Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
about
K/BxN Serum-Transfer Arthritis as a Model for Human Inflammatory ArthritisNeutrophils in a mouse model of autoantibody-mediated arthritis: critical producers of Fc receptor gamma, the receptor for C5a, and lymphocyte function-associated antigen 1Gut-residing segmented filamentous bacteria drive autoimmune arthritis via T helper 17 cellsIFN-gamma and STAT1 arrest monocyte migration and modulate RAC/CDC42 pathwaysMicroglia activated with the toll-like receptor 9 ligand CpG attenuate oligomeric amyloid {beta} neurotoxicity in in vitro and in vivo models of Alzheimer's disease.IL-17-producing T cells can augment autoantibody-induced arthritisCD4+ and CD8+ T cells can act separately in tumour rejection after immunization with murine pneumotropic virus chimeric Her2/neu virus-like particlesCentral nervous system (CNS)-resident natural killer cells suppress Th17 responses and CNS autoimmune pathology.Genetic and therapeutic targeting of properdin in mice prevents complement-mediated tissue injury.How punctual ablation of regulatory T cells unleashes an autoimmune lesion within the pancreatic islets1-Methyl-tryptophan synergizes with methotrexate to alleviate arthritis in a mouse model of arthritis.Abrogation of antibody-induced arthritis in mice by a self-activating viridin prodrug and association with impaired neutrophil and endothelial cell functionDeletion of Syk in neutrophils prevents immune complex arthritisCD11c-mediated deletion of Flip promotes autoreactivity and inflammatory arthritis.A tolerogenic role for Toll-like receptor 9 is revealed by B-cell interaction with DNA complexes expressed on apoptotic cellsEctopic restriction of DNA repair reveals that UNG2 excises AID-induced uracils predominantly or exclusively during G1 phasePromotion of Inflammatory Arthritis by Interferon Regulatory Factor 5 in a Mouse Model.Neutrophils orchestrate their own recruitment in murine arthritis through C5aR and FcγR signaling.Organ-specific features of natural killer cellsProlonged contact with dendritic cells turns lymph node-resident NK cells into anti-tumor effectors.Anti-arthritis activity of cationic materials.Inflammation and the emerging role of the toll-like receptor system in acute brain ischemia.Cross-regulation of signaling pathways by interferon-gamma: implications for immune responses and autoimmune diseases.The role of toll-like receptors in rheumatoid arthritisNatural killer cells in human autoimmune disordersThe defensive alliance between neutrophils and NK cells as a novel arm of innate immunity.Immunoregulation of GVHD by triggering the innate immune system with CpG.B-cell-mediated strategies to fight chronic allograft rejectionTLRs, future potential therapeutic targets for RA.Toll-like receptors: promising therapeutic targets for inflammatory diseases.The TGF-β superfamily cytokine, MIC-1/GDF15: a pleotrophic cytokine with roles in inflammation, cancer and metabolism.Unc93B1 biases Toll-like receptor responses to nucleic acid in dendritic cells toward DNA- but against RNA-sensing.Local Delivery of the Toll-Like Receptor 9 Ligand CpG Downregulates Host Immune and Inflammatory Responses, Ameliorating Established Leishmania (Viannia) panamensis Chronic Infection.The impact of age and gut microbiota on Th17 and Tfh cells in K/BxN autoimmune arthritis.Interleukin-30: a novel antiinflammatory cytokine candidate for prevention and treatment of inflammatory cytokine-induced liver injuryCoordination between TLR9 signaling in macrophages and CD3 signaling in T cells induces robust expression of IL-30.The role of natural killer (NK) and NK T cells in the loss of tolerance in murine primary biliary cirrhosis.Therapeutic Modulation of Plasmacytoid Dendritic Cells in Experimental Arthritis.Immunostimulatory oligodeoxynucleotide containing TTTCGTTT motif from Lactobacillus rhamnosus GG DNA potentially suppresses OVA-specific IgE production in mice.Targeting IgG in Arthritis: Disease Pathways and Therapeutic Avenues.
P2860
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P2860
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
@ast
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
@en
type
label
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
@ast
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
@en
prefLabel
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
@ast
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk.
@en
P2093
P2860
P356
P1476
Inflammatory arthritis can be reined in by CpG-induced DC-NK cell cross talk
@en
P2093
Amanda Blasius
Christophe Benoist
Diane Mathis
Heloisa Sawaya
Hsin-Jung Wu
John Carulli
Leonid Gorelik
Margot Brickelmaier
Umar Mahmood
P2860
P304
P356
10.1084/JEM.20070285
P407
P577
2007-07-23T00:00:00Z