Type I interferon signaling is required for activation of the inflammasome during Francisella infection.
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MiR-155 induction by F. novicida but not the virulent F. tularensis results in SHIP down-regulation and enhanced pro-inflammatory cytokine responseImmune evasion and recognition of the syphilis spirochete in blood and skin of secondary syphilis patients: two immunologically distinct compartmentsPathogen recognition and activation of the innate immune response in zebrafishIFI16 protein mediates the anti-inflammatory actions of the type-I interferons through suppression of activation of caspase-1 by inflammasomesRIG-I-dependent sensing of poly(dA:dT) through the induction of an RNA polymerase III-transcribed RNA intermediateAIM2 recognizes cytosolic dsDNA and forms a caspase-1-activating inflammasome with ASCc-di-AMP secreted by intracellular Listeria monocytogenes activates a host type I interferon responseA host type I interferon response is induced by cytosolic sensing of the bacterial second messenger cyclic-di-GMPImmunomodulatory functions of type I interferonsMycobacterium tuberculosis and the host cell inflammasome: a complex relationshipMolecular mechanisms of inflammasome activation during microbial infectionsRegulation of inflammasome signalingAn Update on PYRIN Domain-Containing Pattern Recognition Receptors: From Immunity to PathologySubversion of host recognition and defense systems by Francisella sppFrancisella tularensis IglG Belongs to a Novel Family of PAAR-Like T6SS Proteins and Harbors a Unique N-terminal Extension Required for VirulenceThe inflammasome: an emerging therapeutic oncotarget for cancer preventionBacterial recognition pathways that lead to inflammasome activationEvasion of inflammasome activation by microbial pathogensRole of the HIN domain in regulation of innate immune responsesMicroarray analysis of human monocytes infected with Francisella tularensis identifies new targets of host response subversionCutting edge: Mycobacterium tuberculosis but not nonvirulent mycobacteria inhibits IFN-β and AIM2 inflammasome-dependent IL-1β production via its ESX-1 secretion systemInterferon-induced guanylate-binding proteins in inflammasome activation and host defenseThe AIM2 inflammasome is essential for host defense against cytosolic bacteria and DNA virusesPyroptosis: host cell death and inflammationThe AIM2 inflammasome is critical for innate immunity to Francisella tularensisMonophosphoryl Lipid A Enhances Efficacy of a Francisella tularensis LVS-Catanionic Nanoparticle Subunit Vaccine against F. tularensis Schu S4 Challenge by Augmenting both Humoral and Cellular Immunity.Characterization of Francisella tularensis Schu S4 defined mutants as live-attenuated vaccine candidatesNovel catanionic surfactant vesicle vaccines protect against Francisella tularensis LVS and confer significant partial protection against F. tularensis Schu S4 strainIdentification of host cytosolic sensors and bacterial factors regulating the type I interferon response to Legionella pneumophilaInnate immune recognition of Yersinia pseudotuberculosis type III secretion.Distinct TLR- and NLR-mediated transcriptional responses to an intracellular pathogen.NOD2, RIP2 and IRF5 play a critical role in the type I interferon response to Mycobacterium tuberculosis.Streptococcus pneumoniae nasopharyngeal colonization induces type I interferons and interferon-induced gene expression.The inflammasomes.Type I interferon induction is detrimental during infection with the Whipple's disease bacterium, Tropheryma whippleiType I interferon signaling regulates activation of the absent in melanoma 2 inflammasome during Streptococcus pneumoniae infection.Phagosomal retention of Francisella tularensis results in TIRAP/Mal-independent TLR2 signaling.Getting "Inside" Type I IFNs: Type I IFNs in Intracellular Bacterial InfectionsA tolC mutant of Francisella tularensis is hypercytotoxic compared to the wild type and elicits increased proinflammatory responses from host cells.Rational design of a plasmid origin that replicates efficiently in both gram-positive and gram-negative bacteria
P2860
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P2860
Type I interferon signaling is required for activation of the inflammasome during Francisella infection.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Type I interferon signaling is ...... during Francisella infection.
@ast
Type I interferon signaling is ...... during Francisella infection.
@en
type
label
Type I interferon signaling is ...... during Francisella infection.
@ast
Type I interferon signaling is ...... during Francisella infection.
@en
prefLabel
Type I interferon signaling is ...... during Francisella infection.
@ast
Type I interferon signaling is ...... during Francisella infection.
@en
P2093
P2860
P356
P1476
Type I interferon signaling is ...... during Francisella infection.
@en
P2093
Anna Brotcke
David S Weiss
Denise M Monack
Lucinda J Thompson
Thomas Henry
P2860
P304
P356
10.1084/JEM.20062665
P407
P577
2007-04-23T00:00:00Z