E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
about
Identification of a cell protein (FIP-3) as a modulator of NF-kappaB activity and as a target of an adenovirus inhibitor of tumor necrosis factor alpha-induced apoptosisThe CED-4-homologous protein FLASH is involved in Fas-mediated activation of caspase-8 during apoptosisBtf, a novel death-promoting transcriptional repressor that interacts with Bcl-2-related proteins.Identification of the cytolinker plectin as a major early in vivo substrate for caspase 8 during CD95- and tumor necrosis factor receptor-mediated apoptosisTumor necrosis factor-alpha induces Bax-Bak interaction and apoptosis, which is inhibited by adenovirus E1B 19KKeratin attenuates tumor necrosis factor-induced cytotoxicity through association with TRADDFLASH links the CD95 signaling pathway to the cell nucleus and nuclear bodiesFas-associated death domain protein interacts with methyl-CpG binding domain protein 4: a potential link between genome surveillance and apoptosisThe role of receptor internalization in CD95 signaling.Caspase-8 prevents sustained activation of NF-kappaB in monocytes undergoing macrophagic differentiationAccumulation of BCL10 at the perinuclear region is required for the BCL10-mediated nuclear factor-kappa B activationCaspase-6 is the direct activator of caspase-8 in the cytochrome c-induced apoptosis pathway: absolute requirement for removal of caspase-6 prodomainMyosin ii light chain phosphorylation regulates membrane localization and apoptotic signaling of tumor necrosis factor receptor-1Bax and Bak independently promote cytochrome C release from mitochondriaCaspase recruitment domain (CARD)-dependent cytoplasmic filaments mediate bcl10-induced NF-kappaB activationComplex and dynamic redistribution of NF-kappaB signaling intermediates in response to T cell receptor stimulationRegulation of the cell cycle and apoptosis by the oncogenes of adenovirusp53/T-antigen complex disruption in T-antigen transformed NIH3T3 fibroblasts exposed to oxidative stress: correlation with the appearance of a Fas/APO-1/CD95 dependent, caspase independent, necrotic pathway.Anti-viral strategies of cytotoxic T lymphocytes are manifested through a variety of granule-bound pathways of apoptosis induction.Immune evasion by adenoviruses.Induction of p53-independent apoptosis by the adenovirus E4orf4 protein requires binding to the Balpha subunit of protein phosphatase 2A.Adenovirus-mediated gene expression in vivo is enhanced by the antiapoptotic bcl-2 gene.Inhibition of TRAIL-induced apoptosis and forced internalization of TRAIL receptor 1 by adenovirus proteins.E1B 19K blocks Bax oligomerization and tumor necrosis factor alpha-mediated apoptosis.Subcellular localization and CARD-dependent oligomerization of the death adaptor RAIDD.Oncolytic Viruses for Cancer Therapy: Overcoming the Obstacles.The Rel/NF-kappaB family directly activates expression of the apoptosis inhibitor Bcl-x(L).DNA damage response and MCL-1 destruction initiate apoptosis in adenovirus-infected cellsBinding of FADD and caspase-8 to molluscum contagiosum virus MC159 v-FLIP is not sufficient for its antiapoptotic function.E1A sensitizes cells to tumor necrosis factor alpha by downregulating c-FLIP S.Role of the polarity determinant crumbs in suppressing mammalian epithelial tumor progressionFunctional analysis of the leukemia protein ELL: evidence for a role in the regulation of cell growth and survival.Lipid rafts and raft-mediated supramolecular entities in the regulation of CD95 death receptor apoptotic signaling.E1B 19,000-molecular-weight protein interacts with and inhibits CED-4-dependent, FLICE-mediated apoptosisBak and Bax function to limit adenovirus replication through apoptosis induction.A novel domain in adenovirus L4-100K is required for stable binding and efficient inhibition of human granzyme B: possible interaction with a species-specific exosite.Regulation of the mitochondrial checkpoint in p53-mediated apoptosis confers resistance to cell death.Caspase-2 can trigger cytochrome C release and apoptosis from the nucleus.Phosphorylation of the tumor necrosis factor receptor CD120a (p55) recruits Bcl-2 and protects against apoptosis.X protein of hepatitis B virus inhibits Fas-mediated apoptosis and is associated with up-regulation of the SAPK/JNK pathway.
P2860
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P2860
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
@ast
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
@en
type
label
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
@ast
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
@en
prefLabel
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
@ast
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
@en
P2860
P356
P1476
E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
@en
P2093
P2860
P304
P356
10.1083/JCB.141.5.1255
P407
P50
P577
1998-06-01T00:00:00Z