Deletion of galectin-3 exacerbates microglial activation and accelerates disease progression and demise in a SOD1(G93A) mouse model of amyotrophic lateral sclerosis
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Targeting miR-155 restores abnormal microglia and attenuates disease in SOD1 mice.Regulation of CuZnSOD and its redox signaling potential: implications for amyotrophic lateral sclerosisMicroglia induce motor neuron death via the classical NF-κB pathway in amyotrophic lateral sclerosis.Spinal but not cortical microglia acquire an atypical phenotype with high VEGF, galectin-3 and osteopontin, and blunted inflammatory responses in ALS rats.Characterization of the Contribution of Genetic Background and Gender to Disease Progression in the SOD1 G93A Mouse Model of Amyotrophic Lateral Sclerosis: A Meta-Analysis.Untangling the brain's neuroinflammatory and neurodegenerative transcriptional responses.Neuroimmunity dynamics and the development of therapeutic strategies for amyotrophic lateral sclerosis.Increased Expressions of Plasma Galectin-3 in Patients with Amyotrophic Lateral Sclerosis.Can we switch microglia's phenotype to foster neuroprotection? Focus on multiple sclerosis.The neuro-immune axis: prospect for novel treatments for mental disorders.Amyotrophic Lateral Sclerosis, 2016: existing therapies and the ongoing search for neuroprotection.System xC- is a mediator of microglial function and its deletion slows symptoms in amyotrophic lateral sclerosis mice.Elevated Galectin-3 Levels in the Serum of Patients With Alzheimer's Disease.Differential contribution of microglia and monocytes in neurodegenerative diseases.Galectin-1 deficiency improves axonal swelling of motor neurones in SOD1(G93A) transgenic mice.Microglial Lectins in Health and Neurological Diseases.Investigation of Gal-3 Expression Pattern in Serum and Cerebrospinal Fluid of Patients Suffering From Neurodegenerative DisordersGalectin-3-Mediated Glial Crosstalk Drives Oligodendrocyte Differentiation and (Re)myelination
P2860
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P2860
Deletion of galectin-3 exacerbates microglial activation and accelerates disease progression and demise in a SOD1(G93A) mouse model of amyotrophic lateral sclerosis
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Deletion of galectin-3 exacerb ...... amyotrophic lateral sclerosis
@ast
Deletion of galectin-3 exacerb ...... amyotrophic lateral sclerosis
@en
type
label
Deletion of galectin-3 exacerb ...... amyotrophic lateral sclerosis
@ast
Deletion of galectin-3 exacerb ...... amyotrophic lateral sclerosis
@en
prefLabel
Deletion of galectin-3 exacerb ...... amyotrophic lateral sclerosis
@ast
Deletion of galectin-3 exacerb ...... amyotrophic lateral sclerosis
@en
P2093
P2860
P356
P1433
P1476
Deletion of galectin-3 exacerb ...... amyotrophic lateral sclerosis
@en
P2093
Bruce J Lerman
Daniel K Hsu
Eric P Hoffman
Fu-Tong Liu
Jeffrey D Rothstein
Khaled Bouri
Margaret L Sutherland
Susan M Knoblach
P2860
P304
P356
10.1002/BRB3.75
P577
2012-07-23T00:00:00Z