Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
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Group VIA Phospholipase A2 (iPLA2β) Modulates Bcl-x 5'-Splice Site Selection and Suppresses Anti-apoptotic Bcl-x(L) in β-Cells.Post-Transcriptional Regulation of Anti-Apoptotic BCL2 Family Members.NG25, an inhibitor of transforming growth factor‑β‑activated kinase 1, ameliorates neuronal apoptosis in neonatal hypoxic‑ischemic rats.Splicing factor NSSR1 reduces neuronal injury after mouse transient global cerebral ischemia.Olive Oil-Supplemented Lipid Emulsion Induces CELF1 Expression and Promotes Apoptosis in Caco-2 Cells.
P2860
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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name
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
@ast
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
@en
type
label
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
@ast
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
@en
prefLabel
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
@ast
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
@en
P2093
P2860
P1476
Bcl-x pre-mRNA splicing regulates brain injury after neonatal hypoxia-ischemia.
@en
P2093
Andria L Ford
Ernesto Gonzales
Jin-Moo Lee
Kuang-Yung Lee
Qingli Xiao
P2860
P304
13587-13596
P356
10.1523/JNEUROSCI.2617-12.2012
P407
P577
2012-09-01T00:00:00Z