Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern. - Wikidata - wikimedia - TriplyDB

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

http://www.wikidata.org/entity/Q36376630

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Caspase-activated DNase is required for maintenance of tolerance to lupus nuclear autoantigens The macrophages in rheumatic diseases Defects in Germinal Center Selection in SLE B cell receptor editing in tolerance and autoimmunity IDO2: A Pathogenic Mediator of Inflammatory Autoimmunity Spontaneous autoimmunity in mice that carry an IghV partial transgene: a required arginine in VHCDR3.CXCR4 and a cell-extrinsic mechanism control immature B lymphocyte egress from bone marrow Genes within the Idd5 and Idd9/11 diabetes susceptibility loci affect the pathogenic activity of B cells in nonobese diabetic mice.Disrupted Mer receptor tyrosine kinase expression leads to enhanced MZ B-cell responses.Receptor editing in lymphocyte development and central tolerance.Activation-induced deaminase-deficient MRL/lpr mice secrete high levels of protective antibodies against lupus nephritis.TLR7 influences germinal center selection in murine SLE.An entity evolving into a community: defining the common ancestor and evolutionary trajectory of chronic lymphocytic leukemia stereotyped subset #4.Positive selection of natural poly-reactive B cells in the periphery occurs independent of heavy chain allelic inclusion Development and selection of marginal zone B cells.Influence of interleukin-2 deficiency on the generation of autoimmune B cells.Light chain editing generates polyreactive antibodies in chronic graft-versus-host reaction.Class A scavenger receptors regulate tolerance against apoptotic cells, and autoantibodies against these receptors are predictive of systemic lupus Reduced receptor editing in lupus-prone MRL/lpr mice Abrogation of lupus nephritis in activation-induced deaminase-deficient MRL/lpr mice.Dual-reactive B cells are autoreactive and highly enriched in the plasmablast and memory B cell subsets of autoimmune mice.The Yaa locus and IFN-α fine-tune germinal center B cell selection in murine systemic lupus erythematosus Human autoantibody silencing by immunoglobulin light chains Use of IGHV3-21 in chronic lymphocytic leukemia is associated with high-risk disease and reflects antigen-driven, post-germinal center leukemogenic selection.The evolution of mathematical immunology.Accelerated atherosclerosis in ApoE deficient lupus mouse models.Intrafollicular location of marginal zone/CD1d(hi) B cells is associated with autoimmune pathology in a mouse model of lupus Temporal dynamics of clonal evolution in chronic lymphocytic leukemia with stereotyped IGHV4-34/IGKV2-30 antigen receptors: longitudinal immunogenetic evidence.Prolactin alters the mechanisms of B cell tolerance induction.Old mice retain bone marrow B1 progenitors, but lose B2 precursors, and exhibit altered immature B cell phenotype and light chain usage.Deviation of the B cell pathway in senescent mice is associated with reduced surrogate light chain expression and altered immature B cell generation, phenotype, and light chain expression Dual immunoglobulin light chain B cells: Trojan horses of autoimmunity?Allelic exclusion of immunoglobulin genes: models and mechanisms.Polyreactive antibodies in adaptive immune responses to viruses.The role of activation-induced deaminase in lupus nephritis.Contributions of B cells to lupus pathogenesis Mechanisms That Shape Human Antibody Repertoire Development in Mice Transgenic for Human Ig H and L Chain Loci.Btk regulates localization, in vivo activation, and class switching of anti-DNA B cells.Autoreactive MZ and B-1 B-cell activation by Faslpr is coincident with an increased frequency of apoptotic lymphocytes and a defect in macrophage clearance.Activation-induced deaminase heterozygous MRL/lpr mice are delayed in the production of high-affinity pathogenic antibodies and in the development of lupus nephritis.

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P2860

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

http://www.wikidata.org/entity/Q36376630

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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2002年學術文章

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name

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

@ast

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

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type

label

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

@ast

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

@en

prefLabel

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

@ast

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

@en

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P1476

Anti-DNA B cells in MRL/lpr mice show altered differentiation and editing pattern.

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P2093

Dongyao Ni

http://www.w3.org/2001/XMLSchema#string

Hui Li

http://www.w3.org/2001/XMLSchema#string

Martin Weigert

http://www.w3.org/2001/XMLSchema#string

Yijin Li

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P2860

Marginal-zone B cells

Atypical VH-D-JH rearrangements in newborn autoimmune MRL mice.

Generation of a large combinatorial library of the immunoglobulin repertoire in phage lambda. 1989.

Activation and differentiation of autoreactive B-1 cells by interleukin 10 induce autoimmune hemolytic anemia in Fas-deficient antierythrocyte immunoglobulin transgenic mice

Genesis and evolution of antichromatin autoantibodies in murine lupus implicates T-dependent immunization with self antigen.

Stochastic control of anti-Sm autoantibodies in MRL/Mp-lpr/lpr mice.

Anti-DNA antibodies from autoimmune mice arise by clonal expansion and somatic mutation.

Variable region sequences of murine IgM anti-IgG monoclonal autoantibodies (rheumatoid factors). II. Comparison of hybridomas derived by lipopolysaccharide stimulation and secondary protein immunization.

Receptor editing: an approach by autoreactive B cells to escape tolerance.

B lymphocytes may escape tolerance by revising their antigen receptors

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12

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12486097

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2196070

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