Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
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Tissue factor and its natural inhibitor in pre-eclampsia and SGAThe imbalance in expression of angiogenic and anti-angiogenic factors as candidate predictive biomarker in preeclampsiaNitric oxide and reactive oxygen species in the pathogenesis of preeclampsiaRole of endothelin in mediating soluble fms-like tyrosine kinase 1-induced hypertension in pregnant ratsHypertension produced by placental ischemia in pregnant rats is associated with increased soluble endoglin expressionAngiogenic factors for the prediction of pre-eclampsia in women with abnormal midtrimester uterine artery Doppler velocimetry.C5a enhances dysregulated inflammatory and angiogenic responses to malaria in vitro: potential implications for placental malaria.Transthyretin regulates the migration and invasion of JEG-3 cells.Prenatal stretching exercise and autonomic responses: preliminary data and a model for reducing preeclampsia.Altered matrix metalloproteinase-2 and -9 expression/activity links placental ischemia and anti-angiogenic sFlt-1 to uteroplacental and vascular remodeling and collagen deposition in hypertensive pregnancy.Recombinant vascular endothelial growth factor 121 infusion lowers blood pressure and improves renal function in rats with placentalischemia-induced hypertension.Relation between maternal angiogenic factors and utero-placental resistance in normal first- and second-trimester pregnancies.Angiogenic dysfunction in molar pregnancy.Maternal plasma concentrations of angiogenic/antiangiogenic factors in the third trimester of pregnancy to identify the patient at risk for stillbirth at or near term and severe late preeclampsia.Maternal plasma soluble TRAIL is decreased in preeclampsia.Dysregulated complement activation as a common pathway of injury in preeclampsia and other pregnancy complications.Progesterone supplementation attenuates hypertension and the autoantibody to the angiotensin II type I receptor in response to elevated interleukin-6 during pregnancy.Elevated maternal C-reactive protein and increased risk of schizophrenia in a national birth cohort.Carbon monoxide prevents hypertension and proteinuria in an adenovirus sFlt-1 preeclampsia-like mouse model.Differential expression of Vegfr-2 and its soluble form in preeclampsiaRecent insights into the pathophysiology of preeclampsia.Regulation of sFlt-1 and VEGF secretion by adenosine under hypoxic conditions in rat placental villous explants.Progress in obstetrics or the road to the promised land.In vivo experiments reveal the good, the bad and the ugly faces of sFlt-1 in pregnancy.Maternal plasma concentrations of sST2 and angiogenic/anti-angiogenic factors in preeclampsia.Circulating angiogenic and antiangiogenic factors in women with eclampsia.Upregulation of long noncoding RNA SPRY4-IT1 modulates proliferation, migration, apoptosis, and network formation in trophoblast cells HTR-8SV/neoRole of reactive oxygen species during hypertension in response to chronic antiangiogenic factor (sFlt-1) excess in pregnant rats.Levels of soluble fms-like tyrosine kinase one in first trimester and outcomes of pregnancy: a systematic review.Preeclampsia, a disease of the maternal endothelium: the role of antiangiogenic factors and implications for later cardiovascular disease.Interferon-α and angiogenic dysregulation in pregnant lupus patients who develop preeclampsia.Full-length human placental sFlt-1-e15a isoform induces distinct maternal phenotypes of preeclampsia in mice.Regulation of soluble fms-like tyrosine kinase-1 production in response to placental ischemia/hypoxia: role of angiotensin II.Correlation between cell-free mRNA expressions and PLGF protein level in severe preeclampsia.L-arginine supplementation abolishes the blood pressure and endothelin response to chronic increases in plasma sFlt-1 in pregnant rats.Decorin-Mediated Inhibition of Human Trophoblast Cells Proliferation, Migration, and Invasion and Promotion of Apoptosis In Vitro.Difference of concentration of placental soluble fms-like tyrosine kinase-1(sFlt-1), placental growth factor (PlGF), and sFlt-1/PlGF ratio in severe preeclampsia and normal pregnancy.Complement activation induces dysregulation of angiogenic factors and causes fetal rejection and growth restriction.Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia.The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancy.
P2860
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P2860
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
@ast
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
@en
type
label
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
@ast
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
@en
prefLabel
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
@ast
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
@en
P921
P1433
P1476
Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia.
@en
P2093
P304
P356
10.1161/01.HYP.0000187899.34379.B0
P407
P577
2005-10-17T00:00:00Z