Alveolar macrophage-mediated killing of Pneumocystis carinii f. sp. muris involves molecular recognition by the Dectin-1 beta-glucan receptor. - Wikidata - wikimedia - TriplyDB

Alveolar macrophage-mediated killing of Pneumocystis carinii f. sp. muris involves molecular recognition by the Dectin-1 beta-glucan receptor.

Alveolar macrophage-mediated killing of Pneumocystis carinii f. sp. muris involves molecular recognition by the Dectin-1 beta-glucan receptor.

http://www.wikidata.org/entity/Q36371523

about

A drug-sensitive genetic network masks fungi from the immune system Pneumocystis murina colonization in immunocompetent surfactant protein A deficient mice following environmental exposure Cdc42 and RhoB activation are required for mannose receptor-mediated phagocytosis by human alveolar macrophages Characterization of a distinct host response profile to Pneumocystis murina asci during clearance of pneumocystis pneumonia Aspergillus fumigatus: principles of pathogenesis and host defense Aspergillus fumigatus triggers inflammatory responses by stage-specific beta-glucan display The beta-glucan receptor dectin-1 recognizes specific morphologies of Aspergillus fumigatus Pathobiology of Pneumocystis pneumonia: life cycle, cell wall and cell signal transduction Innate receptors and cellular defense against pulmonary infections C-type lectins and phagocytosis Echinocandin treatment of pneumocystis pneumonia in rodent models depletes cysts leaving trophic burdens that cannot transmit the infection Stage-specific sampling by pattern recognition receptors during Candida albicans phagocytosis.Novel pneumocystis antigen discovery using fungal surface proteomics.Downregulation of PU.1 leads to decreased expression of Dectin-1 in alveolar macrophages during Pneumocystis pneumonia.Immune modulation with sulfasalazine attenuates immunopathogenesis but enhances macrophage-mediated fungal clearance during Pneumocystis pneumonia.Innate immunity to the pathogenic fungus Coccidioides posadasii is dependent on Toll-like receptor 2 and Dectin-1.Pneumocystis carinii activates the NF-kappaB signaling pathway in alveolar epithelial cells.Rapid host defense against Aspergillus fumigatus involves alveolar macrophages with a predominance of alternatively activated phenotype.In vitro effector activity of Pneumocystis murina-specific T-cytotoxic-1 CD8+ T cells: role of granulocyte-macrophage colony-stimulating factor.Pneumocystis activates human alveolar macrophage NF-kappaB signaling through mannose receptors CD4+ T cell-independent DNA vaccination against opportunistic infections.Role of macrophages in early host resistance to respiratory Acinetobacter baumannii infection.Conserved natural IgM antibodies mediate innate and adaptive immunity against the opportunistic fungus Pneumocystis murina.Toll-like receptor 2 mediates alveolar macrophage response to Pneumocystis murina Dectin-1: a role in antifungal defense and consequences of genetic polymorphisms in humans.Production of functional soluble Dectin-1 glycoprotein using an IRES-linked destabilized-dihydrofolate reductase expression vector IL-33 and M2a alveolar macrophages promote lung defense against the atypical fungal pathogen Pneumocystis murina.Pathological and protective immunity to Pneumocystis infection.Anti-CD20 antibody therapy and susceptibility to Pneumocystis pneumonia Mononuclear phagocyte-mediated antifungal immunity: the role of chemotactic receptors and ligands.Eosinophils Contribute to Early Clearance of Pneumocystis murina Infection.B Lymphocytes Are Required during the Early Priming of CD4+ T Cells for Clearance of Pneumocystis Infection in Mice.Dectin-1 is required for beta-glucan recognition and control of fungal infection.Exogenous heat-killed Escherichia coli improves alveolar macrophage activity and reduces Pneumocystis carinii lung burden in infant mice.Colonization by Pneumocystis jirovecii and its role in disease.Scavenger receptor A dampens induction of inflammation in response to the fungal pathogen Pneumocystis carinii.Immunity against the opportunistic fungal pathogen Pneumocystis.Neutropenia exacerbates infection by Acinetobacter baumannii clinical isolates in a murine wound model.Alveolar macrophages in neonatal mice are inherently unresponsive to Pneumocystis murina infection.Dectin-1 Activation by a Natural Product β-Glucan Converts Immunosuppressive Macrophages into an M1-like Phenotype

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P2860

Alveolar macrophage-mediated killing of Pneumocystis carinii f. sp. muris involves molecular recognition by the Dectin-1 beta-glucan receptor.

http://www.wikidata.org/entity/Q36371523

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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Alveolar macrophage-mediated k ...... Dectin-1 beta-glucan receptor.

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Alveolar macrophage-mediated k ...... Dectin-1 beta-glucan receptor.

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Alveolar macrophage-mediated k ...... Dectin-1 beta-glucan receptor.

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Alveolar macrophage-mediated k ...... Dectin-1 beta-glucan receptor.

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Alveolar macrophage-mediated k ...... Dectin-1 beta-glucan receptor.

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Allen G Harmsen

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Chad Steele

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Gordon D Brown

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Jay K Kolls

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Judd E Shellito

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Luis Marrero

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Mingquan Zheng

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Steve Swain

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P2860

Killing activity of neutrophils is mediated through activation of proteases by K+ flux

Pneumocystis carinii glycoprotein A binds macrophage mannose receptors

Identification of a novel, dendritic cell-associated molecule, dectin-1, by subtractive cDNA cloning

Mannose receptor-mediated regulation of serum glycoprotein homeostasis

Immune recognition. A new receptor for beta-glucans

Mechanisms of host defense against Candida species. I. Phagocytosis by monocytes and monocyte-derived macrophages

Uptake of Pneumocystis carinii mediated by the macrophage mannose receptor

Dectin-1 mediates the biological effects of beta-glucans

Dectin-1 is a major beta-glucan receptor on macrophages

Alveolar macrophage interactions with Pneumocystis carinii.

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