Genome-wide profiles of H2AX and γ-H2AX differentiate endogenous and exogenous DNA damage hotspots in human cells. - Wikidata - wikimedia - TriplyDB

Genome-wide profiles of H2AX and γ-H2AX differentiate endogenous and exogenous DNA damage hotspots in human cells.

Genome-wide profiles of H2AX and γ-H2AX differentiate endogenous and exogenous DNA damage hotspots in human cells.

http://www.wikidata.org/entity/Q36107014

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Personalized synthetic lethality induced by targeting RAD52 in leukemias identified by gene mutation and expression profile.Laying a solid foundation for Manhattan--'setting the functional basis for the post-GWAS era'.Histone deacetylation critically determines T cell subset radiosensitivity.Canonical histone H2Ba and H2A.X dimerize in an opposite genomic localization to H2A.Z/H2B.Z dimers in Toxoplasma gondii.Activation of DNA damage response signaling by condensed chromatin.Genome-wide analysis of the chromatin composition of histone H2A and H3 variants in mouse embryonic stem cells Persistent DNA damage-induced premature senescence alters the functional features of human bone marrow mesenchymal stem cells.Advancing small-molecule-based chemical biology with next-generation sequencing technologies.CD8 Memory Cells Develop Unique DNA Repair Mechanisms Favoring Productive Division γ-H2AX Kinetic Profile in Mouse Lymphocytes Exposed to the Internal Emitters Cesium-137 and Strontium-90.Non-redundant Functions of ATM and DNA-PKcs in Response to DNA Double-Strand Breaks.Nucleotide-resolution DNA double-strand break mapping by next-generation sequencing.Dicer in action at replication-transcription collisions.Methylation of histone H4 lysine 20 by PR-Set7 ensures the integrity of late replicating sequence domains in Drosophila Genome-wide reorganization of histone H2AX toward particular fragile sites on cell activation.Uracil DNA glycosylase (UNG) loss enhances DNA double strand break formation in human cancer cells exposed to pemetrexed Lineage-Specific Genes Are Prominent DNA Damage Hotspots during Leukemic Transformation of B Cell Precursors.Organizing the genome with H2A histone variants.Mutation of cancer driver MLL2 results in transcription stress and genome instability.LORD-Q: a long-run real-time PCR-based DNA-damage quantification method for nuclear and mitochondrial genome analysis.Genome maintenance in the context of 4D chromatin condensation.Greater Than the Sum of Parts: Complexity of the Dynamic Epigenome.Corrupting the DNA damage response: a critical role for Rad52 in tumor cell survival Hot spots of DNA double-strand breaks in human rDNA units are produced in vivo Histone H2AX Is Involved in FoxO3a-Mediated Transcriptional Responses to Ionizing Radiation to Maintain Genome Stability.Genome Instability and γH2AX.Loss of nuclear PTEN in HCV-infected human hepatocytes.Evidence of reduced recombination rate in human regulatory domains Molecular characterization of DNA double strand breaks with tip-enhanced Raman scattering.Reading chromatin signatures after DNA double-strand breaks.Genome-wide multi-parametric analysis of H2AX or γH2AX distributions during ionizing radiation-induced DNA damage response.Designing Blood-Brain Barrier Models Reproducing Alterations in Brain Aging

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Genome-wide profiles of H2AX and γ-H2AX differentiate endogenous and exogenous DNA damage hotspots in human cells.

http://www.wikidata.org/entity/Q36107014

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2012 nî lūn-bûn

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name

Genome-wide profiles of H2AX a ...... amage hotspots in human cells.

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Genome-wide profiles of H2AX a ...... amage hotspots in human cells.

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Eun-Suk Kang

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Heun-Sik Lee

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Hye Jin Shon

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Jae Ho Lee

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Jin Sung Kim

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Jung Kyoon Choi

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Jung Kyu Kim

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Jungmin Seo

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Kartiki Vasant Desai

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Nur Lina Mohd Salleh

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P2860

Model-based analysis of ChIP-Seq (MACS)

WSTF regulates the H2A.X DNA damage response via a novel tyrosine kinase activity

Tyrosine dephosphorylation of H2AX modulates apoptosis and survival decisions

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

Telomeres avoid end detection by severing the checkpoint signal transduction pathway

Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis

Dynamic regulation of nucleosome positioning in the human genome

Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints

Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication

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