The proinflammatory mediators C3a and C5a are essential for liver regeneration - Wikidata - wikimedia - TriplyDB

The proinflammatory mediators C3a and C5a are essential for liver regeneration

The proinflammatory mediators C3a and C5a are essential for liver regeneration

http://www.wikidata.org/entity/Q36371713

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Function, structure and therapeutic potential of complement C5a receptors Liver immunology and its role in inflammation and homeostasis Complement activation in the context of stem cells and tissue repair Therapeutic regulation of complement in patients with renal disease - where is the promise?The role of complement in the pathogenesis of renal ischemia-reperfusion injury and fibrosis Innate immune cells in liver inflammation The importance of C4d in biopsies of kidney transplant recipients The Immune System in Tissue Environments Regaining Homeostasis after Injury: Is "Inflammation" Always Inflammation?Lysophosphatidic acid alters the expression profiles of angiogenic factors, cytokines, and chemokines in mouse liver sinusoidal endothelial cells UPF2 is a critical regulator of liver development, function and regeneration Complement C3 and C5 play critical roles in traumatic brain cryoinjury: blocking effects on neutrophil extravasation by C5a receptor antagonist.Ketamine-xylazine-acepromazine compared with isoflurane for anesthesia during liver transplantation in rodents.The complement cascade: Yin-Yang in neuroinflammation--neuro-protection and -degeneration.SAK-HV Triggered a Short-period Lipid-lowering Biotherapy Based on the Energy Model of Liver Proliferation via a Novel Pathway.Anaphylatoxin C5a contributes to the pathogenesis of cisplatin-induced nephrotoxicity.Expression patterns and action analysis of genes associated with physiological responses during rat liver regeneration: Innate immune response Hepatic fibrosis is enhanced and accompanied by robust oval cell activation after chronic carbon tetrachloride administration to Egr-1-deficient mice A novel role for the fifth component of complement (C5) in cardiac physiology.The receptor for the complement C3a anaphylatoxin (C3aR) provides host protection against Listeria monocytogenes-induced apoptosis Complement and the alternative pathway play an important role in LPS/D-GalN-induced fulminant hepatic failure.Cascades of transcription regulation during liver regeneration.Dissecting the complement pathway in hepatic injury and regeneration with a novel protective strategy.The complement cascade as a mediator of tissue growth and regeneration.Pathogenesis of B-cell superantigen-induced immune complex-mediated inflammation.Complement in animal development: unexpected roles of a highly conserved pathway.Principles of liver regeneration and growth homeostasis.Complement-mediated inhibition of neovascularization reveals a point of convergence between innate immunity and angiogenesis.Role of CYP2E1 immunoglobulin G4 subclass antibodies and complement in pathogenesis of idiosyncratic drug-induced hepatitis.Interleukin-22 regulates the complement system to promote resistance against pathobionts after pathogen-induced intestinal damage.Complement: a novel factor in basal and ischemia-induced neurogenesis Toll-like receptor 4 and myeloid differentiation factor 88 provide mechanistic insights into the cause and effects of interleukin-6 activation in mouse liver regeneration.T cell-derived lymphotoxin regulates liver regeneration New concepts in liver regeneration.Modulation of the antitumor immune response by complement.The C5a anaphylatoxin receptor (C5aR1) protects against Listeria monocytogenes infection by inhibiting type 1 IFN expression The role of the anaphylatoxins in health and disease.Redox signaling and the innate immune system in alcoholic liver disease.Decay-accelerating factor regulates T-cell immunity in the context of inflammation by influencing costimulatory molecule expression on antigen-presenting cells.Elevated interferon gamma signaling contributes to impaired regeneration in the aged liver.A serine protease isolated from the bristles of the Amazonic caterpillar, Premolis semirufa, is a potent complement system activator.

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P2860

The proinflammatory mediators C3a and C5a are essential for liver regeneration

http://www.wikidata.org/entity/Q36371713

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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2003年學術文章

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name

The proinflammatory mediators C3a and C5a are essential for liver regeneration

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The proinflammatory mediators C3a and C5a are essential for liver regeneration

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The proinflammatory mediators C3a and C5a are essential for liver regeneration

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The proinflammatory mediators C3a and C5a are essential for liver regeneration

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prefLabel

The proinflammatory mediators C3a and C5a are essential for liver regeneration

@ast

The proinflammatory mediators C3a and C5a are essential for liver regeneration

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Journal of Experimental Medicine

P1476

The proinflammatory mediators C3a and C5a are essential for liver regeneration

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P2093

Christoph W Strey

http://www.w3.org/2001/XMLSchema#string

Dimitrios Mastellos

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Linda E Greenbaum

http://www.w3.org/2001/XMLSchema#string

Lynn A Spruce

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Maciej Markiewski

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Ruxandra Tudoran

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P2860

The chemoattractant receptor-like protein C5L2 binds the C3a des-Arg77/acylation-stimulating protein

Interactions among LRF-1, JunB, c-Jun, and c-Fos define a regulatory program in the G1 phase of liver regeneration

Functional receptor for C3a anaphylatoxin is expressed by normal hematopoietic stem/progenitor cells, and C3a enhances their homing-related responses to SDF-1

Structure and biology of complement protein C3, a connecting link between innate and acquired immunity

Tumor necrosis factor induces DNA replication in hepatic cells through nuclear factor kappaB activation

CCAAT enhancer- binding protein beta is required for normal hepatocyte proliferation in mice after partial hepatectomy

Liver regeneration

A novel role of complement: mice deficient in the fifth component of complement (C5) exhibit impaired liver regeneration.

Liver regeneration.

Cytokine regulation of liver injury and repair.

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10.1084/JEM.20030374

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6

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198

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John D. Lambris

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10567211

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12975457

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2194207

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