Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord. - Wikidata - wikimedia - TriplyDB

Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

http://www.wikidata.org/entity/Q36090234

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Circuits controlling vertebrate locomotion: moving in a new direction Serotonergic modulation of post-synaptic inhibition and locomotor alternating pattern in the spinal cord Depolarizing GABA/glycine synaptic events switch from excitation to inhibition during frequency increases.Dynamic spike thresholds during synaptic integration preserve and enhance temporal response properties in the avian cochlear nucleus.Neuronal Chloride Regulation via KCC2 Is Modulated through a GABAB Receptor Protein Complex.Immunolocalization of cation-chloride cotransporters in the developing and mature spinal cord of opossums, Monodelphis domestica Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.Contribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in mice.Robust tonic GABA currents can inhibit cell firing in mouse newborn neocortical pyramidal cells Sensory gating of an embryonic zebrafish interneuron during spontaneous motor behaviors.GABA depolarizes immature neurons and inhibits network activity in the neonatal neocortex in vivo.Modulation of inhibitory strength and kinetics facilitates regulation of persistent inward currents and motoneuron excitability following spinal cord injury Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.Using imaging and genetics in zebrafish to study developing spinal circuits in vivo The "mirror" estimate: an intuitive predictor of membrane polarization during extracellular stimulation GABAergic depolarization during early cortical development and implications for anticonvulsive therapy in neonates.Spatial and temporal dynamics in the ionic driving force for GABA(A) receptors.Maturation of the GABAergic transmission in normal and pathologic motoneurons.GABAergic Transmission during Brain Development: Multiple Effects at Multiple Stages.Short-term ionic plasticity at GABAergic synapses.Mild KCC2 Hypofunction Causes Inconspicuous Chloride Dysregulation that Degrades Neural Coding.Intracellular chloride concentration influences the GABAA receptor subunit composition.Cortical cells reveal APP as a new player in the regulation of GABAergic neurotransmission.Down-regulation of the potassium-chloride cotransporter KCC2 contributes to spasticity after spinal cord injury.Random stimulation of spider mechanosensory neurons reveals long-lasting excitation by GABA and muscimol.Developmental changes in the fidelity and short-term plasticity of GABAergic synapses in the neonatal rat dorsal horn.A Latent Propriospinal Network Can Restore Diaphragm Function after High Cervical Spinal Cord Injury.Retracing your footsteps: developmental insights to spinal network plasticity following injury.Glucose is an adequate energy substrate for the depolarizing action of GABA and glycine in the neonatal rat spinal cord in vitro.Effect of depolarizing GABA(A)-mediated membrane responses on excitability of Cajal-Retzius cells in the immature rat neocortex.[Calpain as a new therapeutic target for treating spasticity after a spinal cord injury].The KCC2 Cotransporter and Human Epilepsy: Getting Excited About Inhibition.Cystic fibrosis transmembrane conductance regulator modulates synaptic chloride homeostasis in motoneurons of the rat spinal cord during neonatal development.Incremental Embodied Chaotic Exploration of Self-Organized Motor Behaviors with Proprioceptor Adaptation

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P2860

Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

http://www.wikidata.org/entity/Q36090234

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

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Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

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type

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Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

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Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

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Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

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Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

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PNAS.0704832104

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Dual personality of GABA/glycine-mediated depolarizations in immature spinal cord.

@en

P2093

Céline Jean-Xavier

http://www.w3.org/2001/XMLSchema#string

Daniel Cattaert

http://www.w3.org/2001/XMLSchema#string

George Z Mentis

http://www.w3.org/2001/XMLSchema#string

Laurent Vinay

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Michael J O'Donovan

http://www.w3.org/2001/XMLSchema#string

P2860

Reduction of KCC2 expression and GABAA receptor-mediated excitation after in vivo axonal injury

Functional characterization of the neuronal-specific K-Cl cotransporter: implications for [K+]o regulation

The origin of spontaneous activity in developing networks of the vertebrate nervous system.

Spontaneous correlated activity in developing neural circuits.

Perinatal development of lumbar motoneurons and their inputs in the rat.

On the origin of interictal activity in human temporal lobe epilepsy in vitro.

Use-dependent shift from inhibitory to excitatory GABAA receptor action in SP-O interneurons in the rat hippocampal CA3 area.

Trans-synaptic shift in anion gradient in spinal lamina I neurons as a mechanism of neuropathic pain.

Developing networks play a similar melody.

Inhibitory postsynaptic potentials in lumbar motoneurons remain depolarizing after neonatal spinal cord transection in the rat.

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11477-11482

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10.1073/PNAS.0704832104

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27

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104

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2040923

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