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Hypoxic pulmonary vasoconstriction: mechanisms and controversies.

Hypoxic pulmonary vasoconstriction: mechanisms and controversies.

http://www.wikidata.org/entity/Q36299224

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The human side of hypoxia-inducible factor Pulmonary blast injury in mice: a novel model for studying blast injury in the laboratory using laser-induced stress waves.Hypoxic pulmonary vasodilation: a paradigm shift with a hydrogen sulfide mechanism The NADPH oxidase subunit NOX4 is a new target gene of the hypoxia-inducible factor-1.Endothelial and smooth muscle cell ion channels in pulmonary vasoconstriction and vascular remodeling Inhibition of hypoxic pulmonary vasoconstriction of rats by carbon monoxide.Superoxide differentially controls pulmonary and systemic vascular tone through multiple signalling pathways Pulmonary vasodilation by acetazolamide during hypoxia is unrelated to carbonic anhydrase inhibition.Acute pulmonary edema due to hypoxia during a difficult intubation in a rheumatoid arthritis patient Hypoxia. 4. Hypoxia and ion channel function Endothelin-1 contributes to increased NFATc3 activation by chronic hypoxia in pulmonary arteries.A mitochondrial redox oxygen sensor in the pulmonary vasculature and ductus arteriosus Hypoxia reduces KCa channel activity by inducing Ca2+ spark uncoupling in cerebral artery smooth muscle cells Spatial-temporal dynamics of pulmonary blood flow in the healthy human lung in response to altered FI(O2).Involvement of gap junctions between smooth muscle cells in sustained hypoxic pulmonary vasoconstriction development: a potential role for 15-HETE and 20-HETE Gap junctions support the sustained phase of hypoxic pulmonary vasoconstriction by facilitating calcium sensitization.The increase in pulmonary arterial pressure caused by hypoxia depends on iron status.Mechanisms of hypoxic pulmonary vasoconstriction and their roles in pulmonary hypertension: new findings for an old problem Effects of dimethylarginine dimethylaminohydrolase-1 overexpression on the response of the pulmonary vasculature to hypoxia.Mitochondrial reactive oxygen species regulate hypoxic signaling.HIF and pulmonary vascular responses to hypoxia.The vasorelaxing effect of hydrogen sulfide on isolated rat aortic rings versus pulmonary artery rings Regulation of the pulmonary circulation in the fetus and newborn.Oxygen dependence of metabolism and cellular adaptation in vertebrate muscles: a review.Rho GTPases in the regulation of pulmonary vascular barrier function.Hypoxia-dependent reactive oxygen species signaling in the pulmonary circulation: focus on ion channels.Pulmonary vascular dysfunction in ARDS.Lung Circulation.Mitochondria control acute and chronic responses to hypoxia.Mitochondria-dependent regulation of Kv currents in rat pulmonary artery smooth muscle cells.Role of ROS signaling in differential hypoxic Ca2+ and contractile responses in pulmonary and systemic vascular smooth muscle cells.Analysis of pulmonary vasodilator responses to SB-772077-B [4-(7-((3-amino-1-pyrrolidinyl)carbonyl)-1-ethyl-1H-imidazo(4,5-c)pyridin-2-yl)-1,2,5-oxadiazol-3-amine], a novel aminofurazan-based Rho kinase inhibitor.Diacylglycerol regulates acute hypoxic pulmonary vasoconstriction via TRPC6.Sphingosylphosphorylcholine potentiates vasoreactivity and voltage-gated Ca2+ entry via NOX1 and reactive oxygen species.Hypoxic pulmonary vasoconstriction in the absence of pretone: essential role for intracellular Ca2+ release.The Rho kinase inhibitor azaindole-1 has long-acting vasodilator activity in the pulmonary vascular bed of the intact chest rat.HIF-1 alpha-induced up-regulation of miR-9 contributes to phenotypic modulation in pulmonary artery smooth muscle cells during hypoxia.Heterogeneity of hypoxia-mediated decrease in I(K(V)) and increase in [Ca2+](cyt) in pulmonary artery smooth muscle cells.Mechanisms of the prostaglandin F2alpha-induced rise in [Ca2+]i in rat intrapulmonary arteries.Hydrogen sulfide mediates hypoxic vasoconstriction through a production of mitochondrial ROS in trout gills.

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Hypoxic pulmonary vasoconstriction: mechanisms and controversies.

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Hypoxic pulmonary vasoconstriction: mechanisms and controversies.

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The Journal of Physiology

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Hypoxic pulmonary vasoconstriction: mechanisms and controversies.

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Gregory A Knock

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Jeremy P T Ward

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Philip I Aaronson

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Silke Becker

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Tom P Robertson

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Tristan H Lewis

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Vladimir Snetkov

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P2860

Adp-ribosyl cyclase and cyclic ADP-ribose hydrolase act as a redox sensor. a primary role for cyclic ADP-ribose in hypoxic pulmonary vasoconstriction.

Mechanisms of hypoxic pulmonary vasoconstriction: can anyone be right?

Hypoxic pulmonary vasoconstriction: cyclic adenosine diphosphate-ribose, smooth muscle Ca(2+) stores and the endothelium.

Endothelium-derived mediators and hypoxic pulmonary vasoconstriction.

The mechanism(s) of hypoxic pulmonary vasoconstriction: potassium channels, redox O(2) sensors, and controversies.

Prominent role of intracellular Ca2+ release in hypoxic vasoconstriction of canine pulmonary artery

Hypoxic pulmonary vasoconstriction: redox events in oxygen sensing.

Hypoxic pulmonary vasoconstriction: role of ion channels.

Cellular mechanisms of hypoxia-induced contraction in human and rat pulmonary arteries.

Molecular identification of the role of voltage-gated K+ channels, Kv1.5 and Kv2.1, in hypoxic pulmonary vasoconstriction and control of resting membrane potential in rat pulmonary artery myocytes.

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