Aldosterone deficiency and mineralocorticoid receptor antagonism prevent angiotensin II-induced cardiac, renal, and vascular injury.
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Salt Loading Promotes Kidney Injury via Fibrosis in Young Female Ren2 Rats.Mechanism of salt-sensitive hypertension: focus on adrenal and sympathetic nervous systemsMineralocorticoid Receptor Signaling as a Therapeutic Target for Renal and Cardiac Fibrosis.Estrogen receptor inhibits mineralocorticoid receptor transcriptional regulatory function.Obesity in kidney disease: A heavyweight opponent.Effects of aldosterone on insulin sensitivity and secretion.Mineralocorticoid receptors in vascular disease: connecting molecular pathways to clinical implications.Effects of low-dose spironolactone combined with irbesartan on cardiac hypertrophy induced by pressure overload in rats.Role of epidermal growth factor receptor and endoplasmic reticulum stress in vascular remodeling induced by angiotensin II.ANG II-induced hypertension in the VCD mouse model of menopause is prevented by estrogen replacement during perimenopause.Aldosterone in vascular and metabolic dysfunction.Novel drugs and intervention strategies for the treatment of chronic kidney disease.Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.Glucocorticoids and renal Na+ transport: implications for hypertension and salt sensitivity.Role of Rac1-mineralocorticoid-receptor signalling in renal and cardiac disease.Changing glucocorticoid action: 11β-hydroxysteroid dehydrogenase type 1 in acute and chronic inflammation.Smooth muscle cell mineralocorticoid receptors: role in vascular function and contribution to cardiovascular disease.Activation of mineralocorticoid receptor in salt-sensitive hypertension.Crosstalk between peroxisome proliferator-activated receptor-γ and mineralcorticoid receptor in TNF-α activated renal tubular cell.Chronic hyperaldosteronism in cryptochrome-null mice induces high-salt- and blood pressure-independent kidney damage in mice.Activation of cardiac renin-angiotensin system and plasminogen activator inhibitor-1 gene expressions in oral contraceptive-induced cardiometabolic disorder.Deletion of the EGF receptor in vascular smooth muscle cells prevents chronic angiotensin II-induced arterial wall stiffening and media thickening.Renin-angiotensin system transgenic mouse model recapitulates pathophysiology similar to human preeclampsia with renal injury that may be mediated through VEGF.Smooth muscle cell-specific Hif-1α deficiency suppresses angiotensin II-induced vascular remodelling in mice.Inhibitory effect of D3 dopamine receptor on migration of vascular smooth muscle cells induced by synergistic effect of angiotensin II and aldosterone.
P2860
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P2860
Aldosterone deficiency and mineralocorticoid receptor antagonism prevent angiotensin II-induced cardiac, renal, and vascular injury.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Aldosterone deficiency and min ...... c, renal, and vascular injury.
@ast
Aldosterone deficiency and min ...... c, renal, and vascular injury.
@en
type
label
Aldosterone deficiency and min ...... c, renal, and vascular injury.
@ast
Aldosterone deficiency and min ...... c, renal, and vascular injury.
@en
prefLabel
Aldosterone deficiency and min ...... c, renal, and vascular injury.
@ast
Aldosterone deficiency and min ...... c, renal, and vascular injury.
@en
P2093
P2860
P356
P1433
P1476
Aldosterone deficiency and min ...... ac, renal, and vascular injury
@en
P2093
Agnes B Fogo
Hyung-Suk Kim
Nancy J Brown
Pengcheng Luo
Samuel E Cohen
Zuofei Wang
P2860
P2888
P304
P356
10.1038/KI.2012.170
P407
P577
2012-05-23T00:00:00Z