Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy - Wikidata - wikimedia - TriplyDB

Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

http://www.wikidata.org/entity/Q33765289

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A novel protease-activated receptor-1 interactor, Bicaudal D1, regulates G protein signaling and internalization Tissue factor, protease activated receptors and pathologic heart remodelling Direct Thrombin Inhibitors Prevent Left Atrial Remodeling Associated With Heart Failure in Rats New approaches in small animal echocardiography: imaging the sounds of silence.Tissue factor and heart inflammation.Tissue factor pathway inhibitor overexpression inhibits hypoxia-induced pulmonary hypertension.Protease-activated receptor 2 deficiency reduces cardiac ischemia/reperfusion injury.PAR1-dependent COX-2/PGE2 production contributes to cell proliferation via EP2 receptors in primary human cardiomyocytes Cystamine preparations exhibit anticoagulant activity An immunohistochemical analysis of tissue thrombin expression in the human atria Protease activated receptor-2 contributes to heart failure.Protease-activated receptors and myocardial infarction Platelet specific promoters are insufficient to express protease activated receptor 1 (PAR1) transgene in mouse platelets.Complement component 3 is necessary to preserve myocardium and myocardial function in chronic myocardial infarction.Protease-activated receptor-2 modulates protease-activated receptor-1-driven neointimal hyperplasia.Effects of thrombin and thrombin receptor activation on cardiac function after acute myocardial infarction Humanizing the Protease-Activated Receptor (PAR) Expression Profile in Mouse Platelets by Knocking PAR1 into the Par3 Locus Reveals PAR1 Expression Is Not Tolerated in Mouse Platelets.Defining the cellular repertoire of GPCRs identifies a profibrotic role for the most highly expressed receptor, protease-activated receptor 1, in cardiac fibroblasts.Kallikrein-related peptidase 8 is expressed in myocardium and induces cardiac hypertrophy.Matrix metalloproteases and PAR1 activation.PAR-1 contributes to the innate immune response during viral infection.Cardiac tissue factor: roles in physiology and fibrosis.Protease-activated receptor 1 inhibition by SCH79797 attenuates left ventricular remodeling and profibrotic activities of cardiac fibroblasts.Characterisation of equine satellite cell transcriptomic profile response to β-hydroxy-β-methylbutyrate (HMB).Hydrogen Peroxide-Responsive Nanoparticle Reduces Myocardial Ischemia/Reperfusion Injury.Protease-activated receptor 4 deficiency offers cardioprotection after acute ischemia reperfusion injury.Thrombin-mediated activation of Akt signaling contributes to pulmonary vascular remodeling in pulmonary hypertension.The multivalent activity of the tissue factor-thrombin pathway in thrombotic and non-thrombotic disorders as a target for therapeutic intervention.Cardiac intercellular communication: are myocytes and fibroblasts fair-weather friends?Contributions of cardiomyocyte-cardiac fibroblast-immune cell interactions in heart failure development.Pleiotropic effects of factor Xa and thrombin: what to expect from novel anticoagulants.Novel mechanisms in the pathogenesis of atrial fibrillation: practical applications.Coagulation and non-coagulation effects of thrombin.Proteinases, Their Extracellular Targets, and Inflammatory Signaling.Protease-Activated Receptor 1 Contributes to Angiotensin II-Induced Cardiovascular Remodeling and Inflammation.G Protein-Coupled Receptor-G-Protein βγ-Subunit Signaling Mediates Renal Dysfunction and Fibrosis in Heart Failure.Factor Xa induces pro-inflammatory cytokine expression in RAW 264.7 macrophages via protease-activated receptor-2 activation.Argatroban Attenuates Diabetic Cardiomyopathy in Rats by Reducing Fibrosis, Inflammation, Apoptosis, and Protease-Activated Receptor Expression.A brief exposure to tryptase or thrombin potentiates fibrocyte differentiation in the presence of serum or serum amyloid p.β-adrenergic receptor stimulation transactivates protease-activated receptor 1 via matrix metalloproteinase 13 in cardiac cells.

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P2860

Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

http://www.wikidata.org/entity/Q33765289

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2007 nî lūn-bûn

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2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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name

Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

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Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

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Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

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Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

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Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

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Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy

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Burns C Blaxall

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Craig R Hampton

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Denise Spring

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Edward D Verrier

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Jun-ichi Abe

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Liam M Casey

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Matthias Kotzsch

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Michael Tencati

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Nigel Mackman

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Patricia Andrade-Gordon

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P2860

Activated MEK5 induces serial assembly of sarcomeres and eccentric cardiac hypertrophy.

Transient cardiac expression of constitutively active Galphaq leads to hypertrophy and dilated cardiomyopathy by calcineurin-dependent and independent pathways

Design, synthesis, and biological characterization of a peptide-mimetic antagonist for a tethered-ligand receptor

Thrombin signalling and protease-activated receptors

Stress pathways and heart failure.

Orchestration of coagulation protease signaling by tissue factor.

Inhibition of the tissue factor-thrombin pathway limits infarct size after myocardial ischemia-reperfusion injury by reducing inflammation.

Regulation of macrophage procoagulant responses by the tissue factor cytoplasmic domain in endotoxemia.

The cardiovascular actions of protease-activated receptors.

The cardiac fibroblast: therapeutic target in myocardial remodeling and failure.

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2298-2306

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10.1161/CIRCULATIONAHA.107.692764

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20

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116

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2007-10-29T00:00:00Z

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5878247

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17967980

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