The caspase-3 precursor has a cytosolic and mitochondrial distribution: implications for apoptotic signaling. - Wikidata - wikimedia - TriplyDB

The caspase-3 precursor has a cytosolic and mitochondrial distribution: implications for apoptotic signaling.

The caspase-3 precursor has a cytosolic and mitochondrial distribution: implications for apoptotic signaling.

http://www.wikidata.org/entity/Q36255096

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Presence of a pre-apoptotic complex of pro-caspase-3, Hsp60 and Hsp10 in the mitochondrial fraction of jurkat cells The HIV-1 viral protein R induces apoptosis via a direct effect on the mitochondrial permeability transition pore Identification of the cytolinker plectin as a major early in vivo substrate for caspase 8 during CD95- and tumor necrosis factor receptor-mediated apoptosis Hsp60 accelerates the maturation of pro-caspase-3 by upstream activator proteases during apoptosis.A-kinase-anchoring protein 95 functions as a potential carrier for the nuclear translocation of active caspase 3 through an enzyme-substrate-like association.Caspase-2 is localized at the Golgi complex and cleaves golgin-160 during apoptosis Mitochondrial release of caspase-2 and -9 during the apoptotic process Stress-inducible caspase substrate TRB3 promotes nuclear translocation of procaspase-3 Inactivation of caspase-8 on mitochondria of Bcl-xL-expressing MCF7-Fas cells: role for the bifunctional apoptosis regulator protein Caspase-2 induces apoptosis by releasing proapoptotic proteins from mitochondria Protein kinase Cdelta targets mitochondria, alters mitochondrial membrane potential, and induces apoptosis in normal and neoplastic keratinocytes when overexpressed by an adenoviral vector Bcl-2-dependent oxidation of pyruvate dehydrogenase-E2, a primary biliary cirrhosis autoantigen, during apoptosis Induction of mitochondrial manganese superoxide dismutase confers resistance to apoptosis in acute myeloblastic leukaemia cells exposed to etoposide The mitochondrial permeability transition pore and its role in cell death S-Nitrosylation of mitochondrial caspases Intracellular stability of anti-caspase-3 intrabodies determines efficacy in retargeting the antigen.Traumatic white matter injury and glial activation: from basic science to clinics.Detection of caspases activation by fluorochrome-labeled inhibitors: Multiparameter analysis by laser scanning cytometry.Hypoxia death stimulus induces translocation of p53 protein to mitochondria. Detection by immunofluorescence on whole cells.Detection of localized caspase activity in early apoptotic cells by laser scanning cytometry.Human immunodeficiency virus type 1 Vpr interacts with antiapoptotic mitochondrial protein HAX-1 Postmitochondrial regulation of apoptosis during heart failure Role of p53 family members in apoptosis.Regulation of Fas-mediated apoptosis in CD2-fas transgenic mice.Activation of apoptosis and its inhibition.Characterization of a porcine lung epithelial cell line suitable for influenza virus studies.Macrophages require constitutive NF-kappaB activation to maintain A1 expression and mitochondrial homeostasis Killer proteases and little strokes--how the things that do not kill you make you stronger.Granzyme B short-circuits the need for caspase 8 activity during granule-mediated cytotoxic T-lymphocyte killing by directly cleaving Bid.Apoptosis and its clinical impact.Reovirus-induced apoptosis requires mitochondrial release of Smac/DIABLO and involves reduction of cellular inhibitor of apoptosis protein levels.Mitochondrial defects in neurodegenerative disease.Mitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases.Mitochondria, the killer organelles and their weapons.The role of mitochondrial factors in apoptosis: a Russian roulette with more than one bullet.Wild-type and mutant p53 proteins interact with mitochondrial caspase-3 Apoptosis in myocardial infarction.Pre- and posttreatment with edaravone protects CA1 hippocampus and enhances neurogenesis in the subgranular zone of dentate gyrus after transient global cerebral ischemia in rats.Mitochondria, oxidative stress, and temporal lobe epilepsy Decrease of mitochondrial p53 during late apoptosis is linked to its dephosphorylation on serine 20

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P2860

The caspase-3 precursor has a cytosolic and mitochondrial distribution: implications for apoptotic signaling.

http://www.wikidata.org/entity/Q36255096

description

1998 nî lūn-bûn

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The caspase-3 precursor has a ...... tions for apoptotic signaling.

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A Rosen

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D W Nicholson

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E P Peterson

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L A Casciola-Rosen

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P2860

A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

Investigation of the subcellular distribution of the bcl-2 oncoprotein: residence in the nuclear envelope, endoplasmic reticulum, and outer mitochondrial membranes

Interaction of CED-4 with CED-3 and CED-9: a molecular framework for cell death

Interaction and regulation of subcellular localization of CED-4 by CED-9

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

ICE-LAP6, a novel member of the ICE/Ced-3 gene family, is activated by the cytotoxic T cell protease granzyme B

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9508780

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apoptotic process

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2132665

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