Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
about
SMUG1 is able to excise uracil from immunoglobulin genes: insight into mutation versus repairHuman base excision repair enzymes apurinic/apyrimidinic endonuclease1 (APE1), DNA polymerase beta and poly(ADP-ribose) polymerase 1: interplay between strand-displacement DNA synthesis and proofreading exonuclease activityFormamidopyrimidine-DNA glycosylase enhances arsenic-induced DNA strand breaks in PHA-stimulated and unstimulated human lymphocytes.The APOBEC3 family of retroelement restriction factorsAID mutates E. coli suggesting a DNA deamination mechanism for antibody diversificationhMYH cell cycle-dependent expression, subcellular localization and association with replication foci: evidence suggesting replication-coupled repair of adenine:8-oxoguanine mispairsDeletion of the nucleotide excision repair gene Ercc1 reduces immunoglobulin class switching and alters mutations near switch recombination junctionsA quantitative model of human DNA base excision repair. I. Mechanistic insights.A New Family of HEAT-Like Repeat Proteins Lacking a Critical Substrate Recognition Motif Present in Related DNA GlycosylasesRegulation of DNA repair fidelity by molecular checkpoints: "gates" in DNA polymerase beta's substrate selectionHuman APE2 protein is mostly localized in the nuclei and to some extent in the mitochondria, while nuclear APE2 is partly associated with proliferating cell nuclear antigenAcetylation regulates WRN catalytic activities and affects base excision DNA repair.Differential requirement of Srs2 helicase and Rad51 displacement activities in replication of hairpin-forming CAG/CTG repeats.Functional characterization of Ape1 variants identified in the human populationFolate deficiency provides protection against colon carcinogenesis in DNA polymerase beta haploinsufficient miceMbd4 inactivation increases Cright-arrowT transition mutations and promotes gastrointestinal tumor formation.The Werner syndrome protein stimulates DNA polymerase beta strand displacement synthesis via its helicase activity.Recent advances in the structural mechanisms of DNA glycosylasesSecuring genome stability by orchestrating DNA repair: removal of radiation-induced clustered lesions in DNA.In silico studies of the African swine fever virus DNA polymerase X support an induced-fit mechanismClass-switch recombination: after the dawn of AID.The DNA damage checkpoint and human cancer.Polymorphisms of DNA repair genes XRCC1 and XRCC3, interaction with environmental exposure and risk of chronic gastritis and gastric cancerMammalian Base Excision Repair: Functional Partnership between PARP-1 and APE1 in AP-Site Repair.Vertebrate POLQ and POLbeta cooperate in base excision repair of oxidative DNA damage.Influence of physiologic folate deficiency on human papillomavirus type 16 (HPV16)-harboring human keratinocytes in vitro and in vivo.Mobile telephones and cancer--a review of epidemiological evidence.Attenuation of DNA polymerase beta-dependent base excision repair and increased DMS-induced mutagenicity in aged mice.Association between polymorphisms in APE1 and XRCC1 and the risk of gastric cancer in Korean population.Quantitation of intracellular NAD(P)H can monitor an imbalance of DNA single strand break repair in base excision repair deficient cells in real time.An assay for uracil in human DNA at baseline: effect of marginal vitamin B6 deficiency.Mapping of a functional recombination motif that defines isotype specificity for mu-->gamma3 switch recombination implicates NF-kappaB p50 as the isotype-specific switching factorGastric cancer associated variant of DNA polymerase beta (Leu22Pro) promotes DNA replication associated double strand breaksFive repair pathways in one context: chromatin modification during DNA repair.GammaH2AX and its role in DNA double-strand break repair.Apn1 and Apn2 endonucleases prevent accumulation of repair-associated DNA breaks in budding yeast as revealed by direct chromosomal analysis.Expression of thymidylate synthase in human cells is an early G(1) event regulated by CDK4 and p16INK4A but not E2F.Oxidative DNA damage stalls the human mitochondrial replisomeNucleosome dynamics as modular systems that integrate DNA damage and repair.Mutagenesis is elevated in male germ cells obtained from DNA polymerase-beta heterozygous mice
P2860
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P2860
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
description
2000 nî lūn-bûn
@nan
2000 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@ast
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@en
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@nl
type
label
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@ast
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@en
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@nl
prefLabel
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@ast
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@en
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@nl
P3181
P1433
P1476
Suppression of spontaneous mutagenesis in human cells by DNA base excision-repair
@en
P2093
P304
P3181
P356
10.1016/S1383-5742(00)00024-7
P407
P577
2000-04-01T00:00:00Z