B cell-dependent T cell responses: IgM antibodies are required to elicit contact sensitivity. - Wikidata - wikimedia - TriplyDB

B cell-dependent T cell responses: IgM antibodies are required to elicit contact sensitivity.

B cell-dependent T cell responses: IgM antibodies are required to elicit contact sensitivity.

http://www.wikidata.org/entity/Q36371044

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Role of CD1d-restricted NKT cells in microbial immunity From Mysterious Supernatant Entity to miRNA-150 in Antigen-Specific Exosomes: a History of Hapten-Specific T Suppressor Factor Hapten-induced contact hypersensitivity, autoimmune reactions, and tumor regression: plausibility of mediating antitumor immunity The absence of serum IgM enhances the susceptibility of mice to pulmonary challenge with Cryptococcus neoformans.Bridging innate and adaptive antitumor immunity targeting glycans.Regulatory B cells and allergic diseases Contributions of antinucleoprotein IgG to heterosubtypic immunity against influenza virus Natural autoantibodies as tools to predict the outcome of immune response?Augmentation of skin response by exposure to a combination of allergens and irritants - a review.The complement system in regulation of adaptive immunity.CD19 expression in B cells is important for suppression of contact hypersensitivity.New concepts in antibody-mediated immunity.Disruption of the complement anaphylatoxin receptor C5L2 exacerbates inflammation in allergic contact dermatitis Molecular mechanisms of glucocorticoids in the control of inflammation and lymphocyte apoptosis.Impaired humoral immunity and tolerance in K14-VEGFR-3-Ig mice that lack dermal lymphatic drainage Cutaneous immunization rapidly activates liver invariant Valpha14 NKT cells stimulating B-1 B cells to initiate T cell recruitment for elicitation of contact sensitivity.A subset of AID-dependent B-1a cells initiates hypersensitivity and pneumococcal pneumonia resistance.Early immune events in the induction of allergic contact dermatitis Mast cells regulate the magnitude and the cytokine microenvironment of the contact hypersensitivity response.IDO2 Modulates T Cell-Dependent Autoimmune Responses through a B Cell-Intrinsic Mechanism.CD22 expression mediates the regulatory functions of peritoneal B-1a cells during the remission phase of contact hypersensitivity reactions Molecular mechanisms of leukocyte trafficking in T-cell-mediated skin inflammation: insights from intravital imaging.Effector and regulatory mechanisms in allergic contact dermatitis.Immunological mechanisms of contact hypersensitivity in mice.B-1 B lymphocytes require Blimp-1 for immunoglobulin secretion.Contact sensitizer potassium dichromate alters lymphocyte populations in draining lymph nodes and blood in mice.Anti-hapten antibodies in response to skin sensitization.An immune response study of oakmoss absolute and its constituents atranol and chloroatranol.Genetic parameters and across-line SNP associations differ for natural antibody isotypes IgM and IgG in laying hens.Metal nanoparticles in the presence of lipopolysaccharides trigger the onset of metal allergy in mice.Kinetics of cellular responses to intraperitoneal Brugia pahangi infections in normal and immunodeficient mice.Natural Antibodies: Next Steps Toward Translational Investigation.CD1d and CD1d-restricted iNKT-cells play a pivotal role in contact hypersensitivity.Natural Killer Cell Memory: Progress and Implications Invariant NKT cells as initiators, licensors, and facilitators of the adaptive immune response Macrophages and neutrophils are the targets for immune suppression by glucocorticoids in contact allergy.A critical temporal window for selectin-dependent CD4+ lymphocyte homing and initiation of late-phase inflammation in contact sensitivity.Airway hyper-reactivity mediated by B-1 cell immunoglobulin M antibody generating complement C5a at 1 day post-immunization in a murine hapten model of non-atopic asthma.Evaluation of auricular lymph node cell lymphocyte proliferation and cytokine production as non-radioactive endpoints during murine contact allergy.Natural killer cell-mediated contact sensitivity develops rapidly and depends on interferon-α, interferon-γ and interleukin-12.

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B cell-dependent T cell responses: IgM antibodies are required to elicit contact sensitivity.

http://www.wikidata.org/entity/Q36371044

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B cell-dependent T cell respon ...... to elicit contact sensitivity.

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B cell-dependent T cell respon ...... to elicit contact sensitivity.

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Atsuko Itakura

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Ivana Kawikova

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Leonore A Herzenberg

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Marian Szczepanik

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Moe Akahira-Azuma

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Nicole Baumgarth

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Philip W Askenase

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Regis A Campos

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Ryohei F Tsuji

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Vipin Paliwal

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P2860

Natural antibodies with the T15 idiotype may act in atherosclerosis, apoptotic clearance, and protective immunity

Early life origins of asthma

Anti-C5 monoclonal antibody therapy prevents collagen-induced arthritis and ameliorates established disease.

B-1 B cell IgM antibody initiates T cell elicitation of contact sensitivity.

Cardiac resynchronization pacing therapy.

A B cell superantigen-induced persistent "Hole" in the B-1 repertoire

Ly-1 B cells: functionally distinct lymphocytes that secrete IgM autoantibodies.

T cell-dependent mast cell degranulation and release of serotonin in murine delayed-type hypersensitivity.

Anti-phosphorylcholine antibodies of the T15 idiotype are optimally protective against Streptococcus pneumoniae.

Migration and differentiation of autoreactive B-1 cells induced by activated gamma/delta T cells in antierythrocyte immunoglobulin transgenic mice.

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1277-1290

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10.1084/JEM.20020649

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196

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11030338

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12438420

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2193992

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