NKT cells promote antibody-induced joint inflammation by suppressing transforming growth factor beta1 production - Wikidata - wikimedia - TriplyDB

NKT cells promote antibody-induced joint inflammation by suppressing transforming growth factor beta1 production

NKT cells promote antibody-induced joint inflammation by suppressing transforming growth factor beta1 production

http://www.wikidata.org/entity/Q36402567

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Natural killer T cells and rheumatoid arthritis: friend or foe?K/BxN Serum-Transfer Arthritis as a Model for Human Inflammatory Arthritis The Role of Invariant Natural Killer T Cells in Dendritic Cell Licensing, Cross-Priming, and Memory CD8(+) T Cell Generation Immunomodulation by mesenchymal stem cells in treating human autoimmune disease-associated lung fibrosis The effect of intracellular trafficking of CD1d on the formation of TCR repertoire of NKT cells.The role of natural killer T cells in costimulation blockade-based mixed chimerism Direct engagement of TLR4 in invariant NKT cells regulates immune diseases by differential IL-4 and IFN-γ production in mice.Activation of Invariant NKT cells with glycolipid ligand α-galactosylceramide ameliorates glucose-6-phosphate isomerase peptide-induced arthritis Differential antitumor immunity mediated by NKT cell subsets in vivo Invariant NKT Cell Lines Derived from the NOD·H2 Mouse Enhance Autoimmune Thyroiditis.FcgammaRIII engagement provides activating signals to NKT cells in antibody-induced joint inflammation.Natural killer T (NKT) cells attenuate bleomycin-induced pulmonary fibrosis by producing interferon-gamma.The split personality of NKT cells in malignancy, autoimmune and allergic disorders Comparison of Invariant NKT Cells with Conventional T Cells by Using Gene Set Enrichment Analysis (GSEA).Neutrophil-derived leukotriene B4 is required for inflammatory arthritis Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.TLR4-mediated IL-12 production enhances IFN-γ and IL-1β production, which inhibits TGF-β production and promotes antibody-induced joint inflammation.Antibody-induced arthritis: disease mechanisms and genes involved at the effector phase of arthritis.Natural killer T cells in health and disease.Therapeutic potential of CD1d-restricted invariant natural killer T cell-based treatment for autoimmune diseases.The adaptor molecule signaling lymphocytic activation molecule (SLAM)-associated protein (SAP) is essential in mechanisms involving the Fyn tyrosine kinase for induction and progression of collagen-induced arthritis.Mutation of tyrosine 145 of lymphocyte cytosolic protein 2 protects mice from anaphylaxis and arthritis.Invariant NK T cells: potential for immunotherapeutic targeting with glycolipid antigens.Therapeutic manipulation of natural killer (NK) T cells in autoimmunity: are we close to reality?Activation of iNKT Cells Prevents Salmonella-Enterocolitis and Salmonella-Induced Reactive Arthritis by Downregulating IL-17-Producing γδT Cells.The requirement of natural killer T-cells in tolerogenic APCs-mediated suppression of collagen-induced arthritis.Exposure of polyethylene particles induces interferon-γ expression in a natural killer T lymphocyte and dendritic cell coculture system in vitro: a preliminary study.FTY720, a sphingosine 1-phosphate receptor modulator, inhibits CD1d-restricted NKT cells by suppressing cytokine production but not migration.Lineage Differentiation Program of Invariant Natural Killer T Cells.Palmitate inhibits arthritis by inducing t-bet and gata-3 mRNA degradation in iNKT cells via IRE1α-dependent decay.Sodium chloride inhibits IFN-γ, but not IL-4, production by invariant NKT cells.CD1d-Restricted Type II NKT Cells Reactive With Endogenous Hydrophobic Peptides.Targeting IgG in Arthritis: Disease Pathways and Therapeutic Avenues.Natural killer T-cell characterization through gene expression profiling: an account of versatility bridging T helper type 1 (Th1), Th2 and Th17 immune responses.Activation of natural killer T cells by α-carba-GalCer (RCAI-56), a novel synthetic glycolipid ligand, suppresses murine collagen-induced arthritis.It Takes “Guts” to Cause Joint Inflammation: Role of Innate-Like T Cells.

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P2860

NKT cells promote antibody-induced joint inflammation by suppressing transforming growth factor beta1 production

http://www.wikidata.org/entity/Q36402567

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NKT cells promote antibody-ind ...... growth factor beta1 production

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NKT cells promote antibody-ind ...... growth factor beta1 production

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NKT cells promote antibody-ind ...... growth factor beta1 production

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NKT cells promote antibody-ind ...... growth factor beta1 production

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Doo Hyun Chung

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Hye Sook Min

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Hye Young Kim

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Hyun Jung Kim

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Sanghee Kim

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Seong Hoe Park

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Weon Seo Park

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P2860

Colony-stimulating factor-1-dependent macrophages are responsible for IVIG protection in antibody-induced autoimmune disease.

Cytokines and autoimmunity.

The regulatory role of Valpha14 NKT cells in innate and acquired immune response.

Activation of natural killer T cells in NZB/W mice induces Th1-type immune responses exacerbating lupus

An invariant T cell receptor alpha chain is used by a unique subset of major histocompatibility complex class I-specific CD4+ and CD4-8- T cells in mice and humans.

Genetic influences on the end-stage effector phase of arthritis.

Natural killer T cell activation protects mice against experimental autoimmune encephalomyelitis.

Transforming growth factor-beta production and myeloid cells are an effector mechanism through which CD1d-restricted T cells block cytotoxic T lymphocyte-mediated tumor immunosurveillance: abrogation prevents tumor recurrence.

Cutaneous immunization rapidly activates liver invariant Valpha14 NKT cells stimulating B-1 B cells to initiate T cell recruitment for elicitation of contact sensitivity.

Protective effect of transforming growth factor beta 1 on experimental autoimmune diseases in mice.

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15630137

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