Glucocorticoids and renal Na+ transport: implications for hypertension and salt sensitivity. - Wikidata - wikimedia - TriplyDB

Glucocorticoids and renal Na+ transport: implications for hypertension and salt sensitivity.

Glucocorticoids and renal Na+ transport: implications for hypertension and salt sensitivity.

http://www.wikidata.org/entity/Q37727017

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Pressure natriuresis and the renal control of arterial blood pressure Epigenetic Pathways in Human Disease: The Impact of DNA Methylation on Stress-Related Pathogenesis and Current Challenges in Biomarker Development.Hypertrophy in the Distal Convoluted Tubule of an 11β-Hydroxysteroid Dehydrogenase Type 2 Knockout Model.Glucocorticoids Induce Nondipping Blood Pressure by Activating the Thiazide-Sensitive Cotransporter.Hydration status, drug interactions, and determinants in a Spanish elderly population: a pilot study.Dexamethasone and insulin activate serum and glucocorticoid-inducible kinase 1 (SGK1) via different molecular mechanisms in cortical collecting duct cells.11β-Hydroxysteroid Dehydrogenases and Hypertension in the Metabolic Syndrome.The interstitium conducts extrarenal storage of sodium and represents a third compartment essential for extracellular volume and blood pressure homeostasis.Genomic and rapid effects of aldosterone: what we know and do not know thus far.Prednisone lowers serum uric acid levels in patients with decompensated heart failure by increasing renal uric acid clearance.

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Glucocorticoids and renal Na+ transport: implications for hypertension and salt sensitivity.

http://www.wikidata.org/entity/Q37727017

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 17 February 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Glucocorticoids and renal Na+ ...... rtension and salt sensitivity.

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Glucocorticoids and renal Na+ ...... rtension and salt sensitivity.

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Glucocorticoids and renal Na+ ...... rtension and salt sensitivity.

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Glucocorticoids and renal Na+ ...... rtension and salt sensitivity.

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Glucocorticoids and renal Na+ ...... rtension and salt sensitivity.

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Glucocorticoids and renal Na+ ...... rtension and salt sensitivity.

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JPHYSIOL.2013.267609

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The Journal of Physiology

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Glucocorticoids and renal Na+ ...... rtension and salt sensitivity.

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Jessica R Ivy

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Matthew A Bailey

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Creative Commons Attribution 3.0 Unported

P2860

Cloning of human mineralocorticoid receptor complementary DNA: structural and functional kinship with the glucocorticoid receptor

A switch in the mechanism of hypertension in the syndrome of apparent mineralocorticoid excess

An SGK1 site in WNK4 regulates Na+ channel and K+ channel activity and has implications for aldosterone signaling and K+ homeostasis

Hypertension in the syndrome of apparent mineralocorticoid excess due to mutation of the 11 beta-hydroxysteroid dehydrogenase type 2 gene

Mineralocorticoid receptor knockout mice: pathophysiology of Na+ metabolism

Hexose-6-phosphate dehydrogenase and 11beta-hydroxysteroid dehydrogenase-1 tissue distribution in the rat

Mineralocorticoid effects in the kidney: correlation between alphaENaC, GILZ, and Sgk-1 mRNA expression and urinary excretion of Na+ and K+

Renal Na/H exchanger NHE-3 and Na-PO4 cotransporter NaPi-2 protein expression in glucocorticoid excess and deficient states

Serum and glucocorticoid-regulated kinase modulates Nedd4-2-mediated inhibition of the epithelial Na+ channel.

The glucocorticoid receptor in the distal nephron is not necessary for the development or maintenance of dexamethasone-induced hypertension

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