The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancy.
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Preeclampsia: long-term consequences for vascular healthPathophysiology of hypertension in pre-eclampsia: a lesson in integrative physiologyA leading role for the immune system in the pathophysiology of preeclampsiaThe renal circulation in normal pregnancy and preeclampsia: is there a place for relaxin?Angiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyondA Dormant Microbial Component in the Development of PreeclampsiaPre-eclampsia part 1: current understanding of its pathophysiology.Soluble endoglin, transforming growth factor-Beta 1 and soluble tumor necrosis factor alpha receptors in different clinical manifestations of preeclampsia.The therapeutic potential of antioxidants, ER chaperones, NO and H2S donors, and statins for treatment of preeclampsia.B cells: the old new players in reproductive immunology.The role of inflammation in the pathology of preeclampsiaProgesterone supplementation attenuates hypertension and the autoantibody to the angiotensin II type I receptor in response to elevated interleukin-6 during pregnancy.CD4(+) T Cells Play a Critical Role in Mediating Hypertension in Response to Placental Ischemia.Agonistic Autoantibodies to the Angiotensin II Type 1 Receptor Enhance Angiotensin II-Induced Renal Vascular Sensitivity and Reduce Renal Function During Pregnancy.Heterozygous deficiency of endoglin decreases insulin and hepatic triglyceride levels during high fat dietRole of angiotensin II type I receptor agonistic autoantibodies (AT1-AA) in preeclampsia.Hypertension in response to placental ischemia during pregnancy: role of B lymphocytes.Mesenchymal stem cells ameliorate Th1-induced pre-eclampsia-like symptoms in mice via the suppression of TNF-α expression.CD4+ T-helper cells stimulated in response to placental ischemia mediate hypertension during pregnancyRegulation of soluble fms-like tyrosine kinase-1 production in response to placental ischemia/hypoxia: role of angiotensin II.Recombinant vascular endothelial growth factor 121 attenuates autoantibody-induced features of pre-eclampsia in pregnant mice.Receptor-activating autoantibodies and disease: preeclampsia and beyond.Activating autoantibodies to the angiotensin II type I receptor play an important role in mediating hypertension in response to adoptive transfer of CD4+ T lymphocytes from placental ischemic ratsHypertension in response to CD4(+) T cells from reduced uterine perfusion pregnant rats is associated with activation of the endothelin-1 system.A model of preeclampsia in rats: the reduced uterine perfusion pressure (RUPP) model.IL-17-mediated oxidative stress is an important stimulator of AT1-AA and hypertension during pregnancy.Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation.Beneficial effect of human umbilical cord-derived mesenchymal stem cells on an endotoxin-induced rat model of preeclampsiaAn increased population of regulatory T cells improves the pathophysiology of placental ischemia in a rat model of preeclampsia.Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia.GPCRs as potential therapeutic targets in preeclampsia.Immune Mechanisms Linking Obesity and Preeclampsia.Increased risk for the development of preeclampsia in obese pregnancies: weighing in on the mechanisms.Control of soluble fms-like tyrosine-1 (sFlt-1) production response to placental ischemia/hypoxia: role of tumor necrosis factor-α.Vitamin D supplementation improves pathophysiology in a rat model of preeclampsiaHypoxia-independent upregulation of placental hypoxia inducible factor-1α gene expression contributes to the pathogenesis of preeclampsia.Increased Risk of Intracranial Hemorrhage in Patients With Pregnancy-Induced Hypertension: A Nationwide Population-Based Retrospective Cohort Study.IL-10 supplementation increases Tregs and decreases hypertension in the RUPP rat model of preeclampsia.Transglutaminase is a Critical Link Between Inflammation and Hypertension.TNF-mediated damage to glomerular endothelium is an important determinant of acute kidney injury in sepsis.
P2860
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P2860
The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancy.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
The effect of immune factors, ...... hypertension during pregnancy.
@ast
The effect of immune factors, ...... hypertension during pregnancy.
@en
type
label
The effect of immune factors, ...... hypertension during pregnancy.
@ast
The effect of immune factors, ...... hypertension during pregnancy.
@en
prefLabel
The effect of immune factors, ...... hypertension during pregnancy.
@ast
The effect of immune factors, ...... hypertension during pregnancy.
@en
P2093
P2860
P50
P356
P1476
The effect of immune factors, ...... hypertension during pregnancy.
@en
P2093
Babbette LaMarca
James N Martin
Joey P Granger
Katrin Wenzel
Lillian Fournier Ray
Marc R Parrish
Sarah Rutland
Sharon Keiser
Sydney R Murphy
P2860
P304
P356
10.1038/AJH.2010.70
P577
2010-04-29T00:00:00Z