NIK-dependent RelB activation defines a unique signaling pathway for the development of V alpha 14i NKT cells. - Wikidata - wikimedia - TriplyDB

NIK-dependent RelB activation defines a unique signaling pathway for the development of V alpha 14i NKT cells.

NIK-dependent RelB activation defines a unique signaling pathway for the development of V alpha 14i NKT cells.

http://www.wikidata.org/entity/Q36370941

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The role of NKT cells in tumor immunity The intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the games Regulating the immune system via IL-15 transpresentation Diacylglycerol Kinases in T Cell Tolerance and Effector Function Central tolerance: learning self-control in the thymus Critical roles of RasGRP1 for invariant NKT cell development Deficiency of transcription factor RelB perturbs myeloid and DC development by hematopoietic-extrinsic mechanisms.IL-15: a central regulator of celiac disease immunopathology.Roles of the NF-kappaB pathway in lymphocyte development and function.Regulation of natural killer T-cell development by deubiquitinase CYLD Commitment toward the natural T (iNKT) cell lineage occurs at the CD4+8+ stage of thymic ontogeny Treatment of murine Th1- and Th2-mediated inflammatory bowel disease with NF-kappa B decoy oligonucleotides.Thymic and peripheral microenvironments differentially mediate development and maturation of iNKT cells by IL-15 transpresentation.Transcriptional control of invariant NKT cell development.Nuclear factor-kappaB inducing kinase is required for graft-versus-host disease.Temporal differences in the dependency on phosphoinositide-dependent kinase 1 distinguish the development of invariant Valpha14 NKT cells and conventional T cells A unique lymphotoxin {alpha}beta-dependent pathway regulates thymic emigration of V{alpha}14 invariant natural killer T cells Tight regulation of diacylglycerol-mediated signaling is critical for proper invariant NKT cell development.Let-7 microRNAs target the lineage-specific transcription factor PLZF to regulate terminal NKT cell differentiation and effector function.TNF Induction of NF-κB RelB Enhances RANKL-Induced Osteoclastogenesis by Promoting Inflammatory Macrophage Differentiation but also Limits It through Suppression of NFATc1 Expression Parallels and distinctions between T and NKT cell development in the thymus.Genetic control of NKT cell numbers.A unique role for ITK in survival of invariant NKT cells associated with the p53-dependent pathway in mice.Molecular and cellular pathogenesis of X-linked lymphoproliferative disease.A role for the IkappaB family member Bcl-3 in the control of central immunologic tolerance Natural Th17 cells are critically regulated by functional medullary thymic microenvironments.Recognition of lipid antigens by T cells.Selection of self-reactive T cells in the thymus.The NFκB-inducing kinase is essential for the developmental programming of skin-resident and IL-17-producing γδ T cells.Abnormal lymphoid organ development in immunodeficient mutant mice.Does the developmental status of Valpha14i NKT cells play a role in disease?Regulation of thymic NKT cell development by the B7-CD28 costimulatory pathway Regulation of Lipid Signaling by Diacylglycerol Kinases during T Cell Development and Function Humanizing mice for the identification of novel anticancer lipids targeting iNKT cells.Differential requirement for CARMA1 in agonist-selected T-cell development.Differential dependence of CD4+CD25+ regulatory and natural killer-like T cells on signals leading to NF-kappaB activation.An essential role for medullary thymic epithelial cells during the intrathymic development of invariant NKT cells.NF-κB RelB negatively regulates osteoblast differentiation and bone formation.Raising the NKT cell family.NF-κB inducing kinase: a key regulator in the immune system and in cancer

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P2860

NIK-dependent RelB activation defines a unique signaling pathway for the development of V alpha 14i NKT cells.

http://www.wikidata.org/entity/Q36370941

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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2003年學術文章

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name

NIK-dependent RelB activation ...... ment of V alpha 14i NKT cells.

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NIK-dependent RelB activation ...... ment of V alpha 14i NKT cells.

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type

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NIK-dependent RelB activation ...... ment of V alpha 14i NKT cells.

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NIK-dependent RelB activation ...... ment of V alpha 14i NKT cells.

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prefLabel

NIK-dependent RelB activation ...... ment of V alpha 14i NKT cells.

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NIK-dependent RelB activation ...... ment of V alpha 14i NKT cells.

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Journal of Experimental Medicine

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NIK-dependent RelB activation ...... ment of V alpha 14i NKT cells.

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Andrew J Leishman

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Carl F Ware

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David Lo

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Dirk Elewaut

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Hilde De Winter

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Kirsten J L Hammond

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Lisa Ma

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Olga Turovskaya

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Raziya B Shaikh

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Stephane Sidobre

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P2860

NF-kappaB-inducing kinase regulates the processing of NF-kappaB2 p100

NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

The I kappa B kinase (IKK) and NF-kappa B: key elements of proinflammatory signalling

Specific inhibition of RelB/p52 transcriptional activity by the C-terminal domain of p100

CD1d-restricted and TCR-mediated activation of valpha14 NKT cells by glycosylceramides

NKT cells derive from double-positive thymocytes that are positively selected by CD1d

Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B

Involvement of distinct cellular compartments in the abnormal lymphoid organogenesis in lymphotoxin-alpha-deficient mice and alymphoplasia (aly) mice defined by the chimeric analysis

Expression of relB is required for the development of thymic medulla and dendritic cells

Phosphorylation of I kappa B alpha precedes but is not sufficient for its dissociation from NF-kappa B

P304

1623-1633

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10.1084/JEM.20030141

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12

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197

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Mitchell Kronenberg

Hilde Cheroutre

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2003-06-01T00:00:00Z

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6966671

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12810685

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2193960

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