Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
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Herpes simplex virus latency-associated transcript is a stable intronRole of ND10 nuclear bodies in the chromatin repression of HSV-1Viral vectors for gene therapy in Parkinson's disease.Identification of a novel latency-specific splice donor signal within the herpes simplex virus type 1 2.0-kilobase latency-associated transcript (LAT): translation inhibition of LAT open reading frames by the intron within the 2.0-kilobase LAT.Interferon alpha induces establishment of alphaherpesvirus latency in sensory neurons in vitro.A 2.9-kilobase noncoding nuclear RNA functions in the establishment of persistent Hz-1 viral infection.Immunohistochemical analysis of primary sensory neurons latently infected with herpes simplex virus type 1.Pseudotyping of glycoprotein D-deficient herpes simplex virus type 1 with vesicular stomatitis virus glycoprotein G enables mutant virus attachment and entry.Evidence for a bidirectional element located downstream from the herpes simplex virus type 1 latency-associated promoter that increases its activity during latencyViral vectors in the treatment of Parkinson's disease.Enhancer and long-term expression functions of herpes simplex virus type 1 latency-associated promoter are both located in the same regionContrasts in codon usage of latent versus productive genes of Epstein-Barr virus: data and hypotheses.Herpes simplex virus type 1 latency-associated transcription unit promotes anatomical site-dependent establishment and reactivation from latency.HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis.HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part II. Vector systems and applicationsGene therapy with herpes simplex virus vectors: progress and prospects for clinical neuroscience.The stable 2.0-kilobase intron of the herpes simplex virus type 1 latency-associated transcript does not function as an antisense repressor of ICP0 in nonneuronal cellsUpregulation of mouse genes in HSV-1 latent TG after butyrate treatment implicates the multiple roles of the LAT-ICP0 locus.Herpes simplex virus type 1 latently infected neurons differentially express latency-associated and ICP0 transcripts.Experimental investigation of herpes simplex virus latencyThe region of the herpes simplex virus type 1 LAT gene that is colinear with the ICP34.5 gene is not involved in spontaneous reactivation.The spontaneous reactivation function of the herpes simplex virus type 1 LAT gene resides completely within the first 1.5 kilobases of the 8.3-kilobase primary transcript.A 371-nucleotide region between the herpes simplex virus type 1 (HSV-1) LAT promoter and the 2-kilobase LAT is not essential for efficient spontaneous reactivation of latent HSV-1Helper virus-free transfer of herpes simplex virus type 1 plasmid vectors into neural cells.The abundant latency-associated transcripts of herpes simplex virus type 1 are bound to polyribosomes in cultured neuronal cells and during latent infection in mouse trigeminal ganglia.Utilization of the herpes simplex virus type 1 latency-associated regulatory region to drive stable reporter gene expression in the nervous system.A LAT-associated function reduces productive-cycle gene expression during acute infection of murine sensory neurons with herpes simplex virus type 1.Altering central nervous system physiology with a defective herpes simplex virus vector expressing the glucose transporter gene.Expression of the herpes simplex virus type 2 latency-associated transcript enhances spontaneous reactivation of genital herpes in latently infected guinea pigsA review of the molecular mechanism of HSV-1 latency.Herpes simplex virus 1 (HSV-1) for cancer treatment.Competitive quantitative PCR analysis of herpes simplex virus type 1 DNA and latency-associated transcript RNA in latently infected cells of the rat brain.A novel latency-active promoter is contained within the herpes simplex virus type 1 UL flanking repeats.Effect of the transcription start region of the herpes simplex virus type 1 latency-associated transcript promoter on expression of productively infected neurons in vivo.PCR-based analysis of herpes simplex virus type 1 latency in the rat trigeminal ganglion established with a ribonucleotide reductase-deficient mutant.Long-term promoter activity during herpes simplex virus latencyStructure and function of the murine cytomegalovirus sgg1 gene: a determinant of viral growth in salivary gland acinar cells.The latency-associated transcript gene of herpes simplex virus type 1 (HSV-1) is required for efficient in vivo spontaneous reactivation of HSV-1 from latencyTwo open reading frames (ORF1 and ORF2) within the 2.0-kilobase latency-associated transcript of herpes simplex virus type 1 are not essential for reactivation from latencyInduction of reactivation of herpes simplex virus in murine sensory ganglia in vivo by cadmium.
P2860
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P2860
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
description
1989 nî lūn-bûn
@nan
1989 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
@ast
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
@en
Herpes simplex virus latent RNA
@nl
type
label
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
@ast
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
@en
Herpes simplex virus latent RNA
@nl
prefLabel
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
@ast
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
@en
Herpes simplex virus latent RNA
@nl
P2860
P356
P1476
Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.
@en
P2093
P2860
P304
P356
10.1073/PNAS.86.19.7596
P407
P577
1989-10-01T00:00:00Z