Bad can act as a key regulator of T cell apoptosis and T cell development.
about
The growth-promoting activity of the Bad protein in chicken embryo fibroblasts requires binding to protein 14-3-3Regulation of thymocyte homeostasis by Fliz1Direct reversal of glucocorticoid resistance by AKT inhibition in acute lymphoblastic leukemiaExpression of the Bcl-2 protein BAD promotes prostate cancer growthDeficiency of PTEN in Jurkat T cells causes constitutive localization of Itk to the plasma membrane and hyperresponsiveness to CD3 stimulationAkt phosphorylates and regulates the orphan nuclear receptor Nur77Tcl1 enhances Akt kinase activity and mediates its nuclear translocationComplex formation and cooperation of protein kinase C theta and Akt1/protein kinase B alpha in the NF-kappa B transactivation cascade in Jurkat T cellsTargeting of the transcription factor Max during apoptosis: phosphorylation-regulated cleavage by caspase-5 at an unusual glutamic acid residue in position P1Deletion of Bid impedes cell proliferation and hepatic carcinogenesisIdentification of a novel phosphorylation site, Ser-170, as a regulator of bad pro-apoptotic activityBAD detects coincidence of G2/M phase and growth factor deprivation to regulate apoptosis.Latent membrane protein 2A-mediated effects on the phosphatidylinositol 3-Kinase/Akt pathway.BH3-only proteins in apoptosis and beyond: an overview.Inhibition of apoptosis by gamma interferon in cells and mice infected with Chlamydia muridarum (the mouse pneumonitis strain of Chlamydia trachomatis).Individual and overlapping roles of BH3-only proteins Bim and Bad in apoptosis of lymphocytes and platelets and in suppression of thymic lymphoma developmentThe kinase PDK1 is essential for B-cell receptor mediated survival signaling.Transgenic expression of CXCR3 on T cells enhances susceptibility to cutaneous Leishmania major infection by inhibiting monocyte maturation and promoting a Th2 response.Crosstalk pathway for inhibition of glucocorticoid-induced apoptosis by T cell receptor signaling.BIM and other BCL-2 family proteins exhibit cross-species conservation of function between zebrafish and mammals.PI3K/Akt and apoptosis: size matters.On the link between Bcl-2 family proteins and glucocorticoid-induced apoptosis.The proapoptotic BH3-only protein bim is expressed in hematopoietic, epithelial, neuronal, and germ cells.Interleukin-7 in T-cell acute lymphoblastic leukemia: an extrinsic factor supporting leukemogenesis?Glucocorticoids in T cell apoptosis and function.Protein kinase B regulates T lymphocyte survival, nuclear factor kappaB activation, and Bcl-X(L) levels in vivoCD28-dependent activation of protein kinase B/Akt blocks Fas-mediated apoptosis by preventing death-inducing signaling complex assembly.Transcription factor NFAT1 controls allergic contact hypersensitivity through regulation of activation induced cell death program.BAD overexpression inhibits cell growth and induces apoptosis via mitochondrial-dependent pathway in non-small cell lung cancer.Latent membrane protein 2A inhibits transforming growth factor-beta 1-induced apoptosis through the phosphatidylinositol 3-kinase/Akt pathway.Differences in Bcl-2 expression by T-cell subsets alter their balance after in vivo irradiation to favor CD4+Bcl-2hi NKT cells.BAD: undertaker by night, candyman by day.Phosphorylation of the transcription factor YY1 by CK2α prevents cleavage by caspase 7 during apoptosis.Bik subcellular localization in response to oxidative stress induced by chemotherapy, in Two different breast cancer cell lines and a Non-tumorigenic epithelial cell line.CaMKII-γ mediates phosphorylation of BAD at Ser170 to regulate cytokine-dependent survival and proliferation.Pro-apoptotic protein BIM in apoptosis of glucocorticoid-sensitive and -resistant acute lymphoblastic leukemia CEM cells.Bcl-2 activates a programme of premature senescence in human carcinoma cells.Dexamethasone inhibits maturation, cytokine production and Fc epsilon RI expression of human cord blood-derived mast cells.Selective glucocorticoid receptor translational isoforms reveal glucocorticoid-induced apoptotic transcriptomes.IL-7 determines the homeostatic fitness of T cells by distinct mechanisms at different signalling thresholds in vivo.
P2860
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P2860
Bad can act as a key regulator of T cell apoptosis and T cell development.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Bad can act as a key regulator of T cell apoptosis and T cell development.
@ast
Bad can act as a key regulator of T cell apoptosis and T cell development.
@en
type
label
Bad can act as a key regulator of T cell apoptosis and T cell development.
@ast
Bad can act as a key regulator of T cell apoptosis and T cell development.
@en
prefLabel
Bad can act as a key regulator of T cell apoptosis and T cell development.
@ast
Bad can act as a key regulator of T cell apoptosis and T cell development.
@en
P2093
P2860
P356
P1476
Bad can act as a key regulator of T cell apoptosis and T cell development
@en
P2093
D Kioussis
G Gil-Gómez
O Williams
P2860
P304
P356
10.1084/JEM.189.3.575
P407
P50
P577
1999-02-01T00:00:00Z