resistance

PPAR gamma and human metabolic disease

Adipose-specific peroxisome proliferator-activated receptor gamma knockout causes insulin resistance in fat and liver but not in muscle.

Macrophage PPAR gamma is required for normal skeletal muscle and hepatic insulin sensitivity and full antidiabetic effects of thiazolidinediones

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10.1038/NM.2332

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5

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17

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Saswata Talukdar

Jerrold M. Olefsky

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2011-05-01T00:00:00Z

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51088803

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1037960745

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