Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
about
Mechanisms of epithelial wound detectionStanniocalcin-1 regulates extracellular ATP-induced calcium waves in human epithelial cancer cells by stimulating ATP release from bystander cellsProgress in corneal wound healingP2Y2 nucleotide receptor activation enhances the aggregation and self-organization of dispersed salivary epithelial cells.Hypoxia-induced changes in Ca(2+) mobilization and protein phosphorylation implicated in impaired wound healing.Uridine 5'-triphosphate promotes in vitro Schwannoma cell migration through matrix metalloproteinase-2 activationThe P2Y2 receptor mediates the epithelial injury response and cell migration.Corneal epithelium expresses a variant of P2X(7) receptor in health and diseaseArousal of cancer-associated stroma: overexpression of palladin activates fibroblasts to promote tumor invasion.Communication between corneal epithelial cells and trigeminal neurons is facilitated by purinergic (P2) and glutamatergic receptors.New insights in wound response and repair of epithelium.Physiological impact of abnormal lipoxin A₄ production on cystic fibrosis airway epithelium and therapeutic potential.A comparison of epithelial-to-mesenchymal transition and re-epithelializationInjury and nucleotides induce phosphorylation of epidermal growth factor receptor: MMP and HB-EGF dependent pathway.Regulation by P2X7: epithelial migration and stromal organization in the cornea.Nucleotide receptors as targets in the pharmacological enhancement of dermal wound healing.Palladin regulation of the actin structures needed for cancer invasion.Purinergic Signaling in Corneal Wound Healing: A Tale of 2 Receptors.Purinoreceptor P2X7 Regulation of Ca(2+) Mobilization and Cytoskeletal Rearrangement Is Required for Corneal Reepithelialization after Injury.Extracellular matrix stiffness modulates VEGF calcium signaling in endothelial cells: individual cell and population analysisHB-EGF synthesis and release induced by cholesterol depletion of human epidermal keratinocytes is controlled by extracellular ATP and involves both p38 and ERK1/2 signaling pathways.Epithelial wounds induce differential phosphorylation changes in response to purinergic and EGF receptor activation.Multiple Mechanisms Drive Calcium Signal Dynamics around Laser-Induced Epithelial Wounds.P2Y2 Nucleotide Receptor Prompts Human Cardiac Progenitor Cell Activation by Modulating Hippo Signaling.
P2860
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P2860
Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
@ast
Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
@en
type
label
Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
@ast
Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
@en
prefLabel
Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
@ast
Tri-nucleotide receptors play a critical role in epithelial cell wound repair.
@en
P2860
P1476
Tri-nucleotide receptors play a critical role in epithelial cell wound repair
@en
P2093
Ilene Weinger
Veronica E Klepeis
P2860
P2888
P304
P356
10.1007/S11302-005-8132-6
P577
2005-07-29T00:00:00Z