Mechanisms of liver injury. III. Role of glutathione redox status in liver injury. - Wikidata - wikimedia - TriplyDB

Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

http://www.wikidata.org/entity/Q36406300

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Mechanisms of Diabetes-Induced Liver Damage: The role of oxidative stress and inflammation Redox-based regulation of apoptosis: S-glutathionylation as a regulatory mechanism to control cell death MiR-214 promotes the alcohol-induced oxidative stress via down-regulation of glutathione reductase and cytochrome P450 oxidoreductase in liver cells Severe liver steatosis correlates with nitrosative and oxidative stress in rats S-glutathionylation: from molecular mechanisms to health outcomes Oxidised- and total non-protein bound glutathione and related thiols in gallbladder bile of patients with various gastrointestinal disorders.Oxidative stress promotes D-GalN/LPS-induced acute hepatotoxicity by increasing glycogen synthase kinase 3β activity.Redox regulation of tumor necrosis factor signaling Mitochondrial glutathione, a key survival antioxidant.Fast food diet with CCl4 micro-dose induced hepatic-fibrosis--a novel animal model.Reactive oxygen and nitrogen species in steatotic hepatocytes: a molecular perspective on the pathophysiology of ischemia-reperfusion injury in the fatty liver.Effects of a novel cystine-based glutathione precursor on oxidative stress in vascular smooth muscle cells Exposure of precision-cut rat liver slices to ethanol accelerates fibrogenesis Emodin prevents intrahepatic fat accumulation, inflammation and redox status imbalance during diet-induced hepatosteatosis in rats.Regulation of mitochondrial glutathione redox status and protein glutathionylation by respiratory substrates Analysis of intracellular reducing levels in human hepatocytes on three-dimensional focusing microchip.Therapeutic detoxification of quercetin against carbon tetrachloride-induced acute liver injury in mice and its mechanism.N-acetylcysteine inhibits the up-regulation of mitochondrial biogenesis genes in livers from rats fed ethanol chronically.Profiling the reactive metabolites of xenobiotics using metabolomic technologies Role of cAMP-responsive element-binding protein (CREB)-regulated transcription coactivator 3 (CRTC3) in the initiation of mitochondrial biogenesis and stress response in liver cells Predicting the Kinetic Properties Associated with Redox Imbalance after Oxidative Crisis in G6PD-Deficient Erythrocytes: A Simulation Study A novel cystine based antioxidant attenuates oxidative stress and hepatic steatosis in diet-induced obese mice.Inhibition of mitochondrial respiration and rapid depletion of mitochondrial glutathione by β-phenethyl isothiocyanate: mechanisms for anti-leukemia activity.The effect of low or high molecular weight oat beta-glucans on the inflammatory and oxidative stress status in the colon of rats with LPS-induced enteritis.Interstrain differences in liver injury and one-carbon metabolism in alcohol-fed mice.Reactive oxygen species scavenger N-acetyl cysteine reduces methamphetamine-induced hyperthermia without affecting motor activity in mice.Effects of acute exercise on liver function and blood redox status in heavy drinkers.Myeloperoxidase-Hepatocyte-Stellate Cell Cross Talk Promotes Hepatocyte Injury and Fibrosis in Experimental Nonalcoholic Steatohepatitis Oxidative stress contributes to liver damage in a murine model of alpha-1-antitrypsin deficiency.Mouse liver protein sulfhydryl depletion after acetaminophen exposure.Inhibition of Hepatic Ischemic Reperfusion Injury Using Saline Exposed to Electron Discharge in a Rat Model.Oxidative stress as a mechanism of valproic acid-associated hepatotoxicity.Regulation of H(2)O(2)-induced necrosis by PKC and AMP-activated kinase signaling in primary cultured hepatocytes.Gain-of-function mutations in transient receptor potential C6 (TRPC6) activate extracellular signal-regulated kinases 1/2 (ERK1/2)Molecular mechanisms underlying chemical liver injury.Curcumin pretreatment prevents potassium dichromate-induced hepatotoxicity, oxidative stress, decreased respiratory complex I activity, and membrane permeability transition pore opening Inhibition of apoptotic signalling in spermine-treated vascular smooth muscle cells by a novel glutathione precursor.Dynamics of glutathione and ophthalmate traced with 2H-enriched body water in rats and humans.The energy-redox axis in aging and age-related neurodegeneration.Determination of GSH, GSSG, and GSNO using HPLC with electrochemical detection.

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

http://www.wikidata.org/entity/Q36406300

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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2006年學術文章

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2006年學術文章

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

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AJPGI.00001.2006

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Mechanisms of liver injury. III. Role of glutathione redox status in liver injury.

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Behnam Saberi

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Derick Han

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Naoko Hanawa

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Neil Kaplowitz

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P2860

Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

Direct evidence that sulfhydryl groups of Keap1 are the sensors regulating induction of phase 2 enzymes that protect against carcinogens and oxidants

CD95-Mediated murine hepatic apoptosis requires an intact glutathione status

Redox environment of the cell as viewed through the redox state of the glutathione disulfide/glutathione couple

Regulation of protein function by S-glutathiolation in response to oxidative and nitrosative stress.

Dual regulation of caspase activity by hydrogen peroxide: implications for apoptosis.

Sites and mechanisms of aconitase inactivation by peroxynitrite: modulation by citrate and glutathione.

Hepatocyte-specific inhibition of NF-kappaB leads to apoptosis after TNF treatment, but not after partial hepatectomy.

Depletion of hepatic glutathione prevents death receptor-dependent apoptotic and necrotic liver injury in mice

Mitochondrial dysfunction in hepatitis C.

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10.1152/AJPGI.00001.2006

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