Quantitation of intracellular NAD(P)H can monitor an imbalance of DNA single strand break repair in base excision repair deficient cells in real time.
about
Senataxin, defective in ataxia oculomotor apraxia type 2, is involved in the defense against oxidative DNA damageChk2-dependent phosphorylation of XRCC1 in the DNA damage response promotes base excision repairInterplay between DNA polymerases beta and lambda in repair of oxidation DNA damage in chicken DT40 cellsXRCC1 is required for DNA single-strand break repair in human cells.Tumor growth is suppressed in mice expressing a truncated XRCC1 proteinApoptosis induction by 13-acetoxyrolandrolide through the mitochondrial intrinsic pathway.iNOS-derived nitric oxide promotes glycolysis by inducing pyruvate kinase M2 nuclear translocation in ovarian cancerAccumulation of true single strand breaks and AP sites in base excision repair deficient cellsSWI/SNF complexes are required for full activation of the DNA-damage response.The impact of cyclin-dependent kinase 5 depletion on poly(ADP-ribose) polymerase activity and responses to radiationVertebrate POLQ and POLbeta cooperate in base excision repair of oxidative DNA damage.N-methylpurine DNA glycosylase inhibits p53-mediated cell cycle arrest and coordinates with p53 to determine sensitivity to alkylating agents.Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) induces cancer cell senescence by interacting with telomerase RNA component.Nitric oxide is a positive regulator of the Warburg effect in ovarian cancer cellsAmbiguine I Isonitrile from Fischerella ambigua Induces Caspase-Independent Cell Death in MCF-7 Hormone Dependent Breast Cancer Cells.Enhancement of synthetic lethality via combinations of ABT-888, a PARP inhibitor, and carboplatin in vitro and in vivo using BRCA1 and BRCA2 isogenic models.JWA regulates XRCC1 and functions as a novel base excision repair protein in oxidative-stress-induced DNA single-strand breaks.PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination.PCB-induced endothelial cell dysfunction: role of poly(ADP-ribose) polymerase.Energy use efficiency is characterized by an epigenetic component that can be directed through artificial selection to increase yield.Cellular radiosensitivity: how much better do we understand it?Application of open-access databases to determine functional connectivity between resveratrol-binding protein QR2 and colorectal carcinoma.Selection for Improved Energy Use Efficiency and Drought Tolerance in Canola Results in Distinct Transcriptome and Epigenome Changes.Cells deficient in PARP-1 show an accelerated accumulation of DNA single strand breaks, but not AP sites, over the PARP-1-proficient cells exposed to MMS.ARF induction in response to DNA strand breaks is regulated by PARP1.ALC1/CHD1L, a chromatin-remodeling enzyme, is required for efficient base excision repair.MNNG-induced cell death is controlled by interactions between PARP-1, poly(ADP-ribose) glycohydrolase, and XRCC1.
P2860
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P2860
Quantitation of intracellular NAD(P)H can monitor an imbalance of DNA single strand break repair in base excision repair deficient cells in real time.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Quantitation of intracellular ...... deficient cells in real time.
@ast
Quantitation of intracellular ...... deficient cells in real time.
@en
type
label
Quantitation of intracellular ...... deficient cells in real time.
@ast
Quantitation of intracellular ...... deficient cells in real time.
@en
prefLabel
Quantitation of intracellular ...... deficient cells in real time.
@ast
Quantitation of intracellular ...... deficient cells in real time.
@en
P2093
P2860
P356
P1476
Quantitation of intracellular ...... deficient cells in real time.
@en
P2093
Gilbert de Murcia
James A Swenberg
Jun Nakamura
Keith W Caldecott
Shoji Asakura
Susan D Hester
P2860
P356
10.1093/NAR/GNG105
P407
P577
2003-09-01T00:00:00Z