Heme oxygenase-1 gene promoter polymorphism is associated with reduced incidence of acute chest syndrome among children with sickle cell disease - Wikidata - wikimedia - TriplyDB

Heme oxygenase-1 gene promoter polymorphism is associated with reduced incidence of acute chest syndrome among children with sickle cell disease

Heme oxygenase-1 gene promoter polymorphism is associated with reduced incidence of acute chest syndrome among children with sickle cell disease

http://www.wikidata.org/entity/Q36370871

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Blood transfusion for preventing primary and secondary stroke in people with sickle cell disease Genomic approaches to identifying targets for treating β hemoglobinopathies The intersection between asthma and acute chest syndrome in children with sickle-cell anaemia Perspectives in Genetics and Sickle Cell Disease Prevention in Africa: Beyond the Preliminary Data from Cameroon Acute chest syndrome is associated with single nucleotide polymorphism-defined beta globin cluster haplotype in children with sickle cell anaemia.Heme-induced neutrophil extracellular traps contribute to the pathogenesis of sickle cell disease.MP4CO, a pegylated hemoglobin saturated with carbon monoxide, is a modulator of HO-1, inflammation, and vaso-occlusion in transgenic sickle mice.Hemin controls T cell polarization in sickle cell alloimmunization.Look out heme, Mac is back in town Severe painful vaso-occlusive crises and mortality in a contemporary adult sickle cell anemia cohort study.Polymorphisms in the Haem Oxygenase-1 promoter are not associated with severity of Plasmodium falciparum malaria in Ghanaian children.Genetic variants and cell-free hemoglobin processing in sickle cell nephropathy.Blood transfusion for preventing primary and secondary stroke in people with sickle cell disease.Impairment of neutrophil oxidative burst in children with sickle cell disease is associated with heme oxygenase-1.Inflammatory targets of therapy in sickle cell disease.Gene-centric association study of acute chest syndrome and painful crisis in sickle cell disease patients.Association between plasma free haem and incidence of vaso-occlusive episodes and acute chest syndrome in children with sickle cell disease Minireview: Multiomic candidate biomarkers for clinical manifestations of sickle cell severity: Early steps to precision medicine Alloimmunization is associated with older age of transfused red blood cells in sickle cell disease Extracellular hemin crisis triggers acute chest syndrome in sickle mice Heme on innate immunity and inflammation.Minireview: Genetic basis of heterogeneity and severity in sickle cell disease.Genetics of Sickle Cell-Associated Cardiovascular Disease: An Expert Review with Lessons Learned in Africa.Intravascular hemolysis and the pathophysiology of sickle cell disease.Heme as a Target for Therapeutic Interventions.Comments on: "Impact of iron overload on interleukin-10 levels, biochemical parameters and oxidative stress in patients with sickle cell anemia".Clinical Implications of the Association of Fetal Hemoglobin with Peripheral Oxygen Saturation in Sickle Cell Disease.Clinical and genetic predictors of renal dysfunctions in sickle cell anaemia in Cameroon.Acute Chest Syndrome in Children with Sickle Cell Disease.Clinical and genetic factors are associated with pain and hospitalisation rates in sickle cell anaemia in Cameroon.Genome-wide association study to identify variants associated with acute severe vaso-occlusive pain in sickle cell anemia.Heme oxygenase-1 promoter region (GT)n polymorphism associates with increased neuroimmune activation and risk for encephalitis in HIV infection.Haptoglobin and hemopexin inhibit vaso-occlusion and inflammation in murine sickle cell disease: Role of heme oxygenase-1 induction.Control of Oxidative Stress and Inflammation in Sickle Cell Disease with the Nrf2 Activator Dimethyl Fumarate.

P2860

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P2860

Heme oxygenase-1 gene promoter polymorphism is associated with reduced incidence of acute chest syndrome among children with sickle cell disease

http://www.wikidata.org/entity/Q36370871

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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Heme oxygenase-1 gene promoter ...... ldren with sickle cell disease

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Heme oxygenase-1 gene promoter ...... ldren with sickle cell disease

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Heme oxygenase-1 gene promoter ...... ldren with sickle cell disease

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Heme oxygenase-1 gene promoter ...... ldren with sickle cell disease

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Heme oxygenase-1 gene promoter ...... ldren with sickle cell disease

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Amanda B Payne

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Dorothy Ellingsen

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Emily A Barron-Casella

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Genyan Yang

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Heidi A Trau

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James F Casella

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Jennifer Driggers

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Kimberly Jones

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Meredith E Pyle

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Michael R DeBaun

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P2860

A New Statistical Method for Haplotype Reconstruction from Population Data

The International HapMap Project

Ferritin: a cytoprotective antioxidant strategem of endothelium

The role of heme oxygenase-1 promoter polymorphisms in human disease

Microsatellites are molecular clocks that support accurate inferences about history

Accounting for decay of linkage disequilibrium in haplotype inference and missing-data imputation

Heme oxygenase-1 is a modulator of inflammation and vaso-occlusion in transgenic sickle mice.

Elevation of IgE in children with sickle cell disease is associated with doctor diagnosis of asthma and increased morbidity.

Heme oxygenase-1: unleashing the protective properties of heme.

Variations in the heme oxygenase-1 microsatellite polymorphism are associated with plasma CD14 and viral load in HIV-infected African-Americans.

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3822-3828

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scholarly article

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10.1182/BLOOD-2011-06-361642

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18

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Solomon F Ofori-Acquah

Christopher Bean

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2012-09-10T00:00:00Z

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230830670

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22966170

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sickle-cell disease

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3488892

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