A review of the molecular mechanism of HSV-1 latency.

A review of the molecular mechanism of HSV-1 latency.

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http://www.wikidata.org/entity/Q36408944
Gene array analysis reveals changes in peripheral nervous system gene expression following stimuli that result in reactivation of latent herpes simplex virus type 1: induction of transcription factor Bcl-3.Herpes latency, meningitis, radiculomyelopathy and disseminated infection.Use of differential display reverse transcription-PCR to reveal cellular changes during stimuli that result in herpes simplex virus type 1 reactivation from latency: upregulation of immediate-early cellular response genes TIS7, interferon, and interOcular infection of mice with an avirulent recombinant HSV-1 expressing IL-4 and an attenuated HSV-1 strain generates virulent recombinants in vivo.Selection of a nonconsensus branch point is influenced by an RNA stem-loop structure and is important to confer stability to the herpes simplex virus 2-kilobase latency-associated transcriptInterference with major histocompatibility complex class II-restricted antigen presentation in the brain by herpes simplex virus type 1: a possible mechanism of evasion of the immune response.Prospects for developing an effective vaccine against ocular herpes simplex virus infection.Induction of reactivation of herpes simplex virus in murine sensory ganglia in vivo by cadmium.Herpes simplex virus type 1 latency-associated transcript (LAT) promoter deletion mutants can express a 2-kilobase transcript mapping to the LAT region.Herpes simplex virus type 1 mutant strain in1814 establishes a unique, slowly progressing infection in SCID mice.Microarray analysis of host gene expression for comparison between naïve and HSV-1 latent rabbit trigeminal ganglia.A herpes simplex virus type 1 mutant lacking the ICP0 introns reactivates with normal efficiencyIn vivo and in vitro reactivation impairment of a herpes simplex virus type 1 latency-associated transcript variant in a rabbit eye model.Weight loss and reduced body temperature determine humane endpoints in a mouse model of ocular herpesvirus infectionCharacterization of a nerve growth factor-inducible cellular activity that enhances herpes simplex virus type 1 gene expression and replication of an ICP0 null mutant in cells of neural lineage.The role of LAT in increased CD8+ T cell exhaustion in trigeminal ganglia of mice latently infected with herpes simplex virus 1.A molecular and cellular model to explain the differences in reactivation from latency by herpes simplex and varicella-zoster viruses.
P2860

A review of the molecular mechanism of HSV-1 latency.

Item
http://www.wikidata.org/entity/Q36408944
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年学术文章
@wuu
1991年学术文章
@zh-cn
1991年学术文章
@zh-hans
1991年学术文章
@zh-my
1991年学术文章
@zh-sg
1991年學術文章
@yue
1991年學術文章
@zh
1991年學術文章
@zh-hant
name
A review of the molecular mechanism of HSV-1 latency.
@ast
A review of the molecular mechanism of HSV-1 latency.
@en
type
label
A review of the molecular mechanism of HSV-1 latency.
@ast
A review of the molecular mechanism of HSV-1 latency.
@en
prefLabel
A review of the molecular mechanism of HSV-1 latency.
@ast
A review of the molecular mechanism of HSV-1 latency.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P478
P577
P698
P356
P1433
P1476
A review of the molecular mechanism of HSV-1 latency.
@en
P2093
P2860
P304
P31
P356
P478
P577
P698