Ursodeoxycholic Acid (UDCA) Exerts Anti-Atherogenic Effects by Inhibiting Endoplasmic Reticulum (ER) Stress Induced by Disturbed Flow.

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Calcium signalling and ER stress in insulin resistance and atherosclerosis.Neuroprotective Effects of Protein Tyrosine Phosphatase 1B Inhibition against ER Stress-Induced Toxicity.

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Ursodeoxycholic Acid (UDCA) Exerts Anti-Atherogenic Effects by Inhibiting Endoplasmic Reticulum (ER) Stress Induced by Disturbed Flow.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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Ursodeoxycholic Acid (UDCA) Ex ...... ess Induced by Disturbed Flow.

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Ursodeoxycholic Acid (UDCA) Ex ...... ess Induced by Disturbed Flow.

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Ursodeoxycholic Acid (UDCA) Ex ...... ess Induced by Disturbed Flow.

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Ursodeoxycholic Acid (UDCA) Ex ...... ess Induced by Disturbed Flow.

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Ursodeoxycholic Acid (UDCA) Ex ...... ess Induced by Disturbed Flow.

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Ursodeoxycholic Acid (UDCA) Ex ...... ess Induced by Disturbed Flow.

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P1476

Ursodeoxycholic Acid (UDCA) Ex ...... ess Induced by Disturbed Flow.

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P2093

Jihwa Chung

http://www.w3.org/2001/XMLSchema#string

Kihwan Kwon

http://www.w3.org/2001/XMLSchema#string

Kyoung Hwa Kim

http://www.w3.org/2001/XMLSchema#string

Seok Cheol Lee

http://www.w3.org/2001/XMLSchema#string

Shung Hyun An

http://www.w3.org/2001/XMLSchema#string

P2860

Protein kinase D-dependent phosphorylation and nuclear export of histone deacetylase 5 mediates vascular endothelial growth factor-induced gene expression and angiogenesis

Endoplasmic reticulum stress and atherosclerosis

CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum

IRE1 signaling affects cell fate during the unfolded protein response

GRP78 upregulation by atheroprone shear stress via p38-, alpha2beta1-dependent mechanism in endothelial cells.

Reduced apoptosis and plaque necrosis in advanced atherosclerotic lesions of Apoe-/- and Ldlr-/- mice lacking CHOP.

The role of endoplasmic reticulum stress in the progression of atherosclerosis.

The role of shear stress in the pathogenesis of atherosclerosis.

Coexisting proinflammatory and antioxidative endothelial transcription profiles in a disturbed flow region of the adult porcine aorta.

P304

851-858

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scholarly article

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10.14348/MOLCELLS.2015.0094

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2015-10-07T00:00:00Z

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282659353

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26442866

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atherosclerosis

endoplasmic reticulum

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4625066

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Ursodeoxycholic Acid (UDCA) Exerts Anti-Atherogenic Effects by Inhibiting Endoplasmic Reticulum (ER) Stress Induced by Disturbed Flow.

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Ursodeoxycholic Acid (UDCA) Exerts Anti-Atherogenic Effects by Inhibiting Endoplasmic Reticulum (ER) Stress Induced by Disturbed Flow.

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