Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration. - Wikidata - wikimedia - TriplyDB

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

http://www.wikidata.org/entity/Q36290307

about

60 YEARS OF NEUROENDOCRINOLOGY: The hypothalamo-prolactin axis The role of the prolactin/vasoinhibin axis in rheumatoid arthritis: an integrative overview.Kisspeptin modulates sexual and emotional brain processing in humans.Twice-daily subcutaneous injection of kisspeptin-54 does not abolish menstrual cyclicity in healthy female volunteers.Neuroendocrine causes of amenorrhea--an update.Hyperprolactinemia and infertility: new insights EP24.15 as a Potential Regulator of Kisspeptin Within the Neuroendocrine Hypothalamus.Giant prolactinomas: the therapeutic approach.Endocrine autoimmune diseases and female infertility.Prolactinomas, cabergoline, and pregnancy.New insights in prolactin: pathological implications.An updated view of hypothalamic-vascular-pituitary unit function and plasticity.Interactions between prolactin and kisspeptin to control reproduction.Impact of Triclosan on Female Reproduction through Reducing Thyroid Hormones to Suppress Hypothalamic Kisspeptin Neurons in Mice.Plasma kisspeptin levels in lactational amenorrhea.Hypothalamic-Pituitary-Ovarian Axis Reactivation by Kisspeptin-10 in Hyperprolactinemic Women With Chronic Amenorrhea.

P2860

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P2860

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

http://www.wikidata.org/entity/Q36290307

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

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2012年學術文章

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2012年學術文章

@zh-hant

name

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

@ast

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

@en

type

label

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

@ast

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

@en

prefLabel

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

@ast

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration.

@en

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P1433

Journal of Clinical Investigation

P1476

Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration

@en

P2093

Alain Caraty

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Charlotte Sonigo

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Fabian-Jesus Simony-Conesa

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Justine Bouilly

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Nadine Binart

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Nadège Carré

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Virginie Tolle

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P2860

Prolactin receptor signaling is essential for perinatal brown adipocyte function: a role for insulin-like growth factor-2

The GPR54 gene as a regulator of puberty

Kisspeptin signaling in the brain

Uncovering novel reproductive defects in neurokinin B receptor null mice: closing the gap between mice and men.

Regulation of food intake and gonadotropin-releasing hormone/luteinizing hormone during lactation: role of insulin and leptin.

Neurokinin B and the hypothalamic regulation of reproduction.

Kisspeptin immunoreactive cells of the ovine preoptic area and arcuate nucleus co-express estrogen receptor alpha.

Prolactin regulation of gonadotropin-releasing hormone neurons to suppress luteinizing hormone secretion in mice.

Hypogonadotropic hypogonadism in mice lacking a functional Kiss1 gene.

Kisspeptin can stimulate gonadotropin-releasing hormone (GnRH) release by a direct action at GnRH nerve terminals.

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3791-3795

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scholarly article

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10.1172/JCI63937

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10

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122

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Robert P. Millar

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2012-09-24T00:00:00Z

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23006326

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3461919

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