Infectious agents are not necessary for murine atherogenesis. - Wikidata - wikimedia - TriplyDB

Infectious agents are not necessary for murine atherogenesis.

Infectious agents are not necessary for murine atherogenesis.

http://www.wikidata.org/entity/Q36368432

about

Gut flora metabolism of phosphatidylcholine promotes cardiovascular disease Role of Nucleotide-binding and Oligomerization Domain 2 Protein (NOD2) in the Development of Atherosclerosis S100A8 and S100A9 in cardiovascular biology and disease Cholesterol crystals and inflammation Human Microbiome: When a Friend Becomes an Enemy Inflammasomes and metabolic disorders: old genes in modern diseases Drivers of age-related inflammation and strategies for healthspan extension Therapeutic targeting of NOD1 receptors NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals Vascular cells contribute to atherosclerosis by cytokine- and innate-immunity-related inflammatory mechanisms.Chlamydia pneumoniae and atherosclerosis: critical assessment of diagnostic methods and relevance to treatment studies The quantity of nitric oxide released by macrophages regulates Chlamydia-induced disease.Far from the eyes, close to the heart: dysbiosis of gut microbiota and cardiovascular consequences.The gut microbiota, environment and diseases of modern society.Role of toll-like receptor 4 in intimal foam cell accumulation in apolipoprotein E-deficient mice Atherosclerosis and Alzheimer--diseases with a common cause? Inflammation, oxysterols, vasculature NLRP3 inflammasomes link inflammation and metabolic disease.Toll-like receptor 4 and atherogenesis.Helicobacter pylori vs coronary heart disease - searching for connections.Nucleotide-binding oligomerization domain protein 2 deficiency enhances neointimal formation in response to vascular injury Unraveling the environmental and genetic interactions in atherosclerosis: Central role of the gut microbiota.Immunomodulation and vaccination for atherosclerosis.Cholesterol, Inflammasomes, and Atherogenesis.Role of Toll-like receptor 4 in the initiation and progression of atherosclerotic disease.Linking immune-mediated arterial inflammation and cholesterol-induced atherosclerosis in a transgenic mouse model.Autoreactive preplasma cells break tolerance in the absence of regulation by dendritic cells and macrophages Atherosclerosis and innate immune signaling.Intestinal Immunity and Gut Microbiota in Atherogenesis.Protective role for TLR4 signaling in atherosclerosis progression as revealed by infection with a common oral pathogen.Development potential of rifalazil and other benzoxazinorifamycins.Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E Emerging role of Toll-like receptors in atherosclerosis.The role of innate immunity in atherogenesis.Lipopolysaccharide, CD14 and Toll-like receptors: an emerging link between innate immunity and atherosclerotic disease.The role of gut microbiota (commensal bacteria) and the mucosal barrier in the pathogenesis of inflammatory and autoimmune diseases and cancer: contribution of germ-free and gnotobiotic animal models of human diseases.Toll-like receptor signaling: common pathways that drive cardiovascular disease and rheumatoid arthritis.Molecular biology of atherosclerosis.Processes of sterile inflammation.Inflammasome activation by nucleic acids and nucleosomes in sterile inflammation… or is it sterile?The immunology of atherosclerosis.

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P2860

Infectious agents are not necessary for murine atherogenesis.

http://www.wikidata.org/entity/Q36368432

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2000 nî lūn-bûn

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2000年の論文

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2000年学术文章

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2000年学术文章

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2000年学术文章

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2000年学术文章

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2000年學術文章

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2000年學術文章

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2000年學術文章

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name

Infectious agents are not necessary for murine atherogenesis.

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Infectious agents are not necessary for murine atherogenesis.

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type

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Infectious agents are not necessary for murine atherogenesis.

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Infectious agents are not necessary for murine atherogenesis.

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Infectious agents are not necessary for murine atherogenesis.

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Infectious agents are not necessary for murine atherogenesis.

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Journal of Experimental Medicine

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Infectious agents are not necessary for murine atherogenesis.

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P2093

A Hermanowski-Vosatka

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C Burton

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C P Sparrow

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D J Card

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H Hassing

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J D Bergstrom

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J Montenegro

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M Hernandez

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P A Detmers

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

A simple method for the isolation and purification of total lipides from animal tissues

Atherosclerosis — An Inflammatory Disease

Severe hypercholesterolemia and atherosclerosis in apolipoprotein E-deficient mice created by homologous recombination in ES cells

Toll, a new piece in the puzzle of innate immunity.

The stressful condition as a nutritionally dependent adaptive dichotomy.

Does Chlamydia pneumoniae cause atherosclerosis?

The atherogenic effects of chlamydia are dependent on serum cholesterol and specific to Chlamydia pneumoniae.

Decreased atherosclerosis in mice deficient in both macrophage colony-stimulating factor (op) and apolipoprotein E

Chlamydia pneumoniae (TWAR) in coronary arteries of young adults (15-34 years old)

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1437-1442

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191

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