The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
about
The myofibroblast: one function, multiple originsPeroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac FibrosisStem Cells and Progenitor Cells for Tissue-Engineered Solutions to Congenital Heart DefectsTissue factor, protease activated receptors and pathologic heart remodellingCross talk between cardiac myocytes and fibroblasts: from multiscale investigative approaches to mechanisms and functional consequencesMMI-0100 inhibits cardiac fibrosis in myocardial infarction by direct actions on cardiomyocytes and fibroblasts via MK2 inhibition.Size of the Ovulatory Follicle Dictates Spatial Differences in the Oviductal Transcriptome in CattleKlotho and phosphate are modulators of pathologic uremic cardiac remodeling.Regulators of G-protein signaling in the heart and their potential as therapeutic targets.The fibrosis-cell death axis in heart failureActivation of the intracellular renin-angiotensin system in cardiac fibroblasts by high glucose: role in extracellular matrix productionPharmacological inhibition of epsilon-protein kinase C attenuates cardiac fibrosis and dysfunction in hypertension-induced heart failureMeningeal norepinephrine produces headache behaviors in rats via actions both on dural afferents and fibroblastsWithaferin-A reduces type I collagen expression in vitro and inhibits development of myocardial fibrosis in vivoDownregulation of TGF-β Receptor-2 Expression and Signaling through Inhibition of Na/K-ATPaseCyclic nucleotide phosphodiesterase 1A: a key regulator of cardiac fibroblast activation and extracellular matrix remodeling in the heartAngiotensin II increases periostin expression via Ras/p38 MAPK/CREB and ERK1/2/TGF-β1 pathways in cardiac fibroblastsTransforming growth factor β1 induces the expression of collagen type I by DNA methylation in cardiac fibroblastsβ3 integrin in cardiac fibroblast is critical for extracellular matrix accumulation during pressure overload hypertrophy in mouseCardiac fibroblast: the renaissance cell.Cytokines regulate matrix metalloproteinases and migration in cardiac fibroblasts.Electrotonic myofibroblast-to-myocyte coupling increases propensity to reentrant arrhythmias in two-dimensional cardiac monolayers.PGS:Gelatin nanofibrous scaffolds with tunable mechanical and structural properties for engineering cardiac tissues.Direct intercellular communications dominate the interaction between adipose-derived MSCs and myofibroblasts against cardiac fibrosis.Developmental stage-dependent effects of cardiac fibroblasts on function of stem cell-derived engineered cardiac tissues.Matrine induces apoptosis in angiotensin II-stimulated hyperplasia of cardiac fibroblasts: effects on Bcl-2/Bax expression and caspase-3 activation.P21waf1/cip1/sdi1 as a central regulator of inducible smooth muscle actin expression and differentiation of cardiac fibroblasts to myofibroblasts.Characterization of multiple ion channels in cultured human cardiac fibroblasts.Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodelingBNIP3L promotes cardiac fibrosis in cardiac fibroblasts through [Ca2+]i-TGF-β-Smad2/3 pathway.Diabetes-induced alterations in the extracellular matrix and their impact on myocardial function.Enhanced expression and phosphorylation of Sirt7 activates smad2 and ERK signaling and promotes the cardiac fibrosis differentiation upon angiotensin-II stimulationProtease-activated receptor-1 contributes to cardiac remodeling and hypertrophyTemporal alterations in cardiac fibroblast function following induction of pressure overload.Antifibrotic activity of an inhibitor of histone deacetylases in DOCA-salt hypertensive ratsCardiac hypertrophy is not amplified by deletion of cGMP-dependent protein kinase I in cardiomyocytes.Pertussis toxin up-regulates angiotensin type 1 receptors through Toll-like receptor 4-mediated Rac activation.Stimulation of Adenosine A2B Receptor Inhibits Endothelin-1-Induced Cardiac Fibroblast Proliferation and α-Smooth Muscle Actin Synthesis Through the cAMP/Epac/PI3K/Akt-Signaling PathwayProteomic and genomic studies of non-alcoholic fatty liver disease--clues in the pathogenesisRemodeling of cardiac fibroblasts following myocardial infarction results in increased gap junction intercellular communication
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P2860
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
@ast
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
@en
type
label
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
@ast
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
@en
prefLabel
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
@ast
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
@en
P2093
P1476
The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.
@en
P2093
M Darren Mitchell
R Dale Brown
S Kelly Ambler
P304
P356
10.1146/ANNUREV.PHARMTOX.45.120403.095802
P50
P577
2005-01-01T00:00:00Z