Conversion of an M- group A streptococcus to M+ by transfer of a plasmid containing an M6 gene
about
SipA is required for pilus formation in Streptococcus pyogenes serotype M3A novel endogenous inhibitor of the secreted streptococcal NAD-glycohydrolase.YvqE and CovRS of Group A Streptococcus Play a Pivotal Role in Viability and Phenotypic Adaptations to Multiple Environmental StressesPathogenesis of group A streptococcal infectionsStreptococcal M protein: molecular design and biological behavior.Streptolysin S inhibits neutrophil recruitment during the early stages of Streptococcus pyogenes infection.Molecular characterization of pcp, the structural gene encoding the pyrrolidone carboxylyl peptidase from Streptococcus pyogenes.An intermediate in transposition of the conjugative transposon Tn916.Role of M protein in pharyngeal colonization by group A streptococci in ratsProtein F, a fibronectin-binding protein, is an adhesin of the group A streptococcus Streptococcus pyogenes.Expression of streptococcal M protein in mammalian cells.Streptococcus pyogenes cytolysin-mediated translocation does not require pore formation by streptolysin O.Variation in Streptococcus pyogenes NAD+ glycohydrolase is associated with tissue tropism.A response regulator that represses transcription of several virulence operons in the group A streptococcus.Regulation of mga transcription in the group A streptococcus: specific binding of mga within its own promoter and evidence for a negative regulatorContribution of NADH oxidase to aerobic metabolism of Streptococcus pyogenes.The RofA binding site in Streptococcus pyogenes is utilized in multiple transcriptional pathwaysAlkaline phosphatase reporter transposon for identification of genes encoding secreted proteins in gram-positive microorganismsTwo DNA-binding domains of Mga are required for virulence gene activation in the group A streptococcus.RscA, a member of the MDR1 family of transporters, is repressed by CovR and required for growth of Streptococcus pyogenes under heat stress.Differential recognition of surface proteins in Streptococcus pyogenes by two sortase gene homologs.CovS inactivates CovR and is required for growth under conditions of general stress in Streptococcus pyogenesIdentification of a gene that regulates expression of M protein, the major virulence determinant of group A streptococci.Identification of a fibronectin-binding protein (GfbA) in pathogenic group G streptococci.A novel cholesterol-insensitive mode of membrane binding promotes cytolysin-mediated translocation by Streptolysin OMoonlighting glutamate formiminotransferases can functionally replace 5-formyltetrahydrofolate cycloligase.Membrane cofactor protein (CD46) is a keratinocyte receptor for the M protein of the group A streptococcusMga is sufficient to activate transcription in vitro of sof-sfbX and other Mga-regulated virulence genes in the group A StreptococcusIdentification and characterization of a Streptococcus pyogenes operon involved in binding of hemoproteins and acquisition of iron.Streptolysin O and its co-toxin NAD-glycohydrolase protect group A Streptococcus from Xenophagic killing.Identification of rocA, a positive regulator of covR expression in the group A streptococcus.Fibrinogen binding and resistance to phagocytosis of Streptococcus sanguis expressing cloned M protein of Streptococcus pyogenesExpression of the secondary sigma factor sigmaX in Streptococcus pyogenes is restricted at two levelsMutations in the control of virulence sensor gene from Streptococcus pyogenes after infection in mice lead to clonal bacterial variants with altered gene regulatory activity and virulence.Expression of the Arp protein, a member of the M protein family, is not sufficient to inhibit phagocytosis of Streptococcus pyogenes.An oxygen-induced but protein F-independent fibronectin-binding pathway in Streptococcus pyogenes.Role of putative virulence factors of Streptococcus pyogenes in mouse models of long-term throat colonization and pneumonia.Keratinocyte proinflammatory responses to adherent and nonadherent group A streptococci.Conversion of M serotype 24 of Streptococcus pyogenes to M serotypes 5 and 18: effect on resistance to phagocytosis and adhesion to host cells.Insertional inactivation of Streptococcus pyogenes sod suggests that prtF is regulated in response to a superoxide signal.
P2860
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P2860
Conversion of an M- group A streptococcus to M+ by transfer of a plasmid containing an M6 gene
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年学术文章
@wuu
1986年学术文章
@zh-cn
1986年学术文章
@zh-hans
1986年学术文章
@zh-my
1986年学术文章
@zh-sg
1986年學術文章
@yue
1986年學術文章
@zh
1986年學術文章
@zh-hant
name
Conversion of an M- group A st ...... plasmid containing an M6 gene
@ast
Conversion of an M- group A st ...... plasmid containing an M6 gene
@en
type
label
Conversion of an M- group A st ...... plasmid containing an M6 gene
@ast
Conversion of an M- group A st ...... plasmid containing an M6 gene
@en
prefLabel
Conversion of an M- group A st ...... plasmid containing an M6 gene
@ast
Conversion of an M- group A st ...... plasmid containing an M6 gene
@en
P2093
P2860
P356
P1476
Conversion of an M- group A st ...... plasmid containing an M6 gene
@en
P2093
L M Malone
P C Guenthner
V A Fischetti
P2860
P304
P356
10.1084/JEM.164.5.1641
P407
P577
1986-11-01T00:00:00Z