Re-expression of miR-200 by novel approaches regulates the expression of PTEN and MT1-MMP in pancreatic cancer. - Wikidata - wikimedia - TriplyDB

Re-expression of miR-200 by novel approaches regulates the expression of PTEN and MT1-MMP in pancreatic cancer.

Re-expression of miR-200 by novel approaches regulates the expression of PTEN and MT1-MMP in pancreatic cancer.

http://www.wikidata.org/entity/Q36402024

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Novel therapeutic targets for pancreatic cancer The Role of microRNAs in the Diagnosis and Treatment of Pancreatic Adenocarcinoma The miRacle in Pancreatic Cancer by miRNAs: Tiny Angels or Devils in Disease Progression MicroRNAs: novel players in cancer diagnosis and therapies.Therapeutic applications of curcumin for patients with pancreatic cancer.Deregulation of miR-146a expression in a mouse model of pancreatic cancer affecting EGFR signaling.The role of microRNAs in diabetic nephropathy.Catalpol suppresses proliferation and facilitates apoptosis of OVCAR-3 ovarian cancer cells through upregulating microRNA-200 and downregulating MMP-2 expression The Role of Nutraceuticals in Pancreatic Cancer Prevention and Therapy: Targeting Cellular Signaling, MicroRNAs, and Epigenome.Mechanisms underlying the cytotoxicity of a novel quinazolinedione-based redox modulator, QD232, in pancreatic cancer cells MiR-200a inhibits epithelial-mesenchymal transition of pancreatic cancer stem cell.Clinical implications of miRNAs in the pathogenesis, diagnosis and therapy of pancreatic cancer.Potential application of miRNAs as diagnostic and therapeutic tools in chronic pancreatitis.Sp1-driven up-regulation of miR-19a decreases RHOB and promotes pancreatic cancer.Mechanistic insights into the role of microRNAs in cancer: influence of nutrient crosstalk.MiR-200a Suppresses the Proliferation and Metastasis in Pancreatic Ductal Adenocarcinoma through Downregulation of DEK Gene.Role of BioResponse 3,3'-Diindolylmethane in the Treatment of Human Prostate Cancer: Clinical Experience.Matched miRNA and mRNA signatures from an hESC-based in vitro model of pancreatic differentiation reveal novel regulatory interactions.Direct targeting of SUZ12/ROCK2 by miR-200b/c inhibits cholangiocarcinoma tumourigenesis and metastasis.Role of microRNA-mediated MMP regulation in the treatment and diagnosis of malignant tumors.MicroRNA-1291 targets the FOXA2-AGR2 pathway to suppress pancreatic cancer cell proliferation and tumorigenesis C66 ameliorates diabetic nephropathy in mice by both upregulating NRF2 function via increase in miR-200a and inhibiting miR-21 Globally increased ultraconserved noncoding RNA expression in pancreatic adenocarcinoma.Interleukin-9 Promotes Pancreatic Cancer Cells Proliferation and Migration via the miR-200a/Beta-Catenin Axis.Epigenetic regulation of miRNA-cancer stem cells nexus by nutraceuticals.Curcumin, a component of turmeric: from farm to pharmacy.Effects of dietary phytophenols on the expression of microRNAs involved in mammalian cell homeostasis.An Update on Potential Perspectives of Glucosinolates on Protection against Microbial Pathogens and Endocrine Dysfunctions in Humans.NO• and Pancreatic Cancer: A Complex Interaction with Therapeutic Potential.MicroRNA-200a Regulates the Development of Mandibular Condylar Cartilage.MiR200c targets IRS1 and suppresses prostate cancer cell growth.MiR-200a is involved in proliferation and apoptosis in the human endometrial adenocarcinoma cell line HEC-1B by targeting the tumor suppressor PTEN.Effects of resveratrol, curcumin, berberine and other nutraceuticals on aging, cancer development, cancer stem cells and microRNAs.Molecular docking and inhibition of matrix metalloproteinase-2 by novel difluorinatedbenzylidene curcumin analog.Combined treatment with artesunate and bromocriptine has synergistic anticancer effects in pituitary adenoma cell lines Endothelial Nitric Oxide Synthase Traffic Inducer (NOSTRIN) is a Negative Regulator of Disease Aggressiveness in Pancreatic Cancer.Curcumin induces G2/M arrest, apoptosis, NF-κB inhibition, and expression of differentiation genes in thyroid carcinoma cells.miR-21a negatively modulates tumor suppressor genes PTEN and miR-200c and further promotes the transformation of M2 macrophages.

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Re-expression of miR-200 by novel approaches regulates the expression of PTEN and MT1-MMP in pancreatic cancer.

http://www.wikidata.org/entity/Q36402024

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2012 nî lūn-bûn

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Re-expression of miR-200 by no ...... MT1-MMP in pancreatic cancer.

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Re-expression of miR-200 by no ...... MT1-MMP in pancreatic cancer.

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Re-expression of miR-200 by no ...... MT1-MMP in pancreatic cancer.

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Re-expression of miR-200 by no ...... MT1-MMP in pancreatic cancer.

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Re-expression of miR-200 by no ...... MT1-MMP in pancreatic cancer.

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Re-expression of miR-200 by no ...... MT1-MMP in pancreatic cancer.

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Re-expression of miR-200 by no ...... MT1-MMP in pancreatic cancer.

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Azfur S Ali

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Farah Logna

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Fazlul H Sarkar

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Omar Soubani

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Philip A Philip

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Shadan Ali

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P2860

Collagen regulation of let-7 in pancreatic cancer involves TGF-β1-mediated membrane type 1-matrix metalloproteinase expression

Gemcitabine sensitivity can be induced in pancreatic cancer cells through modulation of miR-200 and miR-21 expression by curcumin or its analogue CDF

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

Cancer statistics, 2012

Anti-tumor activity of a novel compound-CDF is mediated by regulating miR-21, miR-200, and PTEN in pancreatic cancer

Targeting bone remodeling by isoflavone and 3,3'-diindolylmethane in the context of prostate cancer bone metastasis

The EMT-activator ZEB1 promotes tumorigenicity by repressing stemness-inhibiting microRNAs

Epithelial to mesenchymal transition is mechanistically linked with stem cell signatures in prostate cancer cells

Proteases in invasion: matrix metalloproteinases.

Differentially expressed miRNAs in the plasma may provide a molecular signature for aggressive pancreatic cancer

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22637745

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