Semaphorin-3C signals through Neuropilin-1 and PlexinD1 receptors to inhibit pathological angiogenesis.
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Sustained inflammation after pericyte depletion induces irreversible blood-retina barrier breakdownThe Neuropilin-1 Inhibitor, ATWLPPR Peptide, Prevents Experimental Diabetes-Induced Retinal Injury by Preserving Vascular Integrity and Decreasing Oxidative Stress.A Sema3C Mutant Resistant to Cleavage by Furin (FR-Sema3C) Inhibits Choroidal Neovascularization.Neuropilin 1 Involvement in Choroidal and Retinal Neovascularisation.Dry age-related macular degeneration like pathology in aged 5XFAD mice: Ultrastructure and microarray analysis.Neuropilin Functions as an Essential Cell Surface Receptor.Defective Angiogenesis and Intraretinal Bleeding in Mouse Models With Disrupted Inner Retinal LaminationSemaphorin 3C Released from a Biocompatible Hydrogel Guides and Promotes Axonal Growth of Rodent and Human Dopaminergic NeuronsMiR-148a functions to suppress metastasis and serves as a prognostic indicator in triple-negative breast cancer.Current drug design to target the Semaphorin/Neuropilin/Plexin complexes.Class 3 semaphorins in cardiovascular development.RORα modulates semaphorin 3E transcription and neurovascular interaction in pathological retinal angiogenesis.PlexinD1 is required for proper patterning of the periocular vascular network and for the establishment of corneal avascularity during avian ocular development.Androgen receptor transcriptionally regulates semaphorin 3C in a GATA2-dependent manner.The role of the semaphorins in cancer.Review of the mechanisms and therapeutic avenues for retinal and choroidal vascular dysfunctions in retinopathy of prematurity.Advances in understanding and management of retinopathy of prematurity.Semaphorin 3 C drives epithelial-to-mesenchymal transition, invasiveness, and stem-like characteristics in prostate cells.Retinopathy of prematurity: inflammation, choroidal degeneration, and novel promising therapeutic strategies.Sema3d controls collective endothelial cell migration by distinct mechanisms via Nrp1 and PlxnD1.Bad vessels beware! Semaphorins will sort you out!Higher Aminopeptidase Activity Determined by Electroosmotic Push-Pull Perfusion Contributes to Selective Vulnerability of the Hippocampal CA1 Region to Oxygen Glucose Deprivation.SEMA3C drives cancer growth by transactivating multiple receptor tyrosine kinases via Plexin B1.Bacterial Cellulose Shifts Transcriptome and Proteome of Cultured Endothelial Cells Towards Native Differentiation.MPDZ promotes DLL4-induced Notch signaling during angiogenesis.Semaphorin 3C and Its Receptors in Cancer and Cancer Stem-Like Cells.Role of Semaphorin Signaling During Cardiovascular Development.
P2860
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P2860
Semaphorin-3C signals through Neuropilin-1 and PlexinD1 receptors to inhibit pathological angiogenesis.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Semaphorin-3C signals through ...... bit pathological angiogenesis.
@ast
Semaphorin-3C signals through ...... bit pathological angiogenesis.
@en
type
label
Semaphorin-3C signals through ...... bit pathological angiogenesis.
@ast
Semaphorin-3C signals through ...... bit pathological angiogenesis.
@en
prefLabel
Semaphorin-3C signals through ...... bit pathological angiogenesis.
@ast
Semaphorin-3C signals through ...... bit pathological angiogenesis.
@en
P2093
P2860
P356
P1476
Semaphorin-3C signals through ...... ibit pathological angiogenesis
@en
P2093
Andreas Fischer
Fabian Tetzlaff
Wan-Jen Yang
P2860
P304
P356
10.15252/EMMM.201404922
P577
2015-10-01T00:00:00Z