Bax deletion further orders the cell death pathway in cerebellar granule cells and suggests a caspase-independent pathway to cell death.
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The p53-inducible E3 ubiquitin ligase p53RFP induces p53-dependent apoptosisThe Bax subfamily of Bcl2-related proteins is essential for apoptotic signal transduction by c-Jun NH(2)-terminal kinaseThe histone deacetylase inhibitor and chemotherapeutic agent suberoylanilide hydroxamic acid (SAHA) induces a cell-death pathway characterized by cleavage of Bid and production of reactive oxygen speciesIntrinsic and extrinsic pathway signaling during neuronal apoptosis: lessons from the analysis of mutant miceConcomitant loss of proapoptotic BH3-only Bcl-2 antagonists Bik and Bim arrests spermatogenesisGamma protocadherins are required for synaptic development in the spinal cordNeuroprotection by histone deacetylase-7 (HDAC7) occurs by inhibition of c-jun expression through a deacetylase-independent mechanismCalpain activation is required for glutamate-induced p27 down-regulation in cultured cortical neuronsCofilin activation mediates Bax translocation to mitochondria during excitotoxic neuronal deathDifferences in bcl-2- and bax-independent function in regulating apoptosis in sensory neuron populationsThe role of Bax in glutamate-induced nerve cell deathBax-dependent spermatogonia apoptosis is required for testicular development and spermatogenesisBid induces the oligomerization and insertion of Bax into the outer mitochondrial membraneEstablishment of okadaic acid resistant cell clones using a cDNA expression library.c-Jun regulation in rat neonatal motoneurons postaxotomy.Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.Bax deletion prevents neuronal loss but not neurological symptoms in a transgenic model of inherited prion disease.Early gene expression changes in the retinal ganglion cell layer of a rat glaucoma modelTRPC1 inhibits apoptotic cell degeneration induced by dopaminergic neurotoxin MPTP/MPP(+).MAPK signaling mediates sinomenine hydrochloride-induced human breast cancer cell death via both reactive oxygen species-dependent and -independent pathways: an in vitro and in vivo study.Palmitic and stearic fatty acids induce caspase-dependent and -independent cell death in nerve growth factor differentiated PC12 cells.Implication of TAp73 in the p53-independent pathway of Puma induction and Puma-dependent apoptosis in primary cortical neuronsLysosomal involvement in apoptosis.Bax regulates production of superoxide in both apoptotic and nonapoptotic neurons: role of caspasesThe JNK- and AKT/GSK3β- signaling pathways converge to regulate Puma induction and neuronal apoptosis induced by trophic factor deprivationTrim17, a novel E3 ubiquitin-ligase, initiates neuronal apoptosis.An orchestrated gene expression component of neuronal programmed cell death revealed by cDNA array analysis.Mitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases.Metallothionein and a peptide modeled after metallothionein, EmtinB, induce neuronal differentiation and survival through binding to receptors of the low-density lipoprotein receptor family.Branched chain amino acids induce apoptosis in neural cells without mitochondrial membrane depolarization or cytochrome c release: implications for neurological impairment associated with maple syrup urine disease.The proapoptotic Bcl-2 protein Bax plays an important role in the pathogenesis of reovirus encephalitisIdentification of Bax-voltage-dependent anion channel 1 complexes in digitonin-solubilized cerebellar granule neuronsMen are but worms: neuronal cell death in C elegans and vertebrates.Bax affects production of reactive oxygen by the mitochondria of non-apoptotic neurons.Validation of the Antiproliferative Effects of Organic Extracts from the Green Husk of Juglans regia L. on PC-3 Human Prostate Cancer Cells by Assessment of Apoptosis-Related Genes.Defining the role of the Bcl-2 family of proteins in the nervous system.Caspase-dependent and -independent neuronal death: two distinct pathways to neuronal injury.Role of programmed cell death in normal neuronal development and function.The limited role of NH2-terminal c-Jun phosphorylation in neuronal apoptosis: identification of the nuclear pore complex as a potential target of the JNK pathway.ERK activation promotes neuronal degeneration predominantly through plasma membrane damage and independently of caspase-3.
P2860
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P2860
Bax deletion further orders the cell death pathway in cerebellar granule cells and suggests a caspase-independent pathway to cell death.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Bax deletion further orders th ...... pendent pathway to cell death.
@ast
Bax deletion further orders th ...... pendent pathway to cell death.
@en
type
label
Bax deletion further orders th ...... pendent pathway to cell death.
@ast
Bax deletion further orders th ...... pendent pathway to cell death.
@en
prefLabel
Bax deletion further orders th ...... pendent pathway to cell death.
@ast
Bax deletion further orders th ...... pendent pathway to cell death.
@en
P2093
P2860
P356
P1476
Bax deletion further orders th ...... ependent pathway to cell death
@en
P2093
D J Creedon
E M Johnson
K L Moulder
M Deshmukh
S J Korsmeyer
T M Miller
P2860
P304
P356
10.1083/JCB.139.1.205
P407
P577
1997-10-01T00:00:00Z