Reverse remodeling with left ventricular assist devices: a review of clinical, cellular, and molecular effects - Wikidata - wikimedia - TriplyDB

Reverse remodeling with left ventricular assist devices: a review of clinical, cellular, and molecular effects

Reverse remodeling with left ventricular assist devices: a review of clinical, cellular, and molecular effects

http://www.wikidata.org/entity/Q36065639

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Devices in the management of advanced, chronic heart failure Use of implantable cardioverter defibrillators in patients with left ventricular assist devices Small and large animal models in cardiac contraction research: advantages and disadvantages Human Ventricular Unloading Induces Cardiomyocyte Proliferation Pathophysiology of Heart Failure.Toward 3-D Echocardiographic Determination of Regional Myofiber Structure Assessment of myocardial viability and left ventricular function in patients supported by a left ventricular assist device Translation of Cardiac Myosin Activation with 2-deoxy-ATP to Treat Heart Failure via an Experimental Ribonucleotide Reductase-Based Gene Therapy Molecular and cellular basis of viable dysfunctional myocardium.Divergent mitochondrial biogenesis responses in human cardiomyopathy Mechanical unloading promotes myocardial energy recovery in human heart failure.Sildenafil prevents and reverses transverse-tubule remodeling and Ca(2+) handling dysfunction in right ventricle failure induced by pulmonary artery hypertension.MicroRNA Expression in Myocardial Tissue and Plasma of Patients with End-Stage Heart Failure during LVAD Support: Comparison of Continuous and Pulsatile Devices.Incomplete recovery of myocyte contractile function despite improvement of myocardial architecture with left ventricular assist device support.Long non-coding RNAs in heart failure: an obvious lnc.QSYQ Attenuates Oxidative Stress and Apoptosis Induced Heart Remodeling Rats through Different Subtypes of NADPH-Oxidase.Bridge to recovery: understanding the disconnect between clinical and biological outcomes.Thyroid hormone signalling is altered in response to physical training in patients with end-stage heart failure and mechanical assist devices: potential physiological consequences?Pathological ventricular remodeling: mechanisms: part 1 of 2 IL-33/ST2 pathway and classical cytokines in end-stage heart failure patients submitted to left ventricular assist device support: a paradoxic role for inflammatory mediators?Cellular electrophysiological abnormalities in dyssynchronous hearts and during CRT.Myocardial recovery and the failing heart: myth, magic, or molecular target?The transverse-axial tubular system of cardiomyocytes.Therapeutic implications of biomarkers in chronic heart failure.Molecular- and organelle-based predictive paradigm underlying recovery by left ventricular assist device support.Myocardial recovery: a focus on the impact of left ventricular assist devices.Clinical and Molecular Comparison of Pediatric and Adult Reverse Remodeling With Ventricular Assist Devices.Left ventricular assist device-induced reverse remodeling: it's not just about myocardial recovery.Myocardial recovery with mechanical circulatory support.Proteomics highlights decrease of matricellular proteins in left ventricular assist device therapy†.Current Clinical Applications and Next Steps for Cardiac Innervation Imaging.Cardiac Myosin Activation with Gene Therapy Produces Sustained Inotropic Effects and May Treat Heart Failure with Reduced Ejection Fraction Differential regulation of microRNAs in end-stage failing hearts is associated with left ventricular assist device unloading Heart failure in remission for more than 13 years after removal of a left ventricular assist device A computational model that predicts reverse growth in response to mechanical unloading The miRNA Expression Profile in Acute Myocardial Infarct Using Sheep Model with Left Ventricular Assist Device Unloading.Arrhythmogenic right ventricular cardiomyopathy: use of a left ventricular assist device as a bridge to transplantation?Myocardial fibrosis and pro-fibrotic markers in end-stage heart failure patients during continuous-flow left ventricular assist device support.A hole in the heart.

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P2860

Reverse remodeling with left ventricular assist devices: a review of clinical, cellular, and molecular effects

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Circulation: Heart Failure

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Amrut V Ambardekar

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P2860

PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

Downregulation of metabolic gene expression in failing human heart before and after mechanical unloading

Use of a continuous-flow device in patients awaiting heart transplantation

Myocardial expression of the arginine:glycine amidinotransferase gene is elevated in heart failure and normalized after recovery: potential implications for local creatine synthesis

Left ventricular assist device and drug therapy for the reversal of heart failure

Long-term use of a left ventricular assist device for end-stage heart failure.

Impact of mechanical unloading on microvasculature and associated central remodeling features of the failing human heart.

Reciprocal regulation of myocardial microRNAs and messenger RNA in human cardiomyopathy and reversal of the microRNA signature by biomechanical support.

Markers of autophagy are downregulated in failing human heart after mechanical unloading.

Myocardial insulin-like growth factor-I gene expression during recovery from heart failure after combined left ventricular assist device and clenbuterol therapy.

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