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Plasma matrix metalloproteinase-9 and left ventricular remodelling after acute myocardial infarction in man: a prospective cohort study

Plasma matrix metalloproteinase-9 and left ventricular remodelling after acute myocardial infarction in man: a prospective cohort study

http://www.wikidata.org/entity/Q36387323

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The circular relationship between matrix metalloproteinase-9 and inflammation following myocardial infarction Translating Koch's postulates to identify matrix metalloproteinase roles in postmyocardial infarction remodeling: cardiac metalloproteinase actions (CarMA) postulates Molecular modeling study for inhibition mechanism of human chymase and its application in inhibitor design Coordinated modular functionality and prognostic potential of a heart failure biomarker-driven interaction network.Biomarkers in acute myocardial infarction.Novel biomarkers for cardiovascular risk prediction.Membrane-associated matrix proteolysis and heart failure.Plasma levels of matrix metalloproteinase 9 in patients undergoing off-pump coronary artery bypass grafting.Combining experimental and mathematical modeling to reveal mechanisms of macrophage-dependent left ventricular remodeling Plasma tissue inhibitor of metalloproteinase-1 and matrix metalloproteinase-9: novel indicators of left ventricular remodelling and prognosis after acute myocardial infarction.The influence of autologous bone marrow stem cell transplantation on matrix metalloproteinases in patients treated for acute ST-elevation myocardial infarction.Cardiac extracellular proteome profiling and membrane topology analysis using glycoproteomics.Interferon-gamma increases the ratio of matrix metalloproteinase-9/tissue inhibitor of metalloproteinase-1 in peripheral monocytes from patients with coronary artery disease A panel of 4 microRNAs facilitates the prediction of left ventricular contractility after acute myocardial infarction.Serum MMP-8: a novel indicator of left ventricular remodeling and cardiac outcome in patients after acute myocardial infarction.Markers of progenitor cell recruitment and differentiation rise early during ischemia and continue during resuscitation in a porcine acute ischemia model.Impact of percutaneous coronary intervention on biomarker levels in patients in the subacute phase following myocardial infarction: the Occluded Artery Trial (OAT) biomarker ancillary study Myocardial remodeling: cellular and extracellular events and targets.Finding off-targets, biological pathways, and target diseases for chymase inhibitors via structure-based systems biology approach.Changes in the myocardial interstitium and contribution to the progression of heart failure.Correlations between serum inflammation factors and left ventricular remodeling in acute ST segment elevation myocardial infarction.Matrix metalloproteinase inhibitors as investigative tools in the pathogenesis and management of vascular disease.Prognostic impact of neutrophil gelatinase-associated lipocalin and B-type natriuretic in patients with ST-elevation myocardial infarction treated by primary PCI: a prospective observational cohort study Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction.Matrix metalloproteinase (MMP)-9: a proximal biomarker for cardiac remodeling and a distal biomarker for inflammation.Circulating stromelysin-1 (MMP-3): a novel predictor of LV dysfunction, remodelling and all-cause mortality after acute myocardial infarction Matrix metalloproteinases 2 and 9 are differentially expressed in patients with indeterminate and cardiac clinical forms of Chagas disease Potential novel pharmacological therapies for myocardial remodelling.Ischemia/Reperfusion Injury following Acute Myocardial Infarction: A Critical Issue for Clinicians and Forensic Pathologists.Established and novel biomarkers in ST-elevation myocardial infarction.Cardiac fibroblasts, fibrosis and extracellular matrix remodeling in heart disease.Neutrophil roles in left ventricular remodeling following myocardial infarction.Prognostic value of biological markers in myocardial infarction patients.Matrix metalloproteinases are involved in cardiovascular diseases.Matrix Metalloproteinases in Myocardial Infarction and Heart Failure.Synergic and antagonistic relationship between MMP-2 and MMP-9 with fibrosis and inflammation in Chagas' cardiomyopathy.Matrix Metalloproteinase 3 Gene Polymorphism and Its Level Predict Morbidity After Acute Myocardial Infarction.Association between matrix metalloproteinase-9 and worsening heart failure events in patients with chronic heart failure.Plasma fractionation enriches post-myocardial infarction samples prior to proteomics analysis.Effects of N-acetylcysteine on the cardiac remodeling biomarkers and major adverse events following acute myocardial infarction: a randomized clinical trial.

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Plasma matrix metalloproteinase-9 and left ventricular remodelling after acute myocardial infarction in man: a prospective cohort study

http://www.wikidata.org/entity/Q36387323

description

2007 nî lūn-bûn

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European Heart Journal

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Plasma matrix metalloproteinas ...... an: a prospective cohort study

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P2093

Dominic Kelly

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Gillian Cockerill

http://www.w3.org/2001/XMLSchema#string

Iain B Squire

http://www.w3.org/2001/XMLSchema#string

Matt Thompson

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Sohail Khan

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P2860

The inflammatory response in myocardial infarction.

Proteinases in chronic obstructive pulmonary disease.

Smoking, metalloproteinases, and vascular disease.

Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction

Ventricular remodeling after myocardial infarction. Experimental observations and clinical implications.

Plasma concentrations and genetic variation of matrix metalloproteinase 9 and prognosis of patients with cardiovascular disease.

Cell biology of wound healing.

Near-infrared fluorescent imaging of matrix metalloproteinase activity after myocardial infarction.

Selective matrix metalloproteinase inhibition reduces left ventricular remodeling but does not inhibit angiogenesis after myocardial infarction.

N-terminal pro-B-type natriuretic peptide and long-term mortality in acute coronary syndromes.

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