Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement. - Wikidata - wikimedia - TriplyDB

Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement.

Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement.

http://www.wikidata.org/entity/Q36366602

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Beta-glucan enhances complement-mediated hematopoietic recovery after bone marrow injury Emerging Functions of Natural IgM and Its Fc Receptor FCMR in Immune Homeostasis Molecular mechanisms of inflammation and tissue injury after major trauma--is complement the "bad guy"?The role of complement in the pathogenesis of renal ischemia-reperfusion injury and fibrosis Monoclonal antibodies against peptidorhamnomannans of Scedosporium apiospermum enhance the pathogenicity of the fungus Autoreactivity by design: innate B and T lymphocytes Complement activation by apoptotic endothelial cells following hypoxia/reoxygenation How dying cells alert the immune system to danger Ulk1-mediated autophagy plays an essential role in mitochondrial remodeling and functional regeneration of skeletal muscle.C1 esterase inhibitor reduces lower extremity ischemia/reperfusion injury and associated lung damage.Detrimental effects of complement activation in hemorrhagic shock.Kupffer cell-initiated remote hepatic injury following bilateral hindlimb ischemia is complement dependent.Complement Factor C4d Is a Common Denominator in Thrombotic Microangiopathy.Low-level laser therapy for protection against skeletal muscle damage after ischemia-reperfusion injury in rat hindlimbs.B cells limit repair after ischemic acute kidney injury.Membrane lipid interactions in intestinal ischemia/reperfusion-induced Injury Inhibition of rat gut reperfusion injury with an agent developed for the mouse. Evidence that amplification of injury by innate immunity is conserved between two animal species.Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction Cell membrane modification for rapid display of bi-functional peptides: a novel approach to reduce complement activation.Mast cell protease 5 mediates ischemia-reperfusion injury of mouse skeletal muscle Complement factor 3 deficiency attenuates hemorrhagic shock-related hepatic injury and systemic inflammatory response syndrome.Attenuation of the effects of rat hemorrhagic shock with a reperfusion injury-inhibiting agent specific to mice.Domain V peptides inhibit beta2-glycoprotein I-mediated mesenteric ischemia/reperfusion-induced tissue damage and inflammation.Helicobacter infection alters MyD88 and Trif signalling in response to intestinal ischaemia-reperfusion.CR2+ marginal zone B cell production of pathogenic natural antibodies is C3 independent Sca-1 influences the innate immune response during skeletal muscle regeneration.Oxidative stress sensitizes retinal pigmented epithelial (RPE) cells to complement-mediated injury in a natural antibody-, lectin pathway-, and phospholipid epitope-dependent manner.The inflammatory response after an epidermal burn depends on the activities of mouse mast cell proteases 4 and 5.The inflammatory response to cell death.Heme interacts with c1q and inhibits the classical complement pathway.Complement in ischemia reperfusion injury.The role of complement and natural antibody in intestinal ischemia-reperfusion injury.Pathways to acute humoral rejection Locally produced complement and its role in renal allograft rejection.Evaluation of the contribution of multiple DAMPs and DAMP receptors in cell death-induced sterile inflammatory responses.Targeting pathogenic postischemic self-recognition by natural IgM to protect against posttransplantation cardiac reperfusion injury Optimal modes and targets of gene therapy in transplantation.IgM colocalises with complement and C reactive protein in infarcted human myocardium.Acidosis activates complement system in vitro.Innate response to self-antigen significantly exacerbates burn wound depth.

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Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement.

http://www.wikidata.org/entity/Q36366602

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1996 nî lūn-bûn

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Reperfusion injury of ischemic ...... tural antibody and complement.

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Reperfusion injury of ischemic ...... tural antibody and complement.

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Reperfusion injury of ischemic ...... tural antibody and complement.

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Reperfusion injury of ischemic ...... tural antibody and complement.

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Journal of Experimental Medicine

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Reperfusion injury of ischemic ...... tural antibody and complement.

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P2093

F D Moore

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H B Hechtman

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J P Williams

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L Kobzik

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M C Carroll

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M Ma

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M R Weiser

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P2860

Molecular organization and function of the complement system

RAG-2-deficient mice lack mature lymphocytes owing to inability to initiate V(D)J rearrangement

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity.

Antibody to CD-18 exerts endothelial and cardiac protective effects in myocardial ischemia and reperfusion.

Immunoglobulin M antibodies present in the acute phase of Kawasaki syndrome lyse cultured vascular endothelial cells stimulated by gamma interferon.

Hypoxia induces endothelial cell synthesis of membrane-associated proteins.

Inhibition of leukocyte adherence by anti-CD18 monoclonal antibody attenuates reperfusion injury in the rabbit ear.

Membrane complement receptors specific for bound fragments of C3.

Complement ligand-receptor interactions that mediate biological responses.

Natural (antiphospholipid-PDH,-DNA) autoantibodies and their physiologic serum inhibitors.

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2343-2348

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10.1084/JEM.183.5.2343

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5

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183

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8642343

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P921

reperfusion injury

P932

2192547

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